Pages that link to "Q42988167"

The following pages link to Cholecystokinin-stimulated tyrosine phosphorylation of p125FAK and paxillin is mediated by phospholipase C-dependent and -independent mechanisms and requires the integrity of the actin cytoskeleton and participation of p21rho (Q42988167):

Displaying 37 items.

• Signaling through focal adhesion kinase (Q28198051) (← links)
• Alpha4 integrins and the immune response (Q28202046) (← links)
• Gastrointestinal hormones cause rapid c-Met receptor down-regulation by a novel mechanism involving clathrin-mediated endocytosis and a lysosome-dependent mechanism (Q28570307) (← links)
• Adapter protein CRKII signaling is involved in the rat pancreatic acini response to reactive oxygen species (Q28582518) (← links)
• CCK1 and 2 receptors are expressed in immortalized rat brain neuroblasts: intracellular signals after cholecystokinin stimulation (Q28582970) (← links)
• Regulation of adherens junction protein p120(ctn) by 10 nM CCK precedes actin breakdown in rat pancreatic acini. (Q30845418) (← links)
• Hormonal control of cardiac ion channels and transporters (Q33710653) (← links)
• Gastrointestinal hormones/neurotransmitters and growth factors can activate P21 activated kinase 2 in pancreatic acinar cells by novel mechanisms (Q33809148) (← links)
• Regulation of signaling by protein-tyrosine phosphatases: potential roles in the nervous system (Q33844146) (← links)
• Alpha4 integrins in cardiovascular development and diseases (Q34204749) (← links)
• The paradox of smooth muscle physiology (Q34264530) (← links)
• Ability of various bombesin receptor agonists and antagonists to alter intracellular signaling of the human orphan receptor BRS-3. (Q34468601) (← links)
• Elucidation of the roles of the Src kinases in pancreatic acinar cell signaling (Q34498518) (← links)
• PKCθ activation in pancreatic acinar cells by gastrointestinal hormones/neurotransmitters and growth factors is needed for stimulation of numerous important cellular signaling cascades (Q35552713) (← links)
• The Src kinase Yes is activated in pancreatic acinar cells by gastrointestinal hormones/neurotransmitters, but not pancreatic growth factors, which stimulate its association with numerous other signaling molecules (Q36115139) (← links)
• The p21-activated kinase, PAK2, is important in the activation of numerous pancreatic acinar cell signaling cascades and in the onset of early pancreatitis events (Q36840241) (← links)
• Src kinases play a novel dual role in acute pancreatitis affecting severity but no role in stimulated enzyme secretion (Q37071766) (← links)
• Gastrointestinal growth factors and hormones have divergent effects on Akt activation. (Q37180159) (← links)
• Pervanadate stimulates amylase release and protein tyrosine phosphorylation of paxillin and p125(FAK) in differentiated AR4-2J pancreatic acinar cells (Q41032613) (← links)
• Activation of tyrosine kinases by alpha1A-adrenergic and growth factor receptors in transfected PC12 cells (Q41882896) (← links)
• Tyrosine kinases activate store-mediated Ca2+ entry in human platelets through the reorganization of the actin cytoskeleton. (Q42136904) (← links)
• Evidence for secretion-like coupling involving pp60src in the activation and maintenance of store-mediated Ca2+ entry in mouse pancreatic acinar cells. (Q42157369) (← links)
• Cholecystokinin-stimulated protein kinase C-delta kinase activation, tyrosine phosphorylation, and translocation are mediated by Src tyrosine kinases in pancreatic acinar cells (Q42169406) (← links)
• Phosphospecific site tyrosine phosphorylation of p125FAK and proline-rich kinase 2 is differentially regulated by cholecystokinin receptor type A activation in pancreatic acini (Q42438652) (← links)
• Cholecystokinin activates PYK2/CAKbeta by a phospholipase C-dependent mechanism and its association with the mitogen-activated protein kinase signaling pathway in pancreatic acinar cells (Q42479060) (← links)
• CCK-A receptor activates RhoA through G alpha 12/13 in NIH3T3 cells (Q42799565) (← links)
• Neuromedin B Receptor Activation Causes Tyrosine Phosphorylation of p125FAK by a Phospholipase C Independent Mechanism Which Requires p21rho and Integrity of the Actin Cytoskeleton (Q42833080) (← links)
• Depletion of focal adhesion kinase by antisense depresses contractile activation of smooth muscle (Q43540351) (← links)
• Glutamate induces focal adhesion kinase tyrosine phosphorylation and actin rearrangement in heterologous mGluR1-expressing CHO cells via calcium/calmodulin signaling (Q43681027) (← links)
• Effects of reactive oxygen species on actin filament polymerisation and amylase secretion in mouse pancreatic acinar cells. (Q43919850) (← links)
• Cholecystokinin-stimulated tyrosine phosphorylation of PKC-delta in pancreatic acinar cells is regulated bidirectionally by PKC activation. (Q44216459) (← links)
• The effect of lysophosphatidic acid and Rho-associated kinase patterning on adhesion of dental pulp cells (Q44932495) (← links)
• P21 activated kinase4 in pancreatic acini is activated by GI hormones /growth factors by novel signaling and is needed to stimulate secretory/growth cascades. (Q52578169) (← links)
• A role for phosphoinositides in tyrosine phosphorylation of p125 focal adhesion kinase in rat pancreatic acini (Q73515628) (← links)
• Are tyrosine phosphorylation of p125(FAK) and paxillin or the small GTP binding protein, rho, needed for CCK-stimulated pancreatic amylase secretion? (Q77305194) (← links)
• EGF stimulates tyrosine phosphorylation of focal adhesion kinase (p125FAK) and paxillin in rat pancreatic acini by a phospholipase C-independent process that depends on phosphatidylinositol 3-kinase, the small GTP-binding protein, p21rho, and the in (Q78173207) (← links)
• Cholecystokinin (CCK) Regulation of Pancreatic Acinar Cells: Physiological Actions and Signal Transduction Mechanisms (Q92378335) (← links)