Pages that link to "Q47725275"
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The following pages link to Reversal of collapsing glomerulopathy in mice with the cyclin-dependent kinase inhibitor CYC202. (Q47725275):
Displaying 35 items.
- Roscovitine inhibits activation of promoters in herpes simplex virus type 1 genomes independently of promoter-specific factors (Q24562983) (← links)
- Nef stimulates proliferation of glomerular podocytes through activation of Src-dependent Stat3 and MAPK1,2 pathways (Q24563306) (← links)
- Podocytes (Q26766118) (← links)
- Resolution of inflammation: an integrated view (Q26852569) (← links)
- Viral Subversion Mechanisms in Chronic Kidney Disease Pathogenesis (Q27485200) (← links)
- Collapsing focal segmental glomerulosclerosis in a patient with systemic lupus erythematosus (Q34087122) (← links)
- Pathophysiology and treatment of focal segmental glomerulosclerosis: the role of animal models (Q34645201) (← links)
- HIV-associated nephropathy: clinical presentation, pathology, and epidemiology in the era of antiretroviral therapy (Q34657666) (← links)
- TNFR2 interposes the proliferative and NF-κB-mediated inflammatory response by podocytes to TNF-α. (Q34788604) (← links)
- The Role of Pharmacokinetics and Pharmacodynamics in Early Drug Development with reference to the Cyclin-dependent Kinase (Cdk) Inhibitor - Roscovitine (Q35062145) (← links)
- Cyclin-dependent kinase inhibitors block leukocyte adhesion and migration (Q36215726) (← links)
- HIV and the kidney: a status report after 20 years (Q36223766) (← links)
- Modulating Innate and Adaptive Immunity by (R)-Roscovitine: Potential Therapeutic Opportunity in Cystic Fibrosis (Q36709312) (← links)
- Five years of progress on cyclin-dependent kinases and other cellular proteins as potential targets for antiviral drugs (Q36716750) (← links)
- Cyclin-dependent kinases: molecular switches controlling anergy and potential therapeutic targets for tolerance (Q36769222) (← links)
- Pyrazolo[1,5-a]-1,3,5-triazine as a purine bioisostere: access to potent cyclin-dependent kinase inhibitor (R)-roscovitine analogue. (Q37098618) (← links)
- Therapeutic targets in focal and segmental glomerulosclerosis (Q37228164) (← links)
- Present and future drug treatments for chronic kidney diseases: evolving targets in renoprotection (Q37290939) (← links)
- The cell cycle and acute kidney injury (Q37436067) (← links)
- CDK4/6 inhibition induces epithelial cell cycle arrest and ameliorates acute kidney injury. (Q37575488) (← links)
- Cyclin-dependent kinase inhibitor drugs as potential novel anti-inflammatory and pro-resolution agents (Q37602210) (← links)
- Controversies in the pathogenesis of HIV-associated renal diseases (Q37602527) (← links)
- Collapsing focal segmental glomerulosclerosis: Current concepts (Q39278018) (← links)
- Reduction of ciliary length through pharmacologic or genetic inhibition of CDK5 attenuates polycystic kidney disease in a model of nephronophthisis. (Q41584686) (← links)
- Mice with altered alpha-actinin-4 expression have distinct morphologic patterns of glomerular disease (Q42364564) (← links)
- Persistent NF-kappaB activation in renal epithelial cells in a mouse model of HIV-associated nephropathy (Q42746343) (← links)
- Homology modeling, molecular dynamic simulation and docking studies of cyclin dependent kinase 1. (Q44122747) (← links)
- Casein kinase 1ε and 1α as novel players in polycystic kidney disease and mechanistic targets for (R)-roscovitine and (S)-CR8. (Q52656392) (← links)
- Molecular Mechanisms of Proteinuria in Focal Segmental Glomerulosclerosis. (Q52715295) (← links)
- Therapeutics in renal disease: the road ahead for antiproliferative targets. (Q53644742) (← links)
- Discovery of the antiviral activities of pharmacologic cyclin-dependent kinase inhibitors: from basic to applied science. (Q53674064) (← links)
- Treatment with a cyclin‐dependent kinase inhibitor, seliciclib, is effective in reducing glomerular macrophage numbers and the severity of established experimental glomerulonephritis (Q54594584) (← links)
- Kidney in viral infections (Q57691296) (← links)
- Recent Progress in the Pathogenesis of Nephrotic Proteinuria (Q57825276) (← links)
- A Simple Isomerization of the Purine Scaffold of a Kinase Inhibitor, Roscovitine, Affords a Four- to Seven-Fold Enhancement of Its Affinity for Four CDKs. Could This Be Traced Back to Conjugation-Induced Stiffenings/Loosenings of Rotational Barriers? (Q58736257) (← links)