Pages that link to "Q42169958"
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The following pages link to Distinct repriming and closed-state inactivation kinetics of Nav1.6 and Nav1.7 sodium channels in mouse spinal sensory neurons (Q42169958):
Displaying 50 items.
- Temperature dependence of erythromelalgia mutation L858F in sodium channel Nav1.7 (Q21203947) (← links)
- Fibroblast growth factor homologous factor 2B: association with Nav1.6 and selective colocalization at nodes of Ranvier of dorsal root axons (Q24300238) (← links)
- Where is the spike generator of the cochlear nerve? Voltage-gated sodium channels in the mouse cochlea. (Q24540329) (← links)
- A single sodium channel mutation produces hyper- or hypoexcitability in different types of neurons (Q24546007) (← links)
- Mechanisms of sodium channel clustering and its influence on axonal impulse conduction (Q26777940) (← links)
- Primary erythromelalgia: a review (Q26784432) (← links)
- The perception and endogenous modulation of pain (Q27011310) (← links)
- Structural Basis for the Modulation of the Neuronal Voltage-Gated Sodium Channel NaV1.6 by Calmodulin (Q27679551) (← links)
- De novo pathogenic SCN8A mutation identified by whole-genome sequencing of a family quartet affected by infantile epileptic encephalopathy and SUDEP (Q28111582) (← links)
- An SCN9A channelopathy causes congenital inability to experience pain (Q28278844) (← links)
- Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain (Q28542818) (← links)
- Interaction of voltage-gated sodium channel Nav1.6 (SCN8A) with microtubule-associated protein Map1b (Q28587249) (← links)
- Fibroblast growth factor homologous factors control neuronal excitability through modulation of voltage-gated sodium channels (Q28588873) (← links)
- Subtype-Selective Small Molecule Inhibitors Reveal a Fundamental Role for Nav1.7 in Nociceptor Electrogenesis, Axonal Conduction and Presynaptic Release (Q28601095) (← links)
- Pathogenic mechanism of recurrent mutations of SCN8A in epileptic encephalopathy (Q29347550) (← links)
- The SCN8A encephalopathy mutation p.Ile1327Val displays elevated sensitivity to the anticonvulsant phenytoin (Q30276427) (← links)
- Parallel evolution of tetrodotoxin resistance in three voltage-gated sodium channel genes in the garter snake Thamnophis sirtalis (Q30409940) (← links)
- Computation identifies structural features that govern neuronal firing properties in slowly adapting touch receptors (Q30415180) (← links)
- Ototrauma induces sodium channel plasticity in auditory afferent neurons (Q30473859) (← links)
- The ataxia3 mutation in the N-terminal cytoplasmic domain of sodium channel Na(v)1.6 disrupts intracellular trafficking (Q30487520) (← links)
- Pharmacological characterisation of the highly NaV1.7 selective spider venom peptide Pn3a (Q30835612) (← links)
- Excitability constraints on voltage-gated sodium channels (Q33300128) (← links)
- FGF14 N-terminal splice variants differentially modulate Nav1.2 and Nav1.6-encoded sodium channels (Q33705386) (← links)
- Brain tissue sodium concentration in multiple sclerosis: a sodium imaging study at 3 tesla (Q33742402) (← links)
- Alternative splicing of Na(V)1.7 exon 5 increases the impact of the painful PEPD mutant channel I1461T. (Q33796526) (← links)
- Mutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na V 1.7 produce distinct pain disorders (Q33875626) (← links)
- Isolation and characterization of CvIV4: a pain inducing α-scorpion toxin (Q34009359) (← links)
- Two Nedd4-binding motifs underlie modulation of sodium channel Nav1.6 by p38 MAPK. (Q34074334) (← links)
- Divergent actions of the pyrethroid insecticides S-bioallethrin, tefluthrin, and deltamethrin on rat Na(v)1.6 sodium channels (Q34089504) (← links)
- Congenital insensitivity to pain: novel SCN9A missense and in-frame deletion mutations (Q34254861) (← links)
- Molecular changes in neurons in multiple sclerosis: altered axonal expression of Nav1.2 and Nav1.6 sodium channels and Na+/Ca2+ exchanger (Q34321453) (← links)
- Electrophysiological properties of mutant Nav1.7 sodium channels in a painful inherited neuropathy. (Q34350866) (← links)
- Single-cell analysis of sodium channel expression in dorsal root ganglion neurons (Q34422391) (← links)
- Voltage-gated sodium channel Nav1.6 is modulated by p38 mitogen-activated protein kinase. (Q34433609) (← links)
- Calmodulin regulates current density and frequency-dependent inhibition of sodium channel Nav1.8 in DRG neurons (Q34511133) (← links)
- Resurgent current and voltage sensor trapping enhanced activation by a beta-scorpion toxin solely in Nav1.6 channel. Significance in mice Purkinje neurons. (Q34526433) (← links)
- Ankyrin-G regulates inactivation gating of the neuronal sodium channel, Nav1.6. (Q34538288) (← links)
- Size matters: Erythromelalgia mutation S241T in Nav1.7 alters channel gating (Q34569783) (← links)
- Na(V)1.7 mutant A863P in erythromelalgia: effects of altered activation and steady-state inactivation on excitability of nociceptive dorsal root ganglion neurons. (Q34585853) (← links)
- The roles of sodium channels in nociception: Implications for mechanisms of pain (Q34674268) (← links)
- Expansion of voltage-dependent Na+ channel gene family in early tetrapods coincided with the emergence of terrestriality and increased brain complexity (Q34683283) (← links)
- Nav1.7 is the predominant sodium channel in rodent olfactory sensory neurons (Q35003780) (← links)
- Activation of peripheral nerve fibers by electrical stimulation in the sole of the foot (Q35011119) (← links)
- Beyond faithful conduction: short-term dynamics, neuromodulation, and long-term regulation of spike propagation in the axon (Q35166946) (← links)
- Preferential targeting of Nav1.6 voltage-gated Na+ Channels to the axon initial segment during development (Q35432154) (← links)
- Polarized domains of myelinated axons. (Q35558428) (← links)
- Inactivation properties of sodium channel Nav1.8 maintain action potential amplitude in small DRG neurons in the context of depolarization (Q35842315) (← links)
- Novel sodium channel antagonists in the treatment of neuropathic pain. (Q35843983) (← links)
- Presynaptic Na+ channels: locus, development, and recovery from inactivation at a high-fidelity synapse (Q35878912) (← links)
- Cell signaling and the genesis of neuropathic pain (Q35904210) (← links)