Pages that link to "Q52933605"
Jump to navigation
Jump to search
The following pages link to Role of MOG-stimulated Th1 type "light up" (GFP+) CD4+ T cells for the development of experimental autoimmune encephalomyelitis (EAE). (Q52933605):
Displaying 29 items.
- The immunomodulatory and neuroprotective effects of mesenchymal stem cells (MSCs) in experimental autoimmune encephalomyelitis (EAE): a model of multiple sclerosis (MS) (Q26823691) (← links)
- Multiple sclerosis: the role of cytokines in pathogenesis and in therapies (Q27011014) (← links)
- Design of oral agents for the management of multiple sclerosis: benefit and risk assessment for dimethyl fumarate (Q27021147) (← links)
- Immune modulation by chondroitin sulfate and its degraded disaccharide product in the development of an experimental model of multiple sclerosis (Q34069452) (← links)
- LXR-mediated inhibition of CD4+ T helper cells (Q34431434) (← links)
- Gene therapy in autoimmune, demyelinating disease of the central nervous system (Q35122159) (← links)
- Persistent macrophage/microglial activation and myelin disruption after experimental autoimmune encephalomyelitis in tissue inhibitor of metalloproteinase-1-deficient mice (Q35569899) (← links)
- Current status of the immunomodulation and immunomediated therapeutic strategies for multiple sclerosis (Q35606563) (← links)
- The role of IL-2 in the activation and expansion of regulatory T-cells and the development of experimental autoimmune encephalomyelitis (Q36640032) (← links)
- Targeting endothelial CD146 attenuates neuroinflammation by limiting lymphocyte extravasation to the CNS. (Q36775309) (← links)
- Pathways and gene networks mediating the regulatory effects of cannabidiol, a nonpsychoactive cannabinoid, in autoimmune T cells (Q36965753) (← links)
- Intravenous transfer of apoptotic cell-treated dendritic cells leads to immune tolerance by blocking Th17 cell activity (Q37026218) (← links)
- Inflammation on the mind: visualizing immunity in the central nervous system. (Q37184161) (← links)
- T-bet is essential for encephalitogenicity of both Th1 and Th17 cells (Q37273238) (← links)
- Pathogenic MOG-reactive CD8+ T cells require MOG-reactive CD4+ T cells for sustained CNS inflammation during chronic EAE. (Q37364443) (← links)
- Th1 versus Th17: are T cell cytokines relevant in multiple sclerosis? (Q37769909) (← links)
- Cytokine control of inflammation and repair in the pathology of multiple sclerosis. (Q38067389) (← links)
- Cannabinoids decrease the th17 inflammatory autoimmune phenotype (Q39119286) (← links)
- IL-23R-activated STAT3/STAT4 is essential for Th1/Th17-mediated CNS autoimmunity (Q40063804) (← links)
- Immunotherapy using lipopolysaccharide-stimulated bone marrow-derived dendritic cells to treat experimental autoimmune encephalomyelitis (Q44298534) (← links)
- Dimethyl Fumarate Selectively Reduces Memory T Cells and Shifts the Balance between Th1/Th17 and Th2 in Multiple Sclerosis Patients. (Q46409637) (← links)
- HU-446 and HU-465, Derivatives of the Non-psychoactive Cannabinoid Cannabidiol, Decrease the Activation of Encephalitogenic T Cells. (Q52307551) (← links)
- The HTLV-1 gp21 fusion peptide inhibits antigen specific T-cell activation in-vitro and in mice. (Q54957634) (← links)
- Dysregulated MicroRNA Involvement in Multiple Sclerosis by Induction of T Helper 17 Cell Differentiation. (Q55265126) (← links)
- Cannabidiol Attenuates Experimental Autoimmune Encephalomyelitis Model of Multiple Sclerosis Through Induction of Myeloid-Derived Suppressor Cells (Q58801886) (← links)
- Antibodies to myelin basic protein, myelin oligodendrocytes peptides, alpha-beta-crystallin, lymphocyte activation and cytokine production in patients with multiple sclerosis (Q73943317) (← links)
- Prophylactic treatment against GM-CSF, but not IL-17, abolishes relapses in a chronic murine model of multiple sclerosis (Q91449134) (← links)
- Extracellular vesicles from human umbilical cord blood plasma modulate interleukin-2 signaling of T cells to ameliorate experimental autoimmune encephalomyelitis (Q92063575) (← links)
- ADAM12 is a costimulatory molecule that determines Th1 cell fate and mediates tissue inflammation (Q96640883) (← links)