Pages that link to "Q53317540"
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The following pages link to Intracellular Aβ-oligomers and early inflammation in a model of Alzheimer's disease (Q53317540):
Displaying 45 items.
- Intracellular Aβ pathology and early cognitive impairments in a transgenic rat overexpressing human amyloid precursor protein: a multidimensional study. (Q22001054) (← links)
- Repurposing psychiatric medicines to target activated microglia in anxious mild cognitive impairment and early Parkinson's disease (Q26748711) (← links)
- The Role of Inflammatory Mediators in the Pathogenesis of Alzheimer's Disease (Q26781815) (← links)
- Early accumulation of intracellular fibrillar oligomers and late congophilic amyloid angiopathy in mice expressing the Osaka intra-Aβ APP mutation (Q30534746) (← links)
- Neuroinflammation and related neuropathologies in APPSL mice: further value of this in vivo model of Alzheimer's disease (Q33939172) (← links)
- T cells promote the regeneration of neural precursor cells in the hippocampus of Alzheimer's disease mice (Q34319616) (← links)
- Neuroinflammation and neuronal loss precede Aβ plaque deposition in the hAPP-J20 mouse model of Alzheimer's disease. (Q34656208) (← links)
- Multi-target action of the novel anti-Alzheimer compound CHF5074: in vivo study of long term treatment in Tg2576 mice. (Q34656871) (← links)
- CD47 does not mediate amyloid-β(1-42) protofibril-stimulated microglial cytokine release (Q35032515) (← links)
- Amyloid β oligomers in Alzheimer's disease pathogenesis, treatment, and diagnosis (Q35327172) (← links)
- Monoacylglycerol lipase inhibitor JZL184 reduces neuroinflammatory response in APdE9 mice and in adult mouse glial cells (Q35559526) (← links)
- Immune aging, dysmetabolism, and inflammation in neurological diseases (Q35677347) (← links)
- Probing structural features of Alzheimer's amyloid-β pores in bilayers using site-specific amino acid substitutions (Q35691850) (← links)
- NGF in Early Embryogenesis, Differentiation, and Pathology in the Nervous and Immune Systems (Q35875769) (← links)
- Minocycline corrects early, pre-plaque neuroinflammation and inhibits BACE-1 in a transgenic model of Alzheimer's disease-like amyloid pathology (Q35959633) (← links)
- Targeting protein aggregation for the treatment of degenerative diseases (Q36235980) (← links)
- Molecular interaction study of N1-p-fluorobenzyl-cymserine with TNF-α , p38 kinase and JNK kinase. (Q36727886) (← links)
- A lifespan observation of a novel mouse model: in vivo evidence supports aβ oligomer hypothesis (Q37499825) (← links)
- Alzheimer's Toxic Amyloid Beta Oligomers: Unwelcome Visitors to the Na/K ATPase alpha3 Docking Station (Q37724402) (← links)
- Telomere shortening and Alzheimer's disease. (Q38060615) (← links)
- Neurotoxic saboteurs: straws that break the hippo's (hippocampus) back drive cognitive impairment and Alzheimer's Disease (Q38118901) (← links)
- Modulation of inflammation in transgenic models of Alzheimer's disease (Q38184606) (← links)
- The Distinct Role of ADAM17 in APP Proteolysis and Microglial Activation Related to Alzheimer's Disease (Q38539526) (← links)
- Aggregation of the Inflammatory S100A8 Precedes Aβ Plaque Formation in Transgenic APP Mice: Positive Feedback for S100A8 and Aβ Productions. (Q38774270) (← links)
- Inflammation in Alzheimer's disease: Lessons learned from microglia-depletion models (Q38891157) (← links)
- Of Mice and Men: Comparative Analysis of Neuro-Inflammatory Mechanisms in Human and Mouse Using Cause-and-Effect Models (Q41273323) (← links)
- Hippocampal metabolomics using ultrahigh-resolution mass spectrometry reveals neuroinflammation from Alzheimer's disease in CRND8 mice. (Q46649451) (← links)
- Impairment of Several Immune Functions and Redox State in Blood Cells of Alzheimer's Disease Patients. Relevant Role of Neutrophils in Oxidative Stress. (Q47551263) (← links)
- The Transient Receptor Potential Melastatin 2 (TRPM2) Channel Contributes to β-Amyloid Oligomer-Related Neurotoxicity and Memory Impairment. (Q48106243) (← links)
- White Ginseng Protects Mouse Hippocampal Cells Against Amyloid-Beta Oligomer Toxicity (Q48344833) (← links)
- Multiple inflammatory pathways are involved in the development and progression of cognitive deficits in APPswe/PS1dE9 mice (Q48692630) (← links)
- Differential effect of angiotensin II and blood pressure on hippocampal inflammation in mice. (Q50533718) (← links)
- The Amyloid-β Oligomer Hypothesis: Beginning of the Third Decade. (Q54977832) (← links)
- Cyclosporine A binding to COX-2 reveals a novel signaling pathway that activates the IRE1α unfolded protein response sensor (Q59329111) (← links)
- Experimental Pharmacology in Transgenic Rodent Models of Alzheimer's Disease (Q64067761) (← links)
- Physical Activity Alleviates Cognitive Dysfunction of Alzheimer's Disease through Regulating the mTOR Signaling Pathway (Q64103320) (← links)
- Calcium Ions Stimulate the Hyperphosphorylation of Tau by Activating Microsomal Prostaglandin E Synthase 1 (Q64259983) (← links)
- Qifu-Yin attenuates AGEs-induced Alzheimer-like pathophysiological changes through the RAGE/NF-κB pathway (Q86446427) (← links)
- The potential importance of myeloid-derived suppressor cells (MDSCs) in the pathogenesis of Alzheimer's disease (Q88738698) (← links)
- Early intraneuronal amyloid triggers neuron-derived inflammatory signaling in APP transgenic rats and human brain (Q90117079) (← links)
- Fluoxetine and Vortioxetine Reverse Depressive-Like Phenotype and Memory Deficits Induced by Aβ1-42 Oligomers in Mice: A Key Role of Transforming Growth Factor-β1 (Q91781569) (← links)
- Cell Clearing Systems Bridging Neuro-Immunity and Synaptic Plasticity (Q91795733) (← links)
- Neurophysiological Markers of Alzheimer's Disease: Quantitative EEG Approach (Q91944493) (← links)
- Evaluation on monoamine neurotransmitters changes in depression rats given with sertraline, meloxicam or/and caffeic acid (Q92714877) (← links)
- Remodeling of projections from ventral hippocampus to prefrontal cortex in Alzheimer's mice (Q99544929) (← links)