Pages that link to "Q44307018"
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The following pages link to 5-HT2 receptors promote plateau potentials in turtle spinal motoneurons by facilitating an L-type calcium current. (Q44307018):
Displaying 50 items.
- Serotonin spillover onto the axon initial segment of motoneurons induces central fatigue by inhibiting action potential initiation (Q24599332) (← links)
- Mechanisms intrinsic to 5-HT2B receptor-induced potentiation of NMDA receptor responses in frog motoneurones (Q24671497) (← links)
- The whisking rhythm generator: a novel mammalian network for the generation of movement (Q24680347) (← links)
- Synaptic release of serotonin induced by stimulation of the raphe nucleus promotes plateau potentials in spinal motoneurons of the adult turtle (Q28270000) (← links)
- Increased serotonergic innervation of lumbosacral motoneurons of rolling mouse Nagoya in correlation with abnormal hindlimb extension (Q28510951) (← links)
- Recovery of motoneuron and locomotor function after spinal cord injury depends on constitutive activity in 5-HT2C receptors (Q30500846) (← links)
- The 5-HT2A receptor is widely distributed in the rat spinal cord and mainly localized at the plasma membrane of postsynaptic neurons (Q33200989) (← links)
- Management of spasticity after spinal cord injury: current techniques and future directions (Q33814100) (← links)
- Effect of fluoxetine on disease progression in a mouse model of ALS. (Q33900692) (← links)
- Amphetamine increases persistent inward currents in human motoneurons estimated from paired motor-unit activity (Q33920820) (← links)
- Persistent inward currents in spinal motoneurons: important for normal function but potentially harmful after spinal cord injury and in amyotrophic lateral sclerosis (Q34399203) (← links)
- Motoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity (Q34686451) (← links)
- Polysynaptic excitatory postsynaptic potentials that trigger spasms after spinal cord injury in rats are inhibited by 5-HT1B and 5-HT1F receptors (Q35160555) (← links)
- Decrease of mRNA Editing after Spinal Cord Injury is Caused by Down-regulation of ADAR2 that is Triggered by Inflammatory Response (Q35724883) (← links)
- Persistent inward currents in spinal motoneurons and their influence on human motoneuron firing patterns (Q35889120) (← links)
- Synaptic integration in motoneurons with hyper-excitable dendrites (Q35938012) (← links)
- Altered patterns of reflex excitability, balance, and locomotion following spinal cord injury and locomotor training (Q36193667) (← links)
- Role of endogenous release of norepinephrine in muscle spasms after chronic spinal cord injury (Q36227595) (← links)
- Transition to persistent orofacial pain after nerve injury involves supraspinal serotonin mechanisms (Q36807025) (← links)
- 5-HT2C-like receptors in the brain of Xenopus laevis initiate sex-typical fictive vocalizations (Q37298797) (← links)
- Serotonin 5-HT2 receptors induce a long-lasting facilitation of spinal reflexes independent of ionotropic receptor activity. (Q37351035) (← links)
- Motoneuron excitability: the importance of neuromodulatory inputs (Q37603971) (← links)
- Alpha, beta and gamma motoneurons: functional diversity in the motor system's final pathway. (Q37940907) (← links)
- Exploring the central modulation hypothesis: do ancient memory mechanisms underlie the pathophysiology of trigger points? (Q38109576) (← links)
- Motor unit (Q38110501) (← links)
- Serotonergic transmission after spinal cord injury (Q38215361) (← links)
- Calcium signalling through L-type calcium channels: role in pathophysiology of spinal nociceptive transmission (Q39141992) (← links)
- Voltage-gated calcium channels are abnormal in cultured spinal motoneurons in the G93A-SOD1 transgenic mouse model of ALS. (Q41842390) (← links)
- Fast and reliable identification of axons, axon initial segments and dendrites with local field potential recording. (Q42086851) (← links)
- Activity-dependent initiation of a prolonged depolarization in aplysia bag cell neurons: role for a cation channel (Q42165824) (← links)
- Multiple patterns and components of persistent inward current with serotonergic modulation in locomotor activity-related neurons in Cfos-EGFP mice (Q43158669) (← links)
- Persistent Ca2+ current contributes to a prolonged depolarization in Aplysia bag cell neurons (Q43261430) (← links)
- Recruitment of motor neuronal persistent inward currents shapes withdrawal reflexes in the frog. (Q45140559) (← links)
- Increased persistent Na(+) current and its effect on excitability in motoneurones cultured from mutant SOD1 mice. (Q45223589) (← links)
- Spatial-specific action of serotonin within the leech midbody ganglion (Q46683566) (← links)
- Computational estimation of the distribution of L-type Ca(2+) channels in motoneurons based on variable threshold of activation of persistent inward currents (Q46788543) (← links)
- Persistent sodium currents and repetitive firing in motoneurons of the sacrocaudal spinal cord of adult rats (Q46799657) (← links)
- Serotonin differentially modulates the intrinsic properties of spinal motoneurons from the adult turtle (Q46838506) (← links)
- 5-HT2 receptor activation facilitates a persistent sodium current and repetitive firing in spinal motoneurons of rats with and without chronic spinal cord injury (Q47111379) (← links)
- Apamin-sensitive calcium-activated potassium currents (SK) are activated by persistent calcium currents in rat motoneurons (Q47132787) (← links)
- Serotonin facilitates a persistent calcium current in motoneurons of rats with and without chronic spinal cord injury (Q47145681) (← links)
- Endogenous monoamine receptor activation is essential for enabling persistent sodium currents and repetitive firing in rat spinal motoneurons (Q47148816) (← links)
- Presynaptic serotonin 5-HT1B/D receptor-mediated inhibition of glycinergic transmission to the frog spinal motoneurons (Q47177336) (← links)
- Spastic long-lasting reflexes in the awake rat after sacral spinal cord injury (Q47803877) (← links)
- Human motoneurone excitability is depressed by activation of serotonin 1A receptors with buspirone (Q48032494) (← links)
- Active dendritic integration of inhibitory synaptic inputs in vivo (Q48215283) (← links)
- Histamine modulates spinal motoneurons and locomotor circuits. (Q48346342) (← links)
- Intense Activity of the Raphe Spinal Pathway Depresses Motor Activity via a Serotonin Dependent Mechanism. (Q49392770) (← links)
- Synaptic amplification versus bistability in motoneuron dendritic processing: a top-down modeling approach (Q51031603) (← links)
- Motor Neurons (Q51089900) (← links)