Pages that link to "Q40406151"

The following pages link to Liwu Li (Q40406151):

Displaying 50 items.

• The p53-targeting human phosphatase hCdc14A interacts with the Cdk1/cyclin B complex and is differentially expressed in human cancers (Q21245741) (← links)
• Differential regulation and role of interleukin-1 receptor associated kinase-M in innate immunity signaling (Q24300680) (← links)
• A family of putative tumor suppressors is structurally and functionally conserved in humans and yeast (Q24336385) (← links)
• The ubiquitin ligase Stub1 negatively modulates regulatory T cell suppressive activity by promoting degradation of the transcription factor Foxp3 (Q28118511) (← links)
• Activation of interleukin-1 receptor-associated kinase by gram-negative flagellin (Q28201436) (← links)
• Assembly of inflammation-related genes for pathway-focused genetic analysis (Q28222837) (← links)
• Interactions of sequence variants in interleukin-1 receptor-associated kinase4 and the toll-like receptor 6-1-10 gene cluster increase prostate cancer risk (Q28302052) (← links)
• An innate immunity signaling process suppresses macrophage ABCA1 expression through IRAK-1-mediated downregulation of retinoic acid receptor alpha and NFATc2 (Q28504786) (← links)
• Interleukin-1 Receptor-Associated Kinase-1 (IRAK-1) functionally associates with PKCepsilon and VASP in the regulation of macrophage migration (Q33706807) (← links)
• Molecular and cellular mechanisms responsible for cellular stress and low-grade inflammation induced by a super-low dose of endotoxin (Q33718511) (← links)
• Mathematical modeling for the pathogenesis of Alzheimer's disease. (Q33778539) (← links)
• Toll-like receptor 4 modulates skeletal muscle substrate metabolism (Q33840362) (← links)
• Form, function, and regulation of protein tyrosine phosphatases and their involvement in human diseases. (Q33867275) (← links)
• The human Cdc14 phosphatases interact with and dephosphorylate the tumor suppressor protein p53. (Q33887422) (← links)
• Dynamic modulation of innate immune response by varying dosages of lipopolysaccharide (LPS) in human monocytic cells (Q33985344) (← links)
• A mathematical model for the reciprocal differentiation of T helper 17 cells and induced regulatory T cells (Q33987374) (← links)
• Molecular mechanism underlying persistent induction of LCN2 by lipopolysaccharide in kidney fibroblasts (Q34238633) (← links)
• Network topologies and dynamics leading to endotoxin tolerance and priming in innate immune cells (Q34276989) (← links)
• A simple theoretical framework for understanding heterogeneous differentiation of CD4+ T cells. (Q34304146) (← links)
• In vitro and in vivo reconstitution and stability of vertebrate chromosome ends. (Q34668792) (← links)
• Innate immune programing by endotoxin and its pathological consequences (Q34841012) (← links)
• Alteration of lysosome fusion and low-grade inflammation mediated by super-low-dose endotoxin. (Q35173047) (← links)
• Myeloid cell-derived inducible nitric oxide synthase suppresses M1 macrophage polarization (Q35311873) (← links)
• Trehalose-mediated autophagy impairs the anti-viral function of human primary airway epithelial cells. (Q35448331) (← links)
• Failure of TLR4-driven NF-kappa B activation to stimulate virus replication in models of HIV type 1 activation (Q35556260) (← links)
• Deletion of PPAR-γ in immune cells enhances susceptibility to antiglomerular basement membrane disease (Q35557407) (← links)
• Super-low dose endotoxin pre-conditioning exacerbates sepsis mortality (Q35654379) (← links)
• Regulation of innate immunity signaling and its connection with human diseases (Q35698940) (← links)
• The association between innate immunity gene (IRAK1) and C-reactive protein in the Diabetes Heart Study (Q36015656) (← links)
• Molecular mechanisms responsible for the selective and low-grade induction of proinflammatory mediators in murine macrophages by lipopolysaccharide (Q36084187) (← links)
• Genomic DNA extraction from cells by electroporation on an integrated microfluidic platform (Q36382651) (← links)
• Intervention of Toll-like receptor-mediated human innate immunity and inflammation by synthetic compounds and naturally occurring products (Q36486164) (← links)
• Molecular mechanisms responsible for the reduced expression of cholesterol transporters from macrophages by low-dose endotoxin (Q36534100) (← links)
• Molecular mechanism responsible for the priming of macrophage activation. (Q36596174) (← links)
• Low-grade inflammatory polarization of monocytes impairs wound healing (Q36599048) (← links)
• Subclinical-Dose Endotoxin Sustains Low-Grade Inflammation and Exacerbates Steatohepatitis in High-Fat Diet-Fed Mice (Q36600936) (← links)
• Differential regulation of Foxp3 and IL-17 expression in CD4 T helper cells by IRAK-1 (Q36636243) (← links)
• Regulations and roles of the interleukin-1 receptor associated kinases (IRAKs) in innate and adaptive immunity (Q36685571) (← links)
• The interleukin-1 receptor associated kinase 1 contributes to the regulation of NFAT (Q36965348) (← links)
• The involvement of the interleukin-1 receptor-associated kinases (IRAKs) in cellular signaling networks controlling inflammation (Q37076790) (← links)
• Interleukin-1 receptor-associated kinase M (IRAK-M) promotes human rhinovirus infection in lung epithelial cells via the autophagic pathway (Q37244005) (← links)
• Deficiency in Toll-interacting protein (Tollip) skews inflamed yet incompetent innate leukocytes in vivo during DSS-induced septic colitis (Q37310570) (← links)
• Change in mononuclear leukocyte responsiveness in midpregnancy and subsequent preterm birth (Q37315255) (← links)
• The interleukin-1 receptor-associated kinase M selectively inhibits the alternative, instead of the classical NFkappaB pathway (Q37374200) (← links)
• The persistence of low-grade inflammatory monocytes contributes to aggravated atherosclerosis (Q37408646) (← links)
• IRAK-1 contributes to lipopolysaccharide-induced reactive oxygen species generation in macrophages by inducing NOX-1 transcription and Rac1 activation and suppressing the expression of antioxidative enzymes (Q37467568) (← links)
• Endotoxin tolerance dysregulates MyD88- and Toll/IL-1R domain-containing adapter inducing IFN-beta-dependent pathways and increases expression of negative regulators of TLR signaling (Q37486509) (← links)
• Reprogramming macrophage orientation by microRNA 146b targeting transcription factor IRF5 (Q37510166) (← links)
• Epigallocatechin-3-gallate (EGCG) attenuates inflammation in MRL/lpr mouse mesangial cells (Q37727280) (← links)
• Molecular mechanisms and pathological consequences of endotoxin tolerance and priming (Q37964940) (← links)