Pages that link to "Q44212285"
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The following pages link to Lysosomal malfunction accompanies alpha-synuclein aggregation in a progressive mouse model of Parkinson's disease (Q44212285):
Displaying 50 items.
- Biomedical and clinical promises of human pluripotent stem cells for neurological disorders (Q21284798) (← links)
- Membrane Curvature Induction and Tubulation Are Common Features of Synucleins and Apolipoproteins (Q24633488) (← links)
- Evaluation of Models of Parkinson's Disease (Q26772843) (← links)
- Genetically engineered mouse models of Parkinson's disease (Q26852964) (← links)
- Protein degradation pathways in Parkinson's disease: curse or blessing (Q27026029) (← links)
- Gene-environment interactions: key to unraveling the mystery of Parkinson's disease (Q28388674) (← links)
- Resistance to MPTP-neurotoxicity in α-synuclein knockout mice is complemented by human α-synuclein and associated with increased β-synuclein and Akt activation (Q28477043) (← links)
- Degradative organelles containing mislocalized alpha-and beta-synuclein proliferate in presenilin-1 null neurons (Q28511388) (← links)
- Friedreich ataxia mouse models with progressive cerebellar and sensory ataxia reveal autophagic neurodegeneration in dorsal root ganglia (Q28593887) (← links)
- Aging, lipofuscin formation, and free radical-mediated inhibition of cellular proteolytic systems. (Q30336130) (← links)
- Identification of novel proteins affected by rotenone in mitochondria of dopaminergic cells (Q33294492) (← links)
- Beclin 1 gene transfer activates autophagy and ameliorates the neurodegenerative pathology in alpha-synuclein models of Parkinson's and Lewy body diseases (Q33613123) (← links)
- Selective molecular alterations in the autophagy pathway in patients with Lewy body disease and in models of alpha-synucleinopathy (Q33670282) (← links)
- Lysosomal function in macromolecular homeostasis and bioenergetics in Parkinson's disease (Q33843073) (← links)
- Mitochondrial dysfunction in the striatum of aged chronic mouse model of Parkinson's disease (Q33868426) (← links)
- Neuroprotective effect of long-term NDI1 gene expression in a chronic mouse model of Parkinson disorder (Q34129621) (← links)
- Differential effects of UCHL1 modulation on alpha-synuclein in PD-like models of alpha-synucleinopathy (Q34238665) (← links)
- Protocol for the MPTP mouse model of Parkinson's disease (Q34614573) (← links)
- Neuroprotective effects and mechanisms of exercise in a chronic mouse model of Parkinson's disease with moderate neurodegeneration (Q34812895) (← links)
- Melatonin protects against neurobehavioral and mitochondrial deficits in a chronic mouse model of Parkinson's disease (Q35165389) (← links)
- The role of alpha-synuclein in Parkinson's disease: insights from animal models (Q35209957) (← links)
- Molecular details of α-synuclein membrane association revealed by neutrons and photons. (Q35565655) (← links)
- Cycles of aberrant synaptic sprouting and neurodegeneration in Alzheimer's and dementia with Lewy bodies (Q35570834) (← links)
- Impact of exercise on mitochondrial transcription factor expression and damage in the striatum of a chronic mouse model of Parkinson's disease (Q35583125) (← links)
- Animal models of Parkinson's disease: a source of novel treatments and clues to the cause of the disease. (Q35589979) (← links)
- Autophagy in dementias (Q35624697) (← links)
- Proteomic approach to studying Parkinson's disease (Q35794541) (← links)
- Neuronal pathology in Parkinson's disease (Q35886715) (← links)
- The hypoxia imaging agent CuII(atsm) is neuroprotective and improves motor and cognitive functions in multiple animal models of Parkinson's disease (Q35894167) (← links)
- Differential degradation of motor deficits during gradual dopamine depletion with 6-hydroxydopamine in mice. (Q35922870) (← links)
- α-synuclein, LRRK2 and their interplay in Parkinson's disease. (Q35932488) (← links)
- Ageing as a primary risk factor for Parkinson's disease: evidence from studies of non-human primates (Q36070618) (← links)
- Endurance exercise promotes cardiorespiratory rehabilitation without neurorestoration in the chronic mouse model of parkinsonism with severe neurodegeneration. (Q36177056) (← links)
- A chronic mouse model of Parkinson's disease has a reduced gait pattern certainty (Q36248652) (← links)
- Macroautophagy in sporadic and the genetic form of Parkinson's disease with the A53T α-synuclein mutation (Q36424977) (← links)
- Microglial activation and antioxidant responses induced by the Parkinson's disease protein α-synuclein (Q36640417) (← links)
- Modeling PD pathogenesis in mice: advantages of a chronic MPTP protocol (Q36899298) (← links)
- Animal models of Parkinson's disease progression (Q36924624) (← links)
- α-Synuclein oligomers with broken helical conformation form lipoprotein nanoparticles (Q36929059) (← links)
- The role of autophagy-lysosome pathway in neurodegeneration associated with Parkinson's disease (Q37054679) (← links)
- alpha-Synuclein: a therapeutic target for Parkinson's disease? (Q37070916) (← links)
- Neuronal pigmented autophagic vacuoles: lipofuscin, neuromelanin, and ceroid as macroautophagic responses during aging and disease (Q37126849) (← links)
- Impaired glutamate homeostasis and programmed cell death in a chronic MPTP mouse model of Parkinson's disease (Q37310116) (← links)
- Membrane remodeling by α-synuclein and effects on amyloid formation. (Q37383252) (← links)
- Mitophagy and Parkinson's disease: the PINK1-parkin link. (Q37587126) (← links)
- Pathophysiology of neuropathic lysosomal storage disorders (Q37739318) (← links)
- MPTP mouse models of Parkinson's disease: an update (Q38070400) (← links)
- Models of α-synuclein aggregation in Parkinson's disease (Q38286574) (← links)
- Therapeutic strategies in Parkinson's disease: what we have learned from animal models (Q38290759) (← links)
- Mild MPP+ exposure impairs autophagic degradation through a novel lysosomal acidity-independent mechanism (Q38764175) (← links)