Pages that link to "Q43049804"
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The following pages link to Inhibition of the RAGE products increases survival in experimental models of severe sepsis and systemic infection (Q43049804):
Displaying 50 items.
- Receptor for advanced glycation end products and its involvement in inflammatory diseases (Q21285070) (← links)
- Targeting HMGB1 in inflammation (Q28266138) (← links)
- Receptor for advanced glycation end products (RAGE) on iNKT cells mediates lung ischemia-reperfusion injury (Q30410928) (← links)
- Deletion of RAGE causes hyperactivity and increased sensitivity to auditory stimuli in mice (Q30484598) (← links)
- Alarmins: awaiting a clinical response (Q30523797) (← links)
- Soluble receptor for advanced glycation end products predicts 28-day mortality in critically ill patients with sepsis (Q33709127) (← links)
- The RAGE axis: a fundamental mechanism signaling danger to the vulnerable vasculature (Q33822512) (← links)
- The role of proteomics in the diagnosis of chorioamnionitis and early-onset neonatal sepsis (Q33940421) (← links)
- Anti-receptor for advanced glycation end products therapies as novel treatment for abdominal aortic aneurysm (Q34067128) (← links)
- Soluble RAGE: therapy and biomarker in unraveling the RAGE axis in chronic disease and aging (Q34094369) (← links)
- Anti-RAGE antibody ameliorates severe thermal injury in rats through regulating cellular immune function (Q34138346) (← links)
- High-mobility group protein box-1 and its relevance to cerebral ischemia (Q34170847) (← links)
- Heterogeneous phenotypes of acute respiratory distress syndrome after major trauma. (Q34479261) (← links)
- Paradoxical function for the receptor for advanced glycation end products in mouse models of pulmonary fibrosis (Q34766912) (← links)
- The effect of bortezomib on expression of inflammatory cytokines and survival in a murine sepsis model induced by cecal ligation and puncture (Q34774088) (← links)
- Xuebijing Ameliorates Sepsis-Induced Lung Injury by Downregulating HMGB1 and RAGE Expressions in Mice (Q35188008) (← links)
- Receptor for Advanced Glycation End Products (RAGE) Serves a Protective Role during Klebsiella pneumoniae - Induced Pneumonia (Q35908548) (← links)
- Ligands of the receptor for advanced glycation end products, including high-mobility group box 1, limit bacterial dissemination during Escherichia coli peritonitis (Q35944988) (← links)
- HMGB1 Is a Therapeutic Target for Sterile Inflammation and Infection (Q35953553) (← links)
- sRAGE in diabetic and non-diabetic critically ill patients: effects of intensive insulin therapy (Q36070548) (← links)
- RAGE Deficiency Impairs Bacterial Clearance in Murine Staphylococcal Sepsis, but Has No Significant Impact on Staphylococcal Septic Arthritis (Q36209840) (← links)
- Effects of bone marrow MSCs transfected with sRAGE on the intervention of HMGB1 induced immuno-inflammatory reaction (Q36375004) (← links)
- Identification of haplotype tag single nucleotide polymorphisms within the receptor for advanced glycation end products gene and their clinical relevance in patients with major trauma (Q36633916) (← links)
- RAGE: exacting a toll on the host in response to polymicrobial sepsis and Listeria monocytogenes (Q37055335) (← links)
- High-mobility group box protein 1 (HMGB1): an alarmin mediating the pathogenesis of rheumatic disease (Q37206315) (← links)
- The receptor RAGE: Bridging inflammation and cancer (Q37212720) (← links)
- Sepsis: links between pathogen sensing and organ damage (Q37239817) (← links)
- Senescence-dependent impact of anti-RAGE antibody on endotoxemic liver failure (Q37300540) (← links)
- Characterization of RAGE, HMGB1, and S100beta in inflammation-induced preterm birth and fetal tissue injury (Q37316463) (← links)
- AGE-modified albumin containing infusion solutions boosts septicaemia and inflammation in experimental peritonitis. (Q37326771) (← links)
- Receptor for AGE (RAGE) and its ligands-cast into leading roles in diabetes and the inflammatory response (Q37384331) (← links)
- Tempering the wrath of RAGE: an emerging therapeutic strategy against diabetic complications, neurodegeneration, and inflammation (Q37424029) (← links)
- The alarmin functions of high-mobility group proteins. (Q37685453) (← links)
- High-mobility-group box chromosomal protein 1 as a new target for modulating stress response (Q37768056) (← links)
- The RAGE axis in systemic inflammation, acute lung injury and myocardial dysfunction: an important therapeutic target? (Q37772786) (← links)
- Cecal ligation and puncture: the gold standard model for polymicrobial sepsis? (Q37836156) (← links)
- The role of receptor for advanced glycation endproducts (RAGE) in infection (Q37860412) (← links)
- Toll-like receptor-4 antagonist eritoran tetrasodium for severe sepsis (Q37879190) (← links)
- Immunomodulatory therapy for sepsis: an update (Q37949639) (← links)
- Receptor for AGE (RAGE): signaling mechanisms in the pathogenesis of diabetes and its complications (Q37972674) (← links)
- The receptor for advanced glycation end products and acute lung injury/acute respiratory distress syndrome (Q38025026) (← links)
- Cardiovascular disease and high-mobility group box 1--is a new inflammatory killer in town? (Q38040046) (← links)
- Gut mucosal DAMPs in IBD: from mechanisms to therapeutic implications (Q38756562) (← links)
- HMGB1, IL-1α, IL-33 and S100 proteins: dual-function alarmins (Q38939435) (← links)
- Tissue advanced glycation end products (AGEs), measured by skin autofluorescence, predict mortality in peritoneal dialysis (Q39094081) (← links)
- Structural basis for pattern recognition by the receptor for advanced glycation end products (RAGE). (Q39955806) (← links)
- The impact of RAGE inhibition in animal models of bacterial sepsis: a systematic review and meta-analysis (Q40112947) (← links)
- RAGE deficiency attenuates the protective effect of Lidocaine against sepsis-induced acute lung injury (Q40334113) (← links)
- RAGE-Mediated Suppression of Interleukin-10 Results in Enhanced Mortality in a Murine Model of Acinetobacter baumannii Sepsis (Q40385373) (← links)
- The promoter polymorphisms of receptor for advanced glycation end products were associated with the susceptibility and progression of sepsis (Q40677347) (← links)