Pages that link to "Q73120309"
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The following pages link to Tumor necrosis factor-alpha provokes a hypertrophic growth response in adult cardiac myocytes (Q73120309):
Displaying 50 items.
- Myotrophin-kappaB DNA interaction in the initiation process of cardiac hypertrophy (Q24298140) (← links)
- Cytokines as new treatment targets in chronic heart failure (Q24799394) (← links)
- The case for inhibiting p38 mitogen-activated protein kinase in heart failure (Q28080674) (← links)
- Full Expression of Cardiomyopathy Is Partly Dependent on B-Cells: A Pathway That Involves Cytokine Activation, Immunoglobulin Deposition, and Activation of Apoptosis (Q31037009) (← links)
- Serum uric acid and target organ damage in essential hypertension (Q33583766) (← links)
- Cardiac hypertrophy: sorting out the circuitry (Q33665828) (← links)
- Soluble tumor necrosis factor receptor 1 level is associated with left ventricular hypertrophy: the northern Manhattan study (Q33765516) (← links)
- Molecular Signals Elicited by GPCR Agonists in Hypertension, Cardiovascular Remodeling: Are MMPs and ADAMs Elusive Therapeutic Targets? (Q33807091) (← links)
- The role of tumor necrosis factor in the pathophysiology of heart failure (Q33862430) (← links)
- The role of tumor necrosis factor alpha in the pathophysiology of congestive heart failure (Q34027484) (← links)
- Recent insights into the role of tumor necrosis factor in the failing heart. (Q34222962) (← links)
- Pathophysiology of chronic heart failure (Q34239875) (← links)
- Tumor necrosis factor-alpha in cardiovascular biology and the potential role for anti-tumor necrosis factor-alpha therapy in heart disease (Q34792814) (← links)
- Mechanical stress-induced apoptosis in the cardiovascular system (Q34995311) (← links)
- Cardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas' disease cardiomyopathy. (Q35085050) (← links)
- Connective tissue growth factor and cardiac diastolic dysfunction: human data from the Taiwan diastolic heart failure registry and molecular basis by cellular and animal models (Q35086172) (← links)
- Temporal and gefitinib-sensitive regulation of cardiac cytokine expression via chronic β-adrenergic receptor stimulation (Q35086445) (← links)
- The role of anticytokine therapy in heart failure: recent lessons from preclinical and clinical trials? (Q35106477) (← links)
- Melatonin protects against myocardial hypertrophy induced by lipopolysaccharide (Q35490630) (← links)
- Association between Inflammation and Cardiac Geometry in Chronic Kidney Disease: Findings from the CRIC Study (Q35536642) (← links)
- The dynamic interaction between matrix metalloproteinase activity and adverse myocardial remodeling (Q35636522) (← links)
- Clinical modifiers for heart failure following myocardial infarction (Q36332855) (← links)
- Influence of tumour necrosis factor alpha on the outcome of ischaemic postconditioning in the presence of obesity and diabetes (Q36355264) (← links)
- Effects of a novel bradykinin B1 receptor antagonist and angiotensin II receptor blockade on experimental myocardial infarction in rats (Q36453772) (← links)
- The methionine 196 arginine polymorphism of the TNF receptor 2 gene (TNFRSF1B) is not associated with worse outcomes in heart failure (Q36669219) (← links)
- Early cardiac changes in a rat model of prediabetes: brain natriuretic peptide overexpression seems to be the best marker (Q36690425) (← links)
- Progressive Left Ventricular Hypertrophy after Heart Transplantation: Insights and Mechanisms Suggested by Multimodal Images (Q36736490) (← links)
- Endogenous tumor necrosis factor protects the adult cardiac myocyte against ischemic-induced apoptosis in a murine model of acute myocardial infarction (Q36977754) (← links)
- How can we cure a heart "in flame"? A translational view on inflammation in heart failure (Q37004826) (← links)
- Treatment with TNF-alpha or bacterial lipopolysaccharide attenuates endocardial endothelial cell-mediated stimulation of cardiac fibroblasts. (Q37122809) (← links)
- Substance P is required for the pathogenesis of EMCV infection in mice (Q37186717) (← links)
- Tumor necrosis factor-alpha and mortality in heart failure: a community study (Q37192858) (← links)
- Proinflammatory consequences of transgenic fas ligand expression in the heart (Q37194997) (← links)
- Proinflammatory cytokines in heart failure: double-edged swords (Q37734294) (← links)
- TNFα in myocardial ischemia/reperfusion, remodeling and heart failure (Q37767172) (← links)
- A role for sphingolipids in the pathophysiology of obesity-induced inflammation (Q37980387) (← links)
- Cardiac intercellular communication: are myocytes and fibroblasts fair-weather friends? (Q38046798) (← links)
- Fibrosis and heart failure (Q38057701) (← links)
- TWEAK-Fn14 Cytokine-Receptor Axis: A New Player of Myocardial Remodeling and Cardiac Failure (Q38194552) (← links)
- Hypertensive heart disease and obesity: a complex interaction between hemodynamic and not hemodynamic factors. (Q38211377) (← links)
- Immune cell and other noncardiomyocyte regulation of cardiac hypertrophy and remodeling (Q38380871) (← links)
- Inflammation - Cause or Consequence of Heart Failure or Both? (Q38664204) (← links)
- Mir-351-5p contributes to the establishment of a pro-inflammatory environment in the H9c2 cell line by repressing PTEN expression (Q38821745) (← links)
- Epigenetics, inflammation and metabolism in right heart failure associated with pulmonary hypertension (Q39382663) (← links)
- Increased Circulating Advanced Oxidation Protein Products and High-Sensitive Troponin T in Cirrhotic Patients with Chronic Hepatitis C: A Preliminary Report (Q40872573) (← links)
- Tumor necrosis factor-alpha induces VCAM-1-mediated inflammation via c-Src-dependent transactivation of EGF receptors in human cardiac fibroblasts (Q41594249) (← links)
- Restoring function in failing hearts: the effects of beta blockers (Q41732517) (← links)
- Effect of vildagliptin, a dipeptidyl peptidase 4 inhibitor, on cardiac hypertrophy induced by chronic beta-adrenergic stimulation in rats (Q41873186) (← links)
- The tumour suppressor Ras-association domain family protein 1A (RASSF1A) regulates TNF-α signalling in cardiomyocytes. (Q41900147) (← links)
- The role of Ca2+/calmodulin-dependent protein kinase II and calcineurin in TNF-α-induced myocardial hypertrophy (Q42095427) (← links)