Pages that link to "Q46917709"
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The following pages link to Both early and late stages of hepatocarcinogenesis are enhanced in Cx32 dominant negative mutant transgenic rats with disrupted gap junctional intercellular communication (Q46917709):
Displaying 11 items.
- Transgenic rat models for mutagenesis and carcinogenesis (Q28817612) (← links)
- Connexin 32 dysfunction promotes ethanol-related hepatocarcinogenesis via activation of Dusp1-Erk axis (Q36739026) (← links)
- Gap junctional intercellular communication as a target for liver toxicity and carcinogenicity (Q37563482) (← links)
- Localization of connexin 32 in spontaneous liver lesions of mice (Q38373175) (← links)
- Connexin 32 and luteolin play protective roles in non-alcoholic steatohepatitis development and its related hepatocarcinogenesis in rats (Q38825737) (← links)
- The role of gap junctions in inflammatory and neoplastic disorders (Review). (Q39093592) (← links)
- Silencing of connexin 43 suppresses invasion, migration and lung metastasis of rat hepatocellular carcinoma cells (Q39399257) (← links)
- Connexin-based signaling and drug-induced hepatotoxicity (Q41455106) (← links)
- Age-dependent carcinogenic susceptibility in rat liver is related to potential of gap junctional intercellular communication (Q42718282) (← links)
- Gap junction dysfunction reduces acetaminophen hepatotoxicity with impact on apoptotic signaling and connexin 43 protein induction in rat. (Q43185237) (← links)
- Cx32 exerts anti-apoptotic and pro-tumor effects via the epidermal growth factor receptor pathway in hepatocellular carcinoma (Q64076789) (← links)