Pages that link to "Q42435249"

The following pages link to Cloning of rat homologue of podocin: expression in proteinuric states and in developing glomeruli (Q42435249):

Displaying 47 items.

• Podocin participates in the assembly of tight junctions between foot processes in nephrotic podocytes (Q24336289) (← links)
• Slit diaphragms contain tight junction proteins (Q28573180) (← links)
• Common variation in GPC5 is associated with acquired nephrotic syndrome (Q28943292) (← links)
• Loss of the BMP antagonist USAG-1 ameliorates disease in a mouse model of the progressive hereditary kidney disease Alport syndrome. (Q33685580) (← links)
• NPHS2 (Podocin) mutations in nephrotic syndrome. Clinical spectrum and fine mechanisms (Q34409698) (← links)
• A novel domain regulating degradation of the glomerular slit diaphragm protein podocin in cell culture systems (Q34600013) (← links)
• Simvastatin reverses podocyte injury but not mesangial expansion in early stage type 2 diabetes mellitus (Q35098290) (← links)
• Marker expression, behaviors, and responses vary in different lines of conditionally immortalized cultured podocytes (Q35216791) (← links)
• GLUT1 enhances mTOR activity independently of TSC2 and AMPK. (Q35216825) (← links)
• Angiotensin II type 1 and type 2 receptors play opposite roles in regulating the barrier function of kidney glomerular capillary wall (Q35855619) (← links)
• New insights into the pathogenesis and the therapy of recurrent focal glomerulosclerosis. (Q36124137) (← links)
• Candesartan Mediated Amelioration of Cisplatin-Induced Testicular Damage Is Associated with Alterations in Expression Patterns of Nephrin and Podocin (Q36204071) (← links)
• Genetics of idiopathic nephrotic syndrome (Q36269119) (← links)
• Role of podocyte slit diaphragm as a filtration barrier (Q36556701) (← links)
• Podocin-related mechanisms in posttransplant [corrected] recurrence of focal segmental glomerulosclerosis [corrected] (Q36686723) (← links)
• Avian Podocytes, Which Lack Nephrin, Use Adherens Junction Proteins at Intercellular Junctions (Q36737090) (← links)
• Role of calcineurin (CN) in kidney glomerular podocyte: CN inhibitor ameliorated proteinuria by inhibiting the redistribution of CN at the slit diaphragm. (Q36748296) (← links)
• Inducible rodent models of acquired podocyte diseases. (Q37265903) (← links)
• Notoginsenoside R1 ameliorates podocyte injury in rats with diabetic nephropathy by activating the PI3K/Akt signaling pathway (Q37269951) (← links)
• Slit diaphragm dysfunction in proteinuric states: identification of novel therapeutic targets for nephrotic syndrome (Q37407881) (← links)
• Notoginsenoside R1 attenuates glucose-induced podocyte injury via the inhibition of apoptosis and the activation of autophagy through the PI3K/Akt/mTOR signaling pathway (Q37713980) (← links)
• Therapeutic target for nephrotic syndrome: Identification of novel slit diaphragm associated molecules (Q38261675) (← links)
• Semiautomated quantitative image analysis of glomerular immunohistochemistry markers desmin, vimentin, podocin, synaptopodin and WT-1 in acute and chronic rat kidney disease models (Q38931612) (← links)
• Opposing effects of podocin on the gating of podocyte TRPC6 channels evoked by membrane stretch or diacylglycerol (Q39154682) (← links)
• Ameliorating Effects of l-Carnitine on Diabetic Podocyte Injury (Q39644251) (← links)
• The reno-protective effect of a phosphoinositide 3-kinase inhibitor wortmannin on streptozotocin-induced proteinuric renal disease rats (Q41050409) (← links)
• Neurexin-1, a presynaptic adhesion molecule, localizes at the slit diaphragm of the glomerular podocytes in kidneys (Q41166606) (← links)
• Impact of Cyclin B2 and Cell division cycle 2 on tubular hyperplasia in progressive chronic renal failure rats (Q41996628) (← links)
• Claudin-6 localized in tight junctions of rat podocytes (Q42526089) (← links)
• Ultrastructural study on nephrin expression in experimental puromycin aminonucleoside nephrosis. (Q45072879) (← links)
• Dissociation of NEPH1 from nephrin is involved in development of a rat model of focal segmental glomerulosclerosis (Q45176454) (← links)
• Tolvaptan, a selective oral vasopressin V2 receptor antagonist, ameliorates podocyte injury in puromycin aminonucleoside nephrotic rats. (Q46006392) (← links)
• Effect of a novel free radical scavenger, edaravone, on puromycin aminonucleoside induced nephrosis in rats (Q46634254) (← links)
• Altered expression of junctional adhesion molecule 4 in injured podocytes (Q46666054) (← links)
• MAGI-1 is a component of the glomerular slit diaphragm that is tightly associated with nephrin (Q46697366) (← links)
• Decreased tyrosine phosphorylation of nephrin in rat and human nephrosis (Q46767425) (← links)
• Doxorubicin-induced glomerulosclerosis with proteinuria in GFP-GABARAP transgenic mice (Q50508580) (← links)
• Hsf-1 affects podocyte markers NPHS1, NPHS2 and WT1 in a transgenic mouse model of TTRVal30Met-related amyloidosis (Q50856561) (← links)
• Possible role for glomerular-derived angiotensinogen in nephrotic syndrome (Q51278033) (← links)
• Nephrin-Binding Ephrin-B1 at the Slit Diaphragm Controls Podocyte Function through the JNK Pathway. (Q51730421) (← links)
• Alteration in the podoplanin-ezrin-cytoskeleton linkage is an important initiation event of the podocyte injury in puromycin aminonucleoside nephropathy, a mimic of minimal change nephrotic syndrome. (Q53526441) (← links)
• Podocyte protection by darbepoetin: preservation of the cytoskeleton and nephrin expression. (Q53557373) (← links)
• Rituximab protects podocytes and exerts anti-proteinuric effects in rat adriamycin-induced nephropathy independent of B-lymphocytes. (Q53637511) (← links)
• Heparin increasing podocyte-specific gene expressions. (Q54227574) (← links)
• Angiotensin II type 1 receptor blockade ameliorates proteinuria in puromycin aminonucleoside nephropathy by inhibiting the reduction of NEPH1 and nephrin (Q85723580) (← links)
• SV2B is essential for the integrity of the glomerular filtration barrier (Q86852377) (← links)
• New insight into podocyte slit diaphragm, a therapeutic target of proteinuria (Q89533672) (← links)