Pages that link to "Q64681190"
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The following pages link to Philippe Mangeot (Q64681190):
Displaying 18 items.
- Hepatitis C virus infection protein network (Q24311906) (← links)
- The interactomes of influenza virus NS1 and NS2 proteins identify new host factors and provide insights for ADAR1 playing a supportive role in virus replication (Q28115759) (← links)
- Generation and comprehensive analysis of an influenza virus polymerase cellular interaction network (Q28118085) (← links)
- IRGM is a common target of RNA viruses that subvert the autophagy network. (Q34102728) (← links)
- Expression of Pitx2 in stromal cells is required for normal hematopoiesis (Q34455431) (← links)
- Lentiviral vectors derived from simian immunodeficiency virus. (Q34562000) (← links)
- Dp412e: a novel human embryonic dystrophin isoform induced by BMP4 in early differentiated cells. (Q36278519) (← links)
- Terminal NK cell maturation is controlled by concerted actions of T-bet and Zeb2 and is essential for melanoma rejection (Q36286054) (← links)
- Protein transfer into human cells by VSV-G-induced nanovesicles. (Q38585869) (← links)
- A universal transgene silencing method based on RNA interference (Q41337955) (← links)
- AIM2/ASC triggers caspase-8-dependent apoptosis in Francisella-infected caspase-1-deficient macrophages. (Q42252067) (← links)
- High density lipoprotein inhibits hepatitis C virus-neutralizing antibodies by stimulating cell entry via activation of the scavenger receptor BI. (Q42999970) (← links)
- Mystery solved: VSV-G-LVs do not allow efficient gene transfer into unstimulated T cells, B cells, and HSCs because they lack the LDL receptor. (Q43944919) (← links)
- Genome editing in primary cells and in vivo using viral-derived Nanoblades loaded with Cas9-sgRNA ribonucleoproteins (Q60921859) (← links)
- Nanoblades: Pseudoviral shuttles for CRISPR-CAS9 delivery (Q92312722) (← links)
- A cohesin/HUSH- and LINC-dependent pathway controls ribosomal DNA double-strand break repair (Q92508214) (← links)
- A Versatile Strategy to Reduce UGA-Selenocysteine Recoding Efficiency of the Ribosome Using CRISPR-Cas9-Viral-Like-Particles Targeting Selenocysteine-tRNA[Ser]Sec Gene (Q92853762) (← links)
- Loop extrusion as a mechanism for formation of DNA damage repair foci (Q112333260) (← links)