Pages that link to "Q44538161"

The following pages link to A20 is dynamically regulated in the heart and inhibits the hypertrophic response (Q44538161):

Displaying 28 items.

• Targeted cardiac overexpression of A20 improves left ventricular performance and reduces compensatory hypertrophy after myocardial infarction (Q24300967) (← links)
• Integrated genomic approaches implicate osteoglycin (Ogn) in the regulation of left ventricular mass (Q24656531) (← links)
• A20 (TNFAIP3) alleviates CVB3-induced myocarditis via inhibiting NF-κB signaling (Q27316099) (← links)
• A-kinase-anchoring protein-Lbc anchors IκB kinase β to support interleukin-6-mediated cardiomyocyte hypertrophy (Q28116093) (← links)
• Tumor suppressor A20 protects against cardiac hypertrophy and fibrosis by blocking transforming growth factor-beta-activated kinase 1-dependent signaling (Q34615889) (← links)
• Cardiomyocyte p65 nuclear factor-κB is necessary for compensatory adaptation to pressure overload (Q35002059) (← links)
• Ubiquitin carboxyl terminal hydrolyase L1-suppressed autophagic degradation of p21WAF1/Cip1 as a novel feedback mechanism in the control of cardiac fibroblast proliferation (Q35147643) (← links)
• Prevention and reversal of cardiac hypertrophy by soluble epoxide hydrolase inhibitors (Q35214761) (← links)
• Nuclear factor-kappaB: its role in health and disease (Q35791198) (← links)
• Interaction between NFκB and NFAT coordinates cardiac hypertrophy and pathological remodeling (Q35926510) (← links)
• Liver X receptors are negative regulators of cardiac hypertrophy via suppressing NF-kappaB signalling (Q37341054) (← links)
• Regulation of nuclear factor κB (NF-κB) in the nucleus of cardiomyocytes by G protein-coupled receptor kinase 5 (GRK5). (Q37388772) (← links)
• Novel Protective Role of Myeloid Differentiation 1 in Pathological Cardiac Remodelling (Q37626086) (← links)
• Tapping the brake on cardiac growth-endogenous repressors of hypertrophic signaling- (Q37876521) (← links)
• Noncanonical Nuclear Factor Kappa B (NF-κB) Signaling and Potential for Therapeutics in Sepsis (Q38131458) (← links)
• Fas-associated death-domain protein inhibits TNF-alpha mediated NF-kappaB activation in cardiomyocytes. (Q40404611) (← links)
• Interleukin-18 is a pro-hypertrophic cytokine that acts through a phosphatidylinositol 3-kinase-phosphoinositide-dependent kinase-1-Akt-GATA4 signaling pathway in cardiomyocytes (Q40485863) (← links)
• The CRM1 nuclear export receptor controls pathological cardiac gene expression (Q40586178) (← links)
• Inhibition of NF-kappaB induces regression of cardiac hypertrophy, independent of blood pressure control, in spontaneously hypertensive rats (Q45304598) (← links)
• Protective effect of the endothelin antagonist CPU0213 against isoprenaline-induced heart failure by suppressing abnormal expression of leptin, calcineurin and SERCA2a in rats (Q46579855) (← links)
• Role of the NF-kappaB signaling cascade and NF-kappaB-targeted genes in failing human hearts (Q46679333) (← links)
• Deubiquitinating enzyme CYLD mediates pressure overload-induced cardiac maladaptive remodeling and dysfunction via downregulating Nrf2. (Q46734603) (← links)
• Blockade of MyD88 attenuates cardiac hypertrophy and decreases cardiac myocyte apoptosis in pressure overload-induced cardiac hypertrophy in vivo (Q50752354) (← links)
• The role of K63-linked polyubiquitination in cardiac hypertrophy (Q58784086) (← links)
• A20 alleviates the vascular remodeling induced by homocysteine (Q60953194) (← links)
• An autocrine role for leptin in mediating the cardiomyocyte hypertrophic effects of angiotensin II and endothelin-1 (Q79808415) (← links)
• Lipopolysaccharide induces cellular hypertrophy through calcineurin/NFAT-3 signaling pathway in H9c2 myocardiac cells (Q81044990) (← links)
• CYLD exaggerates pressure overload-induced cardiomyopathy via suppressing autolysosome efflux in cardiomyocytes (Q96586585) (← links)