Pages that link to "Q37993118"
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The following pages link to Alternative options for DNA-based experimental therapy of β-thalassemia. (Q37993118):
Displaying 16 items.
- An international effort to cure a global health problem: A report on the 19th Hemoglobin Switching Conference (Q33850615) (← links)
- α-Globin as a molecular target in the treatment of β-thalassemia (Q35836891) (← links)
- Peptide nucleic acids: a review on recent patents and technology transfer (Q38176989) (← links)
- Recent trends for novel options in experimental biological therapy of β-thalassemia (Q38220695) (← links)
- Recent trends in the gene therapy of β-thalassemia (Q38367473) (← links)
- Hemin induces mitophagy in a leukemic erythroblast cell line. (Q38803104) (← links)
- Peptide nucleic acids targeting β-globin mRNAs selectively inhibit hemoglobin production in murine erythroleukemia cells (Q38939602) (← links)
- Induction of erythroid differentiation and increased globin mRNA production with furocoumarins and their photoproducts (Q39814424) (← links)
- Generation and Characterization of a Transgenic Mouse Carrying a Functional Human β -Globin Gene with the IVSI-6 Thalassemia Mutation. (Q40746113) (← links)
- An Aγ-globin G->A gene polymorphism associated with β039 thalassemia globin gene and high fetal hemoglobin production (Q41554655) (← links)
- The potential role of cell penetrating peptides in the intracellular delivery of proteins for therapy of erythroid related disorders. (Q42253045) (← links)
- Development and characterization of K562 cell clones expressing BCL11A-XL: Decreased hemoglobin production with fetal hemoglobin inducers and its rescue with mithramycin (Q43142280) (← links)
- Consensus Predictive Model for Human K562 Cell Growth Inhibition through Enalos Cloud Platform (Q47806909) (← links)
- Structural and Functional Insights on an Uncharacterized Aγ-Globin-Gene Polymorphism Present in Four β0-Thalassemia Families with High Fetal Hemoglobin Levels. (Q53771829) (← links)
- Orphan Drugs and Potential Novel Approaches for Therapies of β-Thalassemia: Current Status and Future Expectations (Q54585888) (← links)
- Hemin induces autophagy in a leukemic erythroblast cell line through the LRP1 receptor (Q60921223) (← links)