Pages that link to "Q42562755"
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The following pages link to Paolo Mignatti (Q42562755):
Displaying 50 items.
- Infantile Hemangioma Originates From A Dysregulated But Not Fully Transformed Multipotent Stem Cell. (Q30826592) (← links)
- Nonenzymatic interactions between proteinases and the cell surface: novel roles in normal and malignant cell physiology. (Q33765841) (← links)
- Control of type IV collagenase activity by components of the urokinase-plasmin system: a regulatory mechanism with cell-bound reactants. (Q33886514) (← links)
- Membrane-Type 1 Matrix Metalloproteinase Downregulates Fibroblast Growth Factor-2 Binding to the Cell Surface and Intracellular Signaling. (Q34799472) (← links)
- TIMP-2 Interaction with MT1-MMP Activates the AKT Pathway and Protects Tumor Cells from Apoptosis (Q35762042) (← links)
- Urokinase Receptor Promotes Skin Tumor Formation by Preventing Epithelial Cell Activation of Notch1. (Q35829976) (← links)
- Elevated expression of periostin in human osteoarthritic cartilage and its potential role in matrix degradation via matrix metalloproteinase-13. (Q36051493) (← links)
- In vitro angiogenesis on the human amniotic membrane: requirement for basic fibroblast growth factor-induced proteinases. (Q36220034) (← links)
- Differential modulation of cell phenotype by different molecular weight forms of basic fibroblast growth factor: possible intracellular signaling by the high molecular weight forms. (Q36235250) (← links)
- Expression of the urokinase receptor in vascular endothelial cells is stimulated by basic fibroblast growth factor (Q36529792) (← links)
- Growth factor control of extracellular proteolysis (Q36659989) (← links)
- Basic fibroblast growth factor (FGF-2): the high molecular weight forms come of age. (Q36666638) (← links)
- Release of basic fibroblast growth factor, an angiogenic factor devoid of secretory signal sequence: A trivial phenomenon or a novel secretion mechanism? (Q36715068) (← links)
- Vascular injury and modulation of MAPKs: a targeted approach to therapy of restenosis. (Q36797552) (← links)
- Inhibition of smooth muscle cell migration and neointima formation in vein grafts by overexpression of matrix metalloproteinase-3. (Q37276960) (← links)
- Basic fibroblast growth factor released by single, isolated cells stimulates their migration in an autocrine manner (Q37639452) (← links)
- Mitral valve prolapse is associated with altered extracellular matrix gene expression patterns. (Q38448735) (← links)
- Inhibition of Breast Cancer Metastasis by Presurgical Treatment with an Oral Matrix Metalloproteinase Inhibitor: A Preclinical Proof-of-Principle Study. (Q38755272) (← links)
- Arrested Development: Infantile Hemangioma and the Stem Cell Teratogenic Hypothesis (Q40196950) (← links)
- Shedding of membrane vesicles mediates fibroblast growth factor-2 release from cells (Q40629439) (← links)
- Biology and biochemistry of proteinases in tumor invasion. (Q40868702) (← links)
- Plasminogen activators and angiogenesis. (Q41117126) (← links)
- TGF-beta1 induces rearrangement of FLK-1-VE-cadherin-beta-catenin complex at the adherens junction through VEGF-mediated signaling. (Q41593699) (← links)
- Basic fibroblast growth factor-induced activation of latent transforming growth factor beta in endothelial cells: regulation of plasminogen activator activity (Q41984680) (← links)
- TGF-β1 induces endothelial cell apoptosis by shifting VEGF activation of p38(MAPK) from the prosurvival p38β to proapoptotic p38α. (Q42175314) (← links)
- Transforming growth factor-beta 1 (TGF-beta1) induces angiogenesis through vascular endothelial growth factor (VEGF)-mediated apoptosis (Q42562736) (← links)
- Basic fibroblast growth factor, a protein devoid of secretory signal sequence, is released by cells via a pathway independent of the endoplasmic reticulum-Golgi complex (Q42820335) (← links)
- New observations on the intracellular localization and release of bFGF. (Q42830014) (← links)
- Thrombin cleaves the high molecular weight forms of basic fibroblast growth factor (FGF-2): a novel mechanism for the control of FGF-2 and thrombin activity (Q43068693) (← links)
- Activation of progelatinase A (MMP-2) by neutrophil elastase, cathepsin G, and proteinase-3: a role for inflammatory cells in tumor invasion and angiogenesis (Q43765113) (← links)
- Activation of mitogen-activated protein kinases during preparation of vein grafts and modulation by a synthetic inhibitor. (Q44591584) (← links)
- Modulation of matrix metalloproteinase activity in human saphenous vein grafts using adenovirus-mediated gene transfer (Q44729484) (← links)
- Vein graft arterialization causes differential activation of mitogen-activated protein kinases. (Q44872730) (← links)
- PDGF-BB induces vascular smooth muscle cell expression of high molecular weight FGF-2, which accumulates in the nucleus (Q47378034) (← links)
- Soluble factor(s) released from neutrophils activates endothelial cell matrix metalloproteinase-2. (Q47721840) (← links)
- Urokinase plasminogen activator and gelatinases are associated with membrane vesicles shed by human HT1080 fibrosarcoma cells. (Q52193484) (← links)
- Topical mitogen-activated protein kinases inhibition reduces intimal hyperplasia in arterialized vein grafts. (Q53452563) (← links)
- Tumor cells secrete an angiogenic factor that stimulates basic fibroblast growth factor and urokinase expression in vascular endothelial cells. (Q54194832) (← links)
- Mechanisms of c-reactive protein up-regulation in arterialized vein grafts. (Q54619598) (← links)
- Plasmin activates pro-matrix metalloproteinase-2 with a membrane-type 1 matrix metalloproteinase-dependent mechanism (Q59571861) (← links)
- Matrix Metalloproteinase Inhibitors in Cancer Therapy: Turning Past Failures Into Future Successes (Q60681786) (← links)
- A quantitative in vitro model of smooth muscle cell migration through the arterial wall using the human amniotic membrane (Q64377843) (← links)
- The role of proteases in matrix breakdown during cellular invasion (Q69547639) (← links)
- Purification and biological activities of an angiogenesis factor from human placenta (Q70147413) (← links)
- Tumor invasion through the human amniotic membrane: requirement for a proteinase cascade (Q70153486) (← links)
- Tumor cell-conditioned medium stimulates expression of the urokinase receptor in vascular endothelial cells (Q71758659) (← links)
- Mechanism of action of angiostatic steroids: suppression of plasminogen activator activity via stimulation of plasminogen activator inhibitor synthesis (Q72080130) (← links)
- Vascular endothelial growth factor increases urokinase receptor expression in vascular endothelial cells (Q72191064) (← links)
- Plasminogen activators, activation inhibitors and alpha 2-macroglobulin produced by cultured normal and malignant human cells (Q72733242) (← links)
- Expression of Von Willebrand factor, an endothelial cell marker, is up-regulated by angiogenesis factors: a potential method for objective assessment of tumor angiogenesis (Q73340903) (← links)