Pages that link to "Q68645460"
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The following pages link to An ATP pool with rapid turnover within the cell membrane (Q68645460):
Displaying 15 items.
- Differentiation of musculoskeletal tissues. (Q38680565) (← links)
- Inhibitory role of cyclic adenosine 3′5′-monophosphate in histamine release from rat peritoneal mast cells in vitro (Q39399219) (← links)
- Emiocytosis: a possible molecular mechanism (Q39435545) (← links)
- A Comparative Survey of the Function, Mechanism and Control of Cellular Oscillators (Q39830493) (← links)
- High cyclic AMP levels in young chick embryonic hearts (Q39918294) (← links)
- Determination of cyclic 3', 5'-AMP accumulation in resting and stimulated thyroid slices: Heterogeneity of the ATP precursor pool (Q39927950) (← links)
- Evidence against dopamine as the mediator of the rise of cyclic AMP in the superior cervical ganglion of the rat (Q39941223) (← links)
- An in Vitro system for studying insulin release caused by secretory granules-plasma membrane interaction: definition of the system (Q39990902) (← links)
- Cardiac adenosine 3':5'-monophosphate. Free and bound forms in the isolated rat atrium (Q40050061) (← links)
- Physiology and pharmacology of developing heart cells (Q40139532) (← links)
- Adenosine 3′:5′-cyclic monophosphate-dependent protein kinase(s) of rat ovarian cells. Gonadotropin regulation of adenosine 3′:5′-cyclic monophosphate-receptor activity (Q40169498) (← links)
- Receptor-mediated gonadotropin action in the ovary. Desensitization of gonadotropin binding sites, activation of adenosine 3‘:5‘-cyclic monophosphate-dependent protein kinase(s), and regulation of steroidogenesis in rat ovary (Q40231617) (← links)
- Stimulus-Secretion Coupling: Variations on the Theme of Calcium-Activated Exocytosis Involving Cellular and Extracellular Sources of Calcium (Q40381593) (← links)
- Effect of cyclic AMP on growth and morphological differentiation of an established myogenic cell line. (Q54393411) (← links)
- Insulin stimulates myogenesis in a rat myoblast line (Q59097025) (← links)