Pages that link to "Q40171802"

The following pages link to Knockdown of DNA ligase IV/XRCC4 by RNA interference inhibits herpes simplex virus type I DNA replication (Q40171802):

Displaying 36 items.

• Involvement of DNA ligase III and ribonuclease H1 in mitochondrial DNA replication in cultured human cells (Q24336833) (← links)
• The DNA-damage response in human biology and disease (Q24606586) (← links)
• The HSV-1 exonuclease, UL12, stimulates recombination by a single strand annealing mechanism (Q28593412) (← links)
• Contributions of nucleotide excision repair, DNA polymerase eta, and homologous recombination to replication of UV-irradiated herpes simplex virus type 1 (Q33538952) (← links)
• Identification of rep-associated factors in herpes simplex virus type 1-induced adeno-associated virus type 2 replication compartments. (Q33614551) (← links)
• Cellular DNA ligase I is recruited to cytoplasmic vaccinia virus factories and masks the role of the vaccinia ligase in viral DNA replication (Q33757344) (← links)
• An E2F1-mediated DNA damage response contributes to the replication of human cytomegalovirus (Q33904045) (← links)
• Processing of lagging-strand intermediates in vitro by herpes simplex virus type 1 DNA polymerase (Q33964152) (← links)
• Structure of the herpes simplex virus 1 genome: manipulation of nicks and gaps can abrogate infectivity and alter the cellular DNA damage response. (Q34059509) (← links)
• Evidence that herpes simplex virus DNA derived from quiescently infected cellsin vitro, and latently infected cellsin vivo, is physically damaged (Q34167741) (← links)
• The modulation of phosphatase expression impacts the proliferation efficiency of HSV-1 in infected astrocytes (Q35048616) (← links)
• Targeting non-coding RNAs with the CRISPR/Cas9 system in human cell lines (Q35088923) (← links)
• Rad51 and Rad52 are involved in homologous recombination of replicating herpes simplex virus DNA. (Q35387821) (← links)
• Changing the ubiquitin landscape during viral manipulation of the DNA damage response (Q35854193) (← links)
• Direct evidence that HSV DNA damaged by ultraviolet (UV) irradiation can be repaired in a cell type-dependent manner (Q35992304) (← links)
• Herpes simplex viruses: mechanisms of DNA replication (Q36192926) (← links)
• Telomeres and viruses: common themes of genome maintenance. (Q36497017) (← links)
• Visualizing the replicating HSV-1 virus using STED super-resolution microscopy. (Q36783523) (← links)
• Bacterial DNA repair by non-homologous end joining (Q36972411) (← links)
• Viral and Cellular Components of AAV2 Replication Compartments (Q37294482) (← links)
• Viral manipulation of DNA repair and cell cycle checkpoints (Q37301053) (← links)
• Argininosuccinate synthetase 1 depletion produces a metabolic state conducive to herpes simplex virus 1 infection (Q37409497) (← links)
• Genomes in Conflict: Maintaining Genome Integrity During Virus Infection (Q37778386) (← links)
• Replication and recombination of herpes simplex virus DNA (Q37848529) (← links)
• Increasing the efficiency of homology-directed repair for CRISPR-Cas9-induced precise gene editing in mammalian cells. (Q38895804) (← links)
• Repair of chromosomal double-strand breaks by precise ligation in human cells (Q39147622) (← links)
• Regulation of Telomere Homeostasis during Epstein-Barr virus Infection and Immortalization. (Q40089376) (← links)
• Reconstitution of uracil DNA glycosylase-initiated base excision repair in herpes simplex virus-1 (Q41946552) (← links)
• Stepwise evolution of the herpes simplex virus origin binding protein and origin of replication (Q42231478) (← links)
• PNKP knockdown by RNA interference inhibits herpes simplex virus-1 replication in astrocytes (Q42256560) (← links)
• Small interfering RNA targeting for infected-cell polypeptide 4 inhibits herpes simplex virus type 1 replication in retinal pigment epithelial cells (Q45363703) (← links)
• Characterization of plasma proteins in children of different Mycobacterium tuberculosis infection status using label-free quantitative proteomics (Q47122587) (← links)
• The Non-Homologous End Joining Protein PAXX Acts to Restrict HSV-1 Infection. (Q47151188) (← links)
• Enhanced non-homologous end joining contributes toward synthetic lethality of pathological RAD51C mutants with poly (ADP-ribose) polymerase. (Q51045691) (← links)
• Structures of DNA-bound human ligase IV catalytic core reveal insights into substrate binding and catalysis. (Q55473320) (← links)
• In search for effective and definitive treatment of herpes simplex virus type 1 (HSV-1) infections (Q56789970) (← links)