Pages that link to "Q45302664"
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The following pages link to Trinucleotide expansion in haploid germ cells by gap repair (Q45302664):
Displaying 50 items.
- Roles of transition nuclear proteins in spermiogenesis (Q24301755) (← links)
- DNA mismatch repair: molecular mechanism, cancer, and ageing (Q24647002) (← links)
- Comparative genomics and molecular dynamics of DNA repeats in eukaryotes (Q24650948) (← links)
- Mechanistic features of CAG*CTG repeat contractions in cultured cells revealed by a novel genetic assay (Q24810710) (← links)
- Inverted repeat-stimulated sister-chromatid exchange events are RAD1-independent but reduced in a msh2 mutant (Q24815214) (← links)
- Advances in mechanisms of genetic instability related to hereditary neurological diseases (Q24816457) (← links)
- DNA triplet repeat expansion and mismatch repair (Q26996542) (← links)
- Repeat-mediated genetic and epigenetic changes at the FMR1 locus in the Fragile X-related disorders (Q27011565) (← links)
- Chromosomal translocations and palindromic AT-rich repeats (Q27024347) (← links)
- Suppression of Somatic Expansion Delays the Onset of Pathophysiology in a Mouse Model of Huntington’s Disease (Q27311065) (← links)
- Crystal structure of actinomycin D bound to the CTG triplet repeat sequences linked to neurological diseases (Q27639967) (← links)
- The structural basis of actinomycin D-binding induces nucleotide flipping out, a sharp bend and a left-handed twist in CGG triplet repeats (Q27676374) (← links)
- Molecular genetics and genetic testing in myotonic dystrophy type 1. (Q27691417) (← links)
- The 26S proteasome drives trinucleotide repeat expansions (Q27933260) (← links)
- Identification of RTG2 as a modifier gene for CTG*CAG repeat instability in Saccharomyces cerevisiae (Q27934694) (← links)
- Expansion, mosaicism and interruption: mechanisms of the CAG repeat mutation in spinocerebellar ataxia type 1 (Q28069787) (← links)
- The fragile X gene and its function (Q28201533) (← links)
- Mechanisms and consequences of somatic mosaicism in humans (Q28205052) (← links)
- Mechanism of trinucleotide repeats instabilities: the necessities of repeat non-B secondary structure formation and the roles of cellular trans-acting factors (Q28294940) (← links)
- Role of oxidative DNA damage in mitochondrial dysfunction and Huntington's disease pathogenesis (Q28386202) (← links)
- Localization of MMR proteins on meiotic chromosomes in mice indicates distinct functions during prophase I (Q28505426) (← links)
- GFP-based fluorescence assay for CAG repeat instability in cultured human cells (Q28545187) (← links)
- Onset and progression of behavioral and molecular phenotypes in a novel congenic R6/2 line exhibiting intergenerational CAG repeat stability (Q28741746) (← links)
- Huntington disease expansion mutations in humans can occur before meiosis is completed (Q30962381) (← links)
- Stoichiometry of base excision repair proteins correlates with increased somatic CAG instability in striatum over cerebellum in Huntington's disease transgenic mice (Q30972892) (← links)
- Diverse effects of individual mismatch repair components on transcription-induced CAG repeat instability in human cells (Q33460873) (← links)
- Progressive GAA.TTC repeat expansion in human cell lines (Q33513958) (← links)
- DNA polymerase beta is critical for mouse meiotic synapsis (Q33519489) (← links)
- Convergent transcription through a long CAG tract destabilizes repeats and induces apoptosis (Q33637169) (← links)
- Palindrome-mediated chromosomal translocations in humans (Q33668703) (← links)
- Two modes of germline instability at human minisatellite MS1 (locus D1S7): complex rearrangements and paradoxical hyperdeletion. (Q33904840) (← links)
- Topoisomerase 1 and single-strand break repair modulate transcription-induced CAG repeat contraction in human cells (Q33917909) (← links)
- A selective inhibitor of histone deacetylase 3 prevents cognitive deficits and suppresses striatal CAG repeat expansions in Huntington's disease mice (Q33920945) (← links)
- Intergenerational instability of the expanded CTG repeat in the DMPK gene: studies in human gametes and preimplantation embryos (Q33998571) (← links)
- Correlation of inter-locus polyglutamine toxicity with CAG•CTG triplet repeat expandability and flanking genomic DNA GC content (Q34097811) (← links)
- Histone deacetylase complexes promote trinucleotide repeat expansions (Q34169694) (← links)
- DNA repair deficiency in neurodegeneration. (Q34183328) (← links)
- Nucleotide excision repair, mismatch repair, and R-loops modulate convergent transcription-induced cell death and repeat instability (Q34442152) (← links)
- Non-B DNA structure-induced genetic instability and evolution (Q34473652) (← links)
- Sequence length dictates repeated CAG folding in three-way junctions (Q34520527) (← links)
- Huntington's disease: new hope for therapeutics (Q34553826) (← links)
- Epigenetic mechanisms for primary differentiation in mammalian embryos (Q34670846) (← links)
- Long CTG Tracts from the Myotonic Dystrophy Gene Induce Deletions and Rearrangements during Recombination at the APRT Locus in CHO Cells (Q34930681) (← links)
- CTG repeat instability and size variation timing in DNA repair-deficient mice (Q35018750) (← links)
- Microsatellite repeat instability and neurological disease (Q35064141) (← links)
- Repair in haploid male germ cells occurs late in differentiation as chromatin is condensing (Q35103004) (← links)
- CAG*CTG repeat instability in cultured human astrocytes (Q35130388) (← links)
- Cockayne syndrome B protein antagonizes OGG1 in modulating CAG repeat length in vivo (Q35166110) (← links)
- Modifiers of (CAG)(n) instability in Machado-Joseph disease (MJD/SCA3) transmissions: an association study with DNA replication, repair and recombination genes (Q35207236) (← links)
- Mechanisms of trinucleotide repeat instability during human development (Q35219284) (← links)