Pages that link to "Q40426449"
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The following pages link to Role of M cells in initial antigen uptake and in ulcer formation in the rabbit intestinal loop model of shigellosis (Q40426449):
Displaying 50 items.
- Type III protein secretion systems in bacterial pathogens of animals and plants (Q24548602) (← links)
- Role of flagella in adherence, internalization, and translocation of Campylobacter jejuni in nonpolarized and polarized epithelial cell cultures (Q24645680) (← links)
- Shigella flexneri enters human colonic Caco-2 epithelial cells through the basolateral pole (Q24646749) (← links)
- Yersinia enterocolitica: the charisma continues (Q24683715) (← links)
- Riboregulators: Fine-Tuning Virulence in Shigella (Q26767390) (← links)
- Shigella IpaH Family Effectors as a Versatile Model for Studying Pathogenic Bacteria (Q26771364) (← links)
- The route less taken: pulmonary models of enteric Gram-negative infection (Q27691303) (← links)
- A bacterial invasin induces macrophage apoptosis by binding directly to ICE (Q28512900) (← links)
- Shigella flexneri infection in Caenorhabditis elegans: cytopathological examination and identification of host responses (Q28542808) (← links)
- Dendritic cells express tight junction proteins and penetrate gut epithelial monolayers to sample bacteria (Q29619620) (← links)
- How bacteria initiate inflammation: aspects of the emerging story (Q33538552) (← links)
- The regulation of apoptosis by microbial pathogens. (Q33599759) (← links)
- The lpf fimbrial operon mediates adhesion of Salmonella typhimurium to murine Peyer's patches (Q33616531) (← links)
- Comprehensive study of the intestinal stage of listeriosis in a rat ligated ileal loop system. (Q33750545) (← links)
- New advances in microsphere-based single-dose vaccines (Q33933148) (← links)
- The pathogenesis of Shigella flexneri infection: lessons from in vitro and in vivo studies (Q33955468) (← links)
- Vaccination strategies for mucosal immune responses (Q33975703) (← links)
- Structure-function analysis of the Shigella virulence factor IpaB. (Q33995573) (← links)
- Parameters underlying successful protection with live attenuated mutants in experimental shigellosis (Q34006071) (← links)
- Monocyte-mediated defense against microbial pathogens. (Q34065898) (← links)
- Molecular basis of the intracellular spreading of Shigella (Q34090958) (← links)
- Polymorphonuclear leukocyte transmigration promotes invasion of colonic epithelial monolayer by Shigella flexneri (Q34125052) (← links)
- Interleukin 1 is released by murine macrophages during apoptosis induced by Shigella flexneri (Q34186287) (← links)
- Involvement of M cells in the bacterial invasion of Peyer's patches: a common mechanism shared by Yersinia enterocolitica and other enteroinvasive bacteria (Q34392887) (← links)
- Identification of substrates and chaperone from the Yersinia enterocolitica 1B Ysa type III secretion system (Q34527081) (← links)
- Host-pathogen interactions: the seduction of molecular cross talk (Q34600953) (← links)
- Convergent evolution in the assembly of polyubiquitin degradation signals by the Shigella flexneri IpaH9.8 ligase (Q34634239) (← links)
- Improved outcome in shigellosis associated with butyrate induction of an endogenous peptide antibiotic (Q34694936) (← links)
- Intestinal M cells and their role in bacterial infection (Q35130619) (← links)
- In vitro and in vivo characterization of an ail mutant of Yersinia enterocolitica (Q35422247) (← links)
- Pathogenesis of defined invasion mutants of Yersinia enterocolitica in a BALB/c mouse model of infection (Q35456987) (← links)
- Induction of a local anti-IpaC antibody response in mice by use of a Shigella flexneri 2a vaccine candidate: implications for use of IpaC as a protein carrier (Q35480510) (← links)
- Infection of rabbit Peyer's patches by Shigella flexneri: effect of adhesive or invasive bacterial phenotypes on follicle-associated epithelium (Q35506252) (← links)
- In vivo apoptosis in Shigella flexneri infections (Q35532651) (← links)
- Human monocyte-derived macrophages infected with virulent Shigella flexneri in vitro undergo a rapid cytolytic event similar to oncosis but not apoptosis (Q35545918) (← links)
- The interleukin 1beta-converting enzyme, caspase 1, is activated during Shigella flexneri-induced apoptosis in human monocyte-derived macrophages (Q35560355) (← links)
- Role of interleukin-1 in the pathogenesis of experimental shigellosis (Q35752669) (← links)
- Epithelial cell invasion by bovine septicemic Escherichia coli (Q35770835) (← links)
- Salmonella typhimurium initiates murine infection by penetrating and destroying the specialized epithelial M cells of the Peyer's patches. (Q36363455) (← links)
- Acute inflammation causes epithelial invasion and mucosal destruction in experimental shigellosis (Q36363758) (← links)
- Monoclonal immunoglobulin A antibody directed against serotype-specific epitope of Shigella flexneri lipopolysaccharide protects against murine experimental shigellosis (Q36365336) (← links)
- The inlAB locus mediates the entry of Listeria monocytogenes into hepatocytes in vivo (Q36366266) (← links)
- Molecular pathogenesis of Shigella spp.: controlling host cell signaling, invasion, and death by type III secretion. (Q36422508) (← links)
- Virulent Shigella flexneri subverts the host innate immune response through manipulation of antimicrobial peptide gene expression (Q36640009) (← links)
- Shigella's ways of manipulating the host intestinal innate and adaptive immune system: a tool box for survival? (Q36703588) (← links)
- Intracellular and cell-to-cell spread of Listeria monocytogenes involves interaction with F-actin in the enterocytelike cell line Caco-2. (Q36979864) (← links)
- Characterization of intracellular growth regulator icgR by utilizing transcriptomics to identify mediators of pathogenesis in Shigella flexneri (Q37123743) (← links)
- Systems-level overview of host protein phosphorylation during Shigella flexneri infection revealed by phosphoproteomics. (Q37214709) (← links)
- Genetic basis of virulence in Shigella species (Q37295263) (← links)
- M cells are involved in pathogenesis of human contact lens-associated giant papillary conjunctivitis (Q37395970) (← links)