Pages that link to "Q40454938"
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The following pages link to Identification of a DEF-type docking domain for extracellular signal-regulated kinases 1/2 that directs phosphorylation and turnover of the BH3-only protein BimEL. (Q40454938):
Displaying 27 items.
- Evidence that Ser87 of BimEL is phosphorylated by Akt and regulates BimEL apoptotic function (Q24294552) (← links)
- Identification of a novel Bcl-2-interacting mediator of cell death (Bim) E3 ligase, tripartite motif-containing protein 2 (TRIM2), and its role in rapid ischemic tolerance-induced neuroprotection (Q24299252) (← links)
- ERK1/2-dependent phosphorylation of BimEL promotes its rapid dissociation from Mcl-1 and Bcl-xL (Q24306894) (← links)
- Substrate discrimination among mitogen-activated protein kinases through distinct docking sequence motifs (Q24647635) (← links)
- Ubiquitin-proteasome system as a modulator of cell fate (Q24656277) (← links)
- Defining the role of the Bcl-2 family proteins in Huntington's disease (Q26822846) (← links)
- betaTrCP- and Rsk1/2-mediated degradation of BimEL inhibits apoptosis (Q28306984) (← links)
- ATP-sensitive potassium (KATP) channel openers diazoxide and nicorandil lower intraocular pressure by activating the Erk1/2 signaling pathway (Q33778841) (← links)
- The MEK-ERK pathway negatively regulates bim expression through the 3' UTR in sympathetic neurons (Q33962497) (← links)
- Quantitative analysis of ERK2 interactions with substrate proteins: roles for kinase docking domains and activity in determining binding affinity (Q34509259) (← links)
- The BCL-2 protein family, BH3-mimetics and cancer therapy (Q36067778) (← links)
- NF-kappaB1 and c-Rel cooperate to promote the survival of TLR4-activated B cells by neutralizing Bim via distinct mechanisms (Q36104876) (← links)
- Current strategies in overcoming resistance of cancer cells to apoptosis melanoma as a model. (Q36580838) (← links)
- Tumour cell survival signalling by the ERK1/2 pathway (Q37290943) (← links)
- Apoptosis and autophagy: BIM as a mediator of tumour cell death in response to oncogene-targeted therapeutics (Q37605160) (← links)
- Insulin-like growth factor 1 rescues R28 retinal neurons from apoptotic death through ERK-mediated BimEL phosphorylation independent of Akt. (Q38752597) (← links)
- Rapid ischemic tolerance induced by adenosine preconditioning results in Bcl-2 interacting mediator of cell death (Bim) degradation by the proteasome. (Q41978908) (← links)
- The sphingolipid degradation product trans-2-hexadecenal induces cytoskeletal reorganization and apoptosis in a JNK-dependent manner (Q42062961) (← links)
- Alternative splicing of Bim and Erk-mediated Bim(EL) phosphorylation are dispensable for hematopoietic homeostasis in vivo. (Q42173208) (← links)
- Loss of pro-apoptotic Bim promotes accumulation of pulmonary T lymphocytes and enhances allergen-induced goblet cell metaplasia (Q42497012) (← links)
- c-Cbl is not required for ERK1/2-dependent degradation of BimEL. (Q43098314) (← links)
- IL-15 modulates the balance between Bcl-2 and Bim via a Jak3/1-PI3K-Akt-ERK pathway to promote CD8αα+ intestinal intraepithelial lymphocyte survival (Q43448874) (← links)
- A cellular threshold for active ERK1/2 levels determines Raf/MEK/ERK-mediated growth arrest versus death responses (Q47848122) (← links)
- A high throughput assay to identify substrate-selective inhibitors of the ERK protein kinases (Q50925733) (← links)
- Extracellular-Regulated Kinases: Signaling From Ras to ERK Substrates to Control Biological Outcomes. (Q52725941) (← links)
- The Solanum chacoense Fertilization-Related Kinase 3 (ScFRK3) is involved in male and female gametophyte development (Q64053204) (← links)
- Regulation of the proapoptotic BH3-only protein BIM by glucocorticoids, survival signals and proteasome in chronic lymphocytic leukemia cells (Q79412060) (← links)