Pages that link to "Q43591891"

The following pages link to Contribution of intralaminar thalamic nuclei to spike-and-wave-discharges during spontaneous seizures in a genetic rat model of absence epilepsy (Q43591891):

Displaying 25 items.

• Cortical control of zona incerta (Q28288252) (← links)
• CNF1 Enhances Brain Energy Content and Counteracts Spontaneous Epileptiform Phenomena in Aged DBA/2J Mice (Q30400926) (← links)
• Childhood absence epilepsy: genes, channels, neurons and networks. (Q30835420) (← links)
• Cellular and network mechanisms of spike-wave seizures (Q33227648) (← links)
• Thalamocortical oscillations in a genetic model of absence seizures. (Q34166027) (← links)
• Impact of network activities on neuronal properties in corticothalamic systems (Q34296211) (← links)
• Cellular and network mechanisms of genetically-determined absence seizures. (Q35199746) (← links)
• Thalamocortical neurons display suppressed burst-firing due to an enhanced Ih current in a genetic model of absence epilepsy (Q35619054) (← links)
• Unilateral and Bilateral Cortical Resection: Effects on Spike-Wave Discharges in a Genetic Absence Epilepsy Model (Q35742532) (← links)
• Disinhibition-induced transitions between absence and tonic-clonic epileptic seizures (Q35902032) (← links)
• The liabilities of mobility: a selection pressure for the transition to consciousness in animal evolution (Q36070760) (← links)
• Frequency-selective control of cortical and subcortical networks by central thalamus (Q36488056) (← links)
• Lack of effect of intranigral transplants of a GABAergic cell line on absence seizures (Q39679686) (← links)
• Absence epilepsy and sinus dysrhythmia in mice lacking the pacemaker channel HCN2 (Q39696685) (← links)
• Beta and gamma range EEG power-spectrum correlation with spiking discharges in DBA/2J mice absence model: role of GABA receptors. (Q39748232) (← links)
• Evidence for a role of the parafascicular nucleus of the thalamus in the control of epileptic seizures by the superior colliculus. (Q42138475) (← links)
• Involvement of the thalamic parafascicular nucleus in mesial temporal lobe epilepsy (Q42481040) (← links)
• Enhancement of anti-absence effects of ethosuximide by low doses of a noncompetitive alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid (AMPA) receptor antagonist in a genetic animal model of absence epilepsy (Q46565823) (← links)
• Impaired regulation of thalamic pacemaker channels through an imbalance of subunit expression in absence epilepsy. (Q46776142) (← links)
• Neo-Timm staining in the thalamus of chronically epileptic rats (Q46782304) (← links)
• Descending projections to the inferior colliculus from the posterior thalamus and the auditory cortex in rat, cat, and monkey (Q47436781) (← links)
• Corticothalamic 5-9 Hz oscillations are more pro-epileptogenic than sleep spindles in rats (Q48504286) (← links)
• Activity of thalamic reticular neurons during spontaneous genetically determined spike and wave discharges. (Q48661080) (← links)
• Diversity of structure and function of GABAB receptors: a complexity of GABAB-mediated signaling (Q90438743) (← links)
• Multiunit cluster firing patterns of piriform cortex and mediodorsal thalamus in absence epilepsy (Q93122884) (← links)