Pages that link to "Q41179543"
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The following pages link to Discovery of a metabolic pathway mediating glucose-induced desensitization of the glucose transport system. Role of hexosamine biosynthesis in the induction of insulin resistance (Q41179543):
Displaying 50 items.
- Glucosamine for osteoarthritis: biological effects, clinical efficacy, and safety on glucose metabolism (Q21284705) (← links)
- Elevated O-GlcNAc-dependent signaling through inducible mOGT expression selectively triggers apoptosis (Q24299073) (← links)
- Hexosamines, insulin resistance, and the complications of diabetes: current status (Q24538581) (← links)
- Caenorhabditis elegans ortholog of a diabetes susceptibility locus: oga-1 (O-GlcNAcase) knockout impacts O-GlcNAc cycling, metabolism, and dauer (Q24670972) (← links)
- Effects of oral glucosamine sulphate on serum glucose and insulin during an oral glucose tolerance test of subjects with osteoarthritis (Q24674885) (← links)
- Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation (Q24682735) (← links)
- Distinct effects of glucose and glucosamine on vascular endothelial and smooth muscle cells: evidence for a protective role for glucosamine in atherosclerosis (Q24813839) (← links)
- Hijacking the Hexosamine Biosynthetic Pathway to Promote EMT-Mediated Neoplastic Phenotypes (Q26752391) (← links)
- O-GlcNAcylation and the Metabolic Shift in High-Proliferating Cells: All the Evidence Suggests that Sugars Dictate the Flux of Lipid Biogenesis in Tumor Processes (Q26770270) (← links)
- Insulin receptor signaling in normal and insulin-resistant states (Q26823171) (← links)
- The role of glucosamine-induced ER stress in diabetic atherogenesis (Q26830592) (← links)
- FOXO transcription factors throughout T cell biology (Q26853741) (← links)
- Functional O-GlcNAc modifications: implications in molecular regulation and pathophysiology (Q27009399) (← links)
- Cancer cell metabolism: implications for therapeutic targets (Q27013632) (← links)
- Glycerol-3-phosphate acyltransferase-1 upregulation by O-GlcNAcylation of Sp1 protects against hypoxia-induced mouse embryonic stem cell apoptosis via mTOR activation (Q27329592) (← links)
- Inhibition of O-GlcNAcase Using a Potent and Cell-Permeable Inhibitor Does Not Induce Insulin Resistance in 3T3-L1 Adipocytes (Q27664569) (← links)
- Glutamine:fructose-6-phosphate aminotransferase enzyme activity is necessary for the induction of TGF-beta1 and fibronectin expression in mesangial cells (Q28178641) (← links)
- Physical mapping of the human glutamine:fructose-6-phosphate amidotransferase gene (GFPT) to chromosome 2p13 (Q28292668) (← links)
- Human glutamine: fructose-6-phosphate amidotransferase: characterization of mRNA and chromosomal assignment to 2p13 (Q28293184) (← links)
- Regulation of cancer cell metabolism (Q28303890) (← links)
- Glucose and glucosamine regulate growth factor gene expression in vascular smooth muscle cells (Q28318657) (← links)
- Glutamine metabolism to glucosamine is necessary for glutamine inhibition of endothelial nitric oxide synthesis (Q28345516) (← links)
- Functional regulation of glutamine:fructose-6-phosphate aminotransferase 1 (GFAT1) of Drosophila melanogaster in a UDP-N-acetylglucosamine and cAMP-dependent manner (Q28364618) (← links)
- Cloning and partial characterization of the mouse glutamine:fructose-6-phosphate amidotransferase (GFAT) gene promoter (Q28587198) (← links)
- A PGC-1alpha-O-GlcNAc transferase complex regulates FoxO transcription factor activity in response to glucose (Q30437005) (← links)
- O-GlcNAc regulates FoxO activation in response to glucose (Q30439420) (← links)
- Quantification of O-GlcNAc protein modification in neutrophils by flow cytometry (Q30532863) (← links)
- Hexosamine pathway but not interstitial changes mediates glucotoxicity in pancreatic β-cells as assessed by cytosolic Ca2+ response to glucose (Q33577717) (← links)
- Protein-protein interaction in insulin signaling and the molecular mechanisms of insulin resistance (Q33588931) (← links)
- The hexosamine signaling pathway: O-GlcNAc cycling in feast or famine (Q33625053) (← links)
- Influence of glucosamine on glomerular mesangial cell turnover: implications for hyperglycemia and hexosamine pathway flux (Q33655472) (← links)
- The role of O-GlcNAc signaling in the pathogenesis of diabetic retinopathy (Q33683197) (← links)
- Quantitative secretome and glycome of primary human adipocytes during insulin resistance (Q33749585) (← links)
- Dynamic O-GlcNAc cycling at promoters of Caenorhabditis elegans genes regulating longevity, stress, and immunity (Q33842100) (← links)
- Bitterness in sugar: O-GlcNAcylation aggravates pre-B acute lymphocytic leukemia through glycolysis via the PI3K/Akt/c-Myc pathway (Q33850060) (← links)
- MPI depletion enhances O-GlcNAcylation of p53 and suppresses the Warburg effect. (Q33866466) (← links)
- O-GlcNAcylation/phosphorylation cycling at Ser10 controls both transcriptional activity and stability of delta-lactoferrin (Q33911287) (← links)
- O-GlcNAcylation stabilizes β-catenin through direct competition with phosphorylation at threonine 41. (Q33913190) (← links)
- O-GlcNAcylation regulates cancer metabolism and survival stress signaling via regulation of the HIF-1 pathway. (Q33923934) (← links)
- X-inactivation normalizes O-GlcNAc transferase levels and generates an O-GlcNAc-depleted Barr body (Q33997535) (← links)
- Elevated nucleocytoplasmic glycosylation by O-GlcNAc results in insulin resistance associated with defects in Akt activation in 3T3-L1 adipocytes (Q34024499) (← links)
- O-GlcNAc cycling: emerging roles in development and epigenetics (Q34049939) (← links)
- O-GlcNAc signaling in the cardiovascular system (Q34056858) (← links)
- Increased O-GlcNAc levels correlate with decreased O-GlcNAcase levels in Alzheimer disease brain (Q34073596) (← links)
- Altered glycan-dependent signaling induces insulin resistance and hyperleptinemia (Q34075585) (← links)
- Quantification of O-glycosylation stoichiometry and dynamics using resolvable mass tags (Q34075637) (← links)
- Comparative effects of fructose and glucose on lipogenic gene expression and intermediary metabolism in HepG2 liver cells (Q34077667) (← links)
- Power training and postmenopausal hormone therapy affect transcriptional control of specific co-regulated gene clusters in skeletal muscle. (Q34080414) (← links)
- O-GlcNAc Modification: Friend or Foe in Diabetic Cardiovascular Disease. (Q34101859) (← links)
- O-GlcNAc signaling: a metabolic link between diabetes and cancer? (Q34172895) (← links)