Pages that link to "Q73509448"
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The following pages link to Advanced glycation end products and their receptors co-localise in rat organs susceptible to diabetic microvascular injury (Q73509448):
Displaying 50 items.
- Accelerated aging in glaucoma: immunohistochemical assessment of advanced glycation end products in the human retina and optic nerve head (Q22306304) (← links)
- Advanced glycation endproduct (AGE) receptor 1 is a negative regulator of the inflammatory response to AGE in mesangial cells (Q24563997) (← links)
- Low levels of serum soluble receptors for advanced glycation end products, biomarkers for disease state: myth or reality (Q26859793) (← links)
- Advanced glycation end products cause epithelial-myofibroblast transdifferentiation via the receptor for advanced glycation end products (RAGE) (Q28366662) (← links)
- RAGE signaling in inflammation and arterial aging (Q33346824) (← links)
- Association analysis of ADPRT1, AKR1B1, RAGE, GFPT2 and PAI-1 gene polymorphisms with chronic renal insufficiency among Asian Indians with type-2 diabetes (Q33547739) (← links)
- Effects of a new advanced glycation inhibitor, LR-90, on mitigating arterial stiffening and improving arterial elasticity and compliance in a diabetic rat model: aortic impedance analysis (Q33746869) (← links)
- Dietary coconut kernel protein beneficially modulates NFκB and RAGE expression in streptozotocin induced diabetes in rats (Q34123437) (← links)
- Diabetes mellitus and risk of age-related macular degeneration: a systematic review and meta-analysis (Q34216036) (← links)
- Novel approaches to the treatment of progressive renal disease (Q34443235) (← links)
- Modulation of the cellular expression of circulating advanced glycation end-product receptors in type 2 diabetic nephropathy (Q34557348) (← links)
- Neurovascular disease, antioxidants and glycation in diabetes (Q34804227) (← links)
- Pulmonary functions in patients with diabetes mellitus (Q35030294) (← links)
- Regulation of RAGE for attenuating progression of diabetic vascular complications (Q35494903) (← links)
- Sugar-induced modification of fibroblast growth factor 2 reduces its angiogenic activity in vivo (Q35747582) (← links)
- Soluble RAGE and atherosclerosis in youth with type 1 diabetes: a 5-year follow-up study (Q36091982) (← links)
- High-mobility group box 1 inhibits HCO(3)(-) absorption in medullary thick ascending limb through a basolateral receptor for advanced glycation end products pathway (Q36174486) (← links)
- The receptor for advanced glycation end products impairs collateral formation in both diabetic and non-diabetic mice (Q36199320) (← links)
- Association of 2184AG Polymorphism in the RAGE Gene with Diabetic Nephropathy in Chinese Patients with Type 2 Diabetes (Q36378760) (← links)
- Heparanase induced by advanced glycation end products (AGEs) promotes macrophage migration involving RAGE and PI3K/AKT pathway (Q36797594) (← links)
- Advanced glycation end products increase lipids accumulation in macrophages through upregulation of receptor of advanced glycation end products: increasing uptake, esterification and decreasing efflux of cholesterol (Q37267563) (← links)
- Association of the receptor for advanced glycation end products gene polymorphisms and circulating RAGE levels with diabetic retinopathy in the Chinese population (Q37328633) (← links)
- Nicousamide protects kidney podocyte by inhibiting the TGFβ receptor II phosphorylation and AGE-RAGE signaling (Q37596871) (← links)
- Trends in advanced glycation end products research in diabetes mellitus and its complications. (Q37716694) (← links)
- Pathophysiology of the diabetic kidney. (Q38111707) (← links)
- Unlocking the biology of RAGE in diabetic microvascular complications (Q38134924) (← links)
- Kidney and eye diseases: common risk factors, etiological mechanisms, and pathways. (Q38171217) (← links)
- Renal injury is accelerated by global hypoxia-inducible factor 1 alpha deficiency in a mouse model of STZ-induced diabetes. (Q38642880) (← links)
- AGEs Promote Oxidative Stress and Induce Apoptosis in Retinal Pigmented Epithelium Cells RAGE-dependently (Q38909591) (← links)
- Advanced glycation end products can induce glial reaction and neuronal degeneration in retinal explants (Q39165652) (← links)
- Hyperglycaemia-induced pro-inflammatory responses by retinal Müller glia are regulated by the receptor for advanced glycation end-products (RAGE). (Q39656639) (← links)
- Association of Diabetic Neuropathy with Duration of Type 2 Diabetes and Glycemic Control. (Q40474030) (← links)
- Below the radar: advanced glycation end products that detour "around the side". Is HbA1c not an accurate enough predictor of long term progression and glycaemic control in diabetes? (Q40802125) (← links)
- Diabetic vascular complications (Q41604480) (← links)
- Sp1-binding elements in the promoter of RAGE are essential for amphoterin-mediated gene expression in cultured neuroblastoma cells (Q42463878) (← links)
- Role of the receptor for advanced glycation end products in hepatic fibrosis (Q42957259) (← links)
- Advanced glycation end-products induce apoptosis of bovine retinal pericytes in culture: involvement of diacylglycerol/ceramide production and oxidative stress induction (Q44054596) (← links)
- Renoprotective effects of vasopeptidase inhibition in an experimental model of diabetic nephropathy (Q44499796) (← links)
- Differential expression of receptors for advanced glycation end-products in peritoneal mesothelial cells exposed to glucose degradation products (Q45151232) (← links)
- The AGE-RAGE Axis: Implications for Age-Associated Arterial Diseases (Q47094524) (← links)
- Association of single-nucleotide polymorphisms in the RAGE gene and its gene- environment interactions with diabetic nephropathy in Chinese patients with type 2 diabetes (Q47119907) (← links)
- AGEs-RAGE overexpression in a patient with smoking-related idiopathic nodular glomerulosclerosis. (Q48225516) (← links)
- Advanced glycation end products decrease mesangial cell MMP-7: a role in matrix accumulation in diabetic nephropathy? (Q50985567) (← links)
- Kinetics and mechanism of the reaction of aminoguanidine with the alpha-oxoaldehydes glyoxal, methylglyoxal, and 3-deoxyglucosone under physiological conditions. (Q52078784) (← links)
- Calycosin protects HUVECs from advanced glycation end products-induced macrophage infiltration (Q54582285) (← links)
- Advanced glycation end-products induce heparanase expression in endothelial cells by the receptor for advanced glycation end products and through activation of the FOXO4 transcription factor (Q54599158) (← links)
- Coronary Microvascular Remodeling in Type 2 Diabetes: Synonymous With Early Aging? (Q58122201) (← links)
- A syndrome of molybdenosis, copper deficiency, and type 2 diabetes in the moose population of south-west Sweden (Q73797609) (← links)
- Polymorphisms in the RAGE gene influence susceptibility to diabetes-associated microvascular dermatoses in NIDDM (Q74215247) (← links)
- Potential role of advanced glycosylation end products in promoting restenosis in diabetes and renal failure (Q74708419) (← links)