Pages that link to "Q56797891"

The following pages link to Tariq Ahmed (Q56797891):

Displaying 32 items.

• Proper synaptic vesicle formation and neuronal network activity critically rely on syndapin I (Q24624560) (← links)
• Quinolinic acid injection in mouse medial prefrontal cortex affects reversal learning abilities, cortical connectivity and hippocampal synaptic plasticity (Q28821994) (← links)
• Spred1 is required for synaptic plasticity and hippocampus-dependent learning (Q33396815) (← links)
• Constitutive hippocampal cholesterol loss underlies poor cognition in old rodents. (Q33991462) (← links)
• TRPM4-dependent post-synaptic depolarization is essential for the induction of NMDA receptor-dependent LTP in CA1 hippocampal neurons (Q36688471) (← links)
• From tau phosphorylation to tau aggregation: what about neuronal death? (Q37775654) (← links)
• Tau deletion promotes brain insulin resistance (Q38708062) (← links)
• Cognitive defects are reversible in inducible mice expressing pro-aggregant full-length human Tau. (Q42280027) (← links)
• Separate Ionotropic and Metabotropic Glutamate Receptor Functions in Depotentiation vs. LTP: A Distinct Role for Group1 mGluR Subtypes and NMDARs. (Q42373065) (← links)
• Cognition and hippocampal synaptic plasticity in mice with a homozygous tau deletion (Q42460373) (← links)
• Expression of the specific type IV phosphodiesterase gene PDE4B3 during different phases of long-term potentiation in single hippocampal slices of rats in vitro. (Q44345685) (← links)
• Distinct and simultaneously active plasticity mechanisms in mouse hippocampus during different phases of Morris water maze training. (Q44750007) (← links)
• Regulation of the phosphodiesterase PDE4B3-isotype during long-term potentiation in the area dentata in vivo. (Q44802677) (← links)
• Postnatal disruption of the disintegrin/metalloproteinase ADAM10 in brain causes epileptic seizures, learning deficits, altered spine morphology, and defective synaptic functions. (Q45896474) (← links)
• System identification of mGluR-dependent long-term depression. (Q46071641) (← links)
• Plasticity-specific phosphorylation of CaMKII, MAP-kinases and CREB during late-LTP in rat hippocampal slices in vitro. (Q46591668) (← links)
• Loss of survivin in neural precursor cells results in impaired long-term potentiation in the dentate gyrus and CA1-region. (Q46610793) (← links)
• Genetically Induced Retrograde Amnesia of Associative Memories After Neuroplastin Ablation (Q47427701) (← links)
• Rescue of impaired late-phase long-term depression in a tau transgenic mouse model. (Q48166125) (← links)
• Aging Triggers a Repressive Chromatin State at Bdnf Promoters in Hippocampal Neurons. (Q48514225) (← links)
• Long-term effects of brief acute stress on cellular signaling and hippocampal LTP. (Q48586159) (← links)
• Phosphodiesterase 4B (PDE4B) and cAMP-level regulation within different tissue fractions of rat hippocampal slices during long-term potentiation in vitro. (Q48929933) (← links)
• Hippocampal tauopathy in tau transgenic mice coincides with impaired hippocampus-dependent learning and memory, and attenuated late-phase long-term depression of synaptic transmission. (Q48945423) (← links)
• Reversibility of Tau-related cognitive defects in a regulatable FTD mouse model (Q48959559) (← links)
• High fat diet treatment impairs hippocampal long-term potentiation without alterations of the core neuropathological features of Alzheimer disease (Q50086292) (← links)
• Single mild traumatic brain injury results in transiently impaired spatial long-term memory and altered search strategies. (Q50421145) (← links)
• Tau-induced defects in synaptic plasticity, learning, and memory are reversible in transgenic mice after switching off the toxic Tau mutant. (Q51019667) (← links)
• An N-methyl-D-aspartate-receptor dependent, late-phase long-term depression in middle-aged mice identifies no GluN2-subunit bias. (Q53083383) (← links)
• Reversal of memory and neuropsychiatric symptoms and reduced tau pathology by selenium in 3xTg-AD mice. (Q53402722) (← links)
• The Anti-Tumor Agent Sodium Selenate Decreases Methylated PP2A, Increases GSK3βY216 Phosphorylation, Including Tau Disease Epitopes and Reduces Neuronal Excitability in SHSY-5Y Neurons (Q64257020) (← links)
• Brain insulin response and peripheral metabolic changes in a Tau transgenic mouse model (Q91144107) (← links)
• Age-associated cholesterol reduction triggers brain insulin resistance by facilitating ligand-independent receptor activation and pathway desensitization (Q92447017) (← links)