Pages that link to "Q71824059"
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The following pages link to Site-directed mutagenesis of the katG gene of Mycobacterium tuberculosis: effects on catalase-peroxidase activities and isoniazid resistance (Q71824059):
Displaying 50 items.
- Evolution of drug resistance in tuberculosis: recent progress and implications for diagnosis and therapy (Q27016579) (← links)
- Catalase-peroxidase KatG of Burkholderia pseudomallei at 1.7A resolution (Q27640683) (← links)
- The molecular basis of resistance to isoniazid, rifampin, and pyrazinamide in Mycobacterium tuberculosis (Q28362898) (← links)
- Characterization of the W321F mutant of Mycobacterium tuberculosis catalase-peroxidase KatG (Q28366190) (← links)
- Analysis of ahpC gene mutations in isoniazid-resistant clinical isolates of Mycobacterium tuberculosis (Q28379128) (← links)
- Acute and persistent Mycobacterium tuberculosis infections depend on the thiol peroxidase TpX (Q28486700) (← links)
- Cu,Zn superoxide dismutase of Mycobacterium tuberculosis contributes to survival in activated macrophages that are generating an oxidative burst (Q28487322) (← links)
- Multidrug resistant Mycobacterium tuberculosis: a retrospective katG and rpoB mutation profile analysis in isolates from a reference center in Brazil (Q28541620) (← links)
- Mycobacterium tuberculosis Is Resistant to Isoniazid at a Slow Growth Rate by Single Nucleotide Polymorphisms in katG Codon Ser315 (Q28548282) (← links)
- Restricted structural gene polymorphism in the Mycobacterium tuberculosis complex indicates evolutionarily recent global dissemination (Q29618475) (← links)
- The molecular peculiarities of catalase-peroxidases. (Q30328007) (← links)
- Noninvasive determination of 2-[18F]-fluoroisonicotinic acid hydrazide pharmacokinetics by positron emission tomography in Mycobacterium tuberculosis-infected mice (Q30421576) (← links)
- Correlations of mutations in katG, oxyR-ahpC and inhA genes and in vitro susceptibility in Mycobacterium tuberculosis clinical strains segregated by spoligotype families from tuberculosis prevalent countries in South America (Q33410693) (← links)
- A Ser315Thr substitution in KatG is predominant in genetically heterogeneous multidrug-resistant Mycobacterium tuberculosis isolates originating from the St. Petersburg area in Russia (Q33697362) (← links)
- Molecular analysis of isoniazid-resistant Mycobacterium tuberculosis isolates from England and Wales reveals the phylogenetic significance of the ahpC -46A polymorphism (Q33722028) (← links)
- A Multinational Analysis of Mutations and Heterogeneity in PZase, RpsA, and PanD Associated with Pyrazinamide Resistance in M/XDR Mycobacterium tuberculosis (Q33812455) (← links)
- Three-dimensional model and molecular mechanism of Mycobacterium tuberculosis catalase-peroxidase (KatG) and isoniazid-resistant KatG mutants (Q33832195) (← links)
- Isoniazid-resistance conferring mutations in Mycobacterium tuberculosis KatG: catalase, peroxidase, and INH-NADH adduct formation activities (Q33836289) (← links)
- The genetics and biochemistry of isoniazid resistance in mycobacterium tuberculosis. (Q33960385) (← links)
- Molecular evidence for heterogeneity of the multiple-drug-resistant Mycobacterium tuberculosis population in Scotland (1990 to 1997). (Q33974563) (← links)
- Identification and characterization of mycobacterial proteins differentially expressed under standing and shaking culture conditions, including Rv2623 from a novel class of putative ATP-binding proteins (Q34009054) (← links)
- Molecular characterization of isoniazid-resistant Mycobacterium tuberculosis isolates collected in Australia (Q34077090) (← links)
- Effect of katG mutations on the virulence of Mycobacterium tuberculosis and the implication for transmission in humans. (Q34131132) (← links)
- Mutations prevalent among rifampin- and isoniazid-resistant Mycobacterium tuberculosis isolates from a hospital in Vietnam (Q34719791) (← links)
- Exploring the structure and function of the mycobacterial KatG protein using trans-dominant mutants (Q34721919) (← links)
- Isoniazid activation defects in recombinant Mycobacterium tuberculosis catalase-peroxidase (KatG) mutants evident in InhA inhibitor production (Q34820887) (← links)
- Population genetics study of isoniazid resistance mutations and evolution of multidrug-resistant Mycobacterium tuberculosis. (Q34973711) (← links)
- The effect of drug resistance on the fitness of Mycobacterium tuberculosis (Q35035354) (← links)
- Molecular detection of resistance to antituberculous therapy (Q35623138) (← links)
- Detection of multidrug resistance in Mycobacterium tuberculosis (Q35690316) (← links)
- Modification of the active site of Mycobacterium tuberculosis KatG after disruption of the Met-Tyr-Trp cross-linked adduct. (Q35829945) (← links)
- Radical sites in Mycobacterium tuberculosis KatG identified using electron paramagnetic resonance spectroscopy, the three-dimensional crystal structure, and electron transfer couplings (Q35829950) (← links)
- Molecular characterization of multidrug-resistant Mycobacterium tuberculosis isolated in Nepal. (Q36018561) (← links)
- A novel mechanism of growth phase-dependent tolerance to isoniazid in mycobacteria (Q36201896) (← links)
- Next-generation ion torrent sequencing of drug resistance mutations in Mycobacterium tuberculosis strains (Q36413991) (← links)
- Detection of a point mutation associated with high-level isoniazid resistance in Mycobacterium tuberculosis by using real-time PCR technology with 3'-minor groove binder-DNA probes (Q36891764) (← links)
- Identification of Mycobacterium tuberculosis clinical isolates with altered phagocytosis by human macrophages due to a truncated lipoarabinomannan (Q36968424) (← links)
- Multidrug-resistant Mycobacterium tuberculosis: molecular perspectives (Q37092384) (← links)
- Isoniazid resistance and the future of drug-resistant tuberculosis (Q37120865) (← links)
- Antibiotic resistance in Mycobacterium tuberculosis: peroxidase intermediate bypass causes poor isoniazid activation by the S315G mutant of M. tuberculosis catalase-peroxidase (KatG) (Q37269089) (← links)
- Screening and characterization of mutations in isoniazid-resistant Mycobacterium tuberculosis isolates obtained in Brazil (Q37489296) (← links)
- Actinobacterial Peroxidases: an Unexplored Resource for Biocatalysis (Q37833552) (← links)
- Resistance to Isoniazid and Ethionamide in Mycobacterium tuberculosis: Genes, Mutations, and Causalities. (Q38535724) (← links)
- Resistant mutants of Mycobacterium tuberculosis selected in vitro do not reflect the in vivo mechanism of isoniazid resistance (Q38905383) (← links)
- Analysis of the oxyR-ahpC region in isoniazid-resistant and -susceptible Mycobacterium tuberculosis complex organisms recovered from diseased humans and animals in diverse localities (Q39783805) (← links)
- katGI and katGII encode two different catalases-peroxidases in Mycobacterium fortuitum (Q39847830) (← links)
- Loss of oxyR in Mycobacterium tuberculosis (Q41592975) (← links)
- Use of site-directed mutagenesis to probe the structure, function and isoniazid activation of the catalase/peroxidase, KatG, from Mycobacterium tuberculosis (Q41818442) (← links)
- In house reverse membrane hybridisation assay versus GenoType MTBDRplus and their performance to detect mutations in the genes rpoB, katG and inhA. (Q41853395) (← links)
- The Bacillus licheniformis BlaP beta-lactamase as a model protein scaffold to study the insertion of protein fragments (Q42138035) (← links)