Pages that link to "Q48355305"
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The following pages link to Slit and glypican-1 mRNAs are coexpressed in the reactive astrocytes of the injured adult brain (Q48355305):
Displaying 48 items.
- Regeneration beyond the glial scar (Q28239915) (← links)
- Immunofluorescence laser confocal expression and localization study of rat nerve growth guidance cues Netrin-1 and Slit2 after spinal cord injury (Q33326353) (← links)
- A chemical screen identifies novel compounds that overcome glial-mediated inhibition of neuronal regeneration. (Q33548229) (← links)
- Recombinant Slit2 Reduces Surgical Brain Injury Induced Blood Brain Barrier Disruption via Robo4 Dependent Rac1 Activation in a Rodent Model. (Q33677789) (← links)
- Heparan sulfate accumulation with Abeta deposits in Alzheimer's disease and Tg2576 mice is contributed by glial cells (Q33795144) (← links)
- Extracellular matrix regulation of inflammation in the healthy and injured spinal cord (Q33906928) (← links)
- Growth and guidance cues for regenerating axons: where have they gone? (Q35685294) (← links)
- Proteoglycans and injury of the central nervous system (Q35964844) (← links)
- Inhibitors of slit protein interactions with the heparan sulphate proteoglycan glypican-1: potential agents for the treatment of spinal cord injury (Q36078175) (← links)
- Gene therapy: can neural stem cells deliver? (Q36350210) (← links)
- Recombinant Slit2 attenuates neuroinflammation after surgical brain injury by inhibiting peripheral immune cell infiltration via Robo1-srGAP1 pathway in a rat model (Q36395875) (← links)
- CNS injury, glial scars, and inflammation: Inhibitory extracellular matrices and regeneration failure (Q36499527) (← links)
- Recapitulate development to promote axonal regeneration: good or bad approach? (Q36581080) (← links)
- Combinatorial treatments for promoting axon regeneration in the CNS: strategies for overcoming inhibitory signals and activating neurons' intrinsic growth state (Q36827341) (← links)
- Navigating their way to the clinic: emerging roles for axon guidance molecules in neurological disorders and injury (Q36827349) (← links)
- Susceptibility to glaucoma: differential comparison of the astrocyte transcriptome from glaucomatous African American and Caucasian American donors (Q36864757) (← links)
- Rewiring the injured CNS: lessons from the optic nerve (Q36870250) (← links)
- Characterization of the interaction between Robo1 and heparin and other glycosaminoglycans (Q37410475) (← links)
- Neural regenerative strategies incorporating biomolecular axon guidance signals (Q37973260) (← links)
- Potentials of endogenous neural stem cells in cortical repair. (Q38003144) (← links)
- Concise review: human pluripotent stem cells in the treatment of spinal cord injury (Q38021973) (← links)
- Neuronal restoration following ischemic stroke: influences, barriers, and therapeutic potential (Q38080032) (← links)
- Scar-modulating treatments for central nervous system injury (Q38222632) (← links)
- Influence of the extracellular matrix on endogenous and transplanted stem cells after brain damage (Q38246263) (← links)
- The role of neuronal versus astrocyte-derived heparan sulfate proteoglycans in brain development and injury (Q38252269) (← links)
- Transcriptomic profiling of astrocytes treated with the Rho kinase inhibitor Fasudil reveals cytoskeletal and pro‐survival responses (Q38502206) (← links)
- Exploring Optic Nerve Axon Regeneration (Q38777316) (← links)
- The role of semaphorin3A in myogenic regeneration and the formation of functional neuromuscular junctions on new fibres (Q38863834) (← links)
- Hyper-IL-6: a potent and efficacious stimulator of RGC regeneration (Q39019470) (← links)
- Brief report: astrogliosis promotes functional recovery of completely transected spinal cord following transplantation of hESC-derived oligodendrocyte and motoneuron progenitors (Q39334966) (← links)
- Increased Expression of Slit2 and its Robo Receptors During Astroglial Scar Formation After Transient Focal Cerebral Ischemia in Rats. (Q39337786) (← links)
- Possible implication of satellite cells in regenerative motoneuritogenesis: HGF upregulates neural chemorepellent Sema3A during myogenic differentiation. (Q42455209) (← links)
- Serotonin axons in the neocortex of the adult female mouse regrow after traumatic brain injury. (Q47769778) (← links)
- Extracellular matrix and traumatic brain injury (Q47854507) (← links)
- Heparan Sulfate Proteoglycans as Relays of Neuroinflammation. (Q47888806) (← links)
- Dynamic changes in glypican-1 expression in dorsal root ganglion neurons after peripheral and central axonal injury (Q47965200) (← links)
- Determinants of Axon Growth, Plasticity, and Regeneration in the Context of Spinal Cord Injury. (Q48020080) (← links)
- Expression pattern of glypican-1 mRNA after brain injury in mice (Q48214277) (← links)
- Maximizing functional axon repair in the injured central nervous system: Lessons from neuronal development (Q48232313) (← links)
- Interaction of reactive astrocytes with type I collagen induces astrocytic scar formation through the integrin-N-cadherin pathway after spinal cord injury (Q48258249) (← links)
- Morphological phenotypes of olfactory ensheathing cells display different migratory responses upon Slit-2. (Q48480401) (← links)
- Intracellular trafficking in neurones and glia of fibroblast growth factor-2, fibroblast growth factor receptor 1 and heparan sulphate proteoglycans in the injured adult rat cerebral cortex (Q48685507) (← links)
- Environmental cues determine the fate of astrocytes after spinal cord injury (Q49397380) (← links)
- Reactive nonproliferative gliosis predominates in a chronic mouse model of glaucoma. (Q53557356) (← links)
- Overexpression of Slit2 improves function of the paravascular pathway in the aging mouse brain (Q58802958) (← links)
- Studies on the development and behavior of the dystrophic growth cone, the hallmark of regeneration failure, in an in vitro model of the glial scar and after spinal cord injury (Q80370660) (← links)
- An update on reactive astrocytes in chronic pain (Q91750699) (← links)
- Mature oligodendrocytes bordering lesions limit demyelination and favor myelin repair via heparan sulphate production (Q96222241) (← links)