Pages that link to "Q28240662"
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The following pages link to A monoclonal antibody that targets a NaV1.7 channel voltage sensor for pain and itch relief (Q28240662):
Displaying 50 items.
- Anti-nerve growth factor in pain management: current evidence (Q26746194) (← links)
- Inhibition of mechanical allodynia in neuropathic pain by TLR5-mediated A-fiber blockade (Q27314839) (← links)
- Intrathecal bone marrow stromal cells inhibit neuropathic pain via TGF-β secretion (Q27332195) (← links)
- Advances in understanding itching and scratching: a new era of targeted treatments (Q28071549) (← links)
- Exacerbating factors of itch in atopic dermatitis (Q28075828) (← links)
- Neuroimmune interactions in itch: Do chronic itch, chronic pain, and chronic cough share similar mechanisms? (Q28083464) (← links)
- Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7 (Q28270611) (← links)
- Nav1.7 and other voltage-gated sodium channels as drug targets for pain relief (Q28273870) (← links)
- Global Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to pain (Q28542818) (← links)
- Modeling nociception in zebrafish: a way forward for unbiased analgesic discovery (Q28543016) (← links)
- Inhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of Pain (Q28548202) (← links)
- Evaluation of recombinant monoclonal antibody SVmab1 binding to Na V1.7 target sequences and block of human Na V1.7 currents (Q28587362) (← links)
- Subtype-Selective Small Molecule Inhibitors Reveal a Fundamental Role for Nav1.7 in Nociceptor Electrogenesis, Axonal Conduction and Presynaptic Release (Q28601095) (← links)
- Information processing in the CNS: a supramolecular chemistry? (Q28607744) (← links)
- New Horizons in Diabetic Neuropathy: Mechanisms, Bioenergetics, and Pain (Q30235132) (← links)
- Toll-like receptor 4 contributes to chronic itch, alloknesis, and spinal astrocyte activation in male mice (Q30785636) (← links)
- Insensitivity to pain induced by a potent selective closed-state Nav1.7 inhibitor (Q33390819) (← links)
- β-arrestin-2 regulates NMDA receptor function in spinal lamina II neurons and duration of persistent pain (Q33814944) (← links)
- Affimer proteins are versatile and renewable affinity reagents (Q33842511) (← links)
- Defining the nociceptor transcriptome (Q34487175) (← links)
- Bacterial voltage-gated sodium channels (BacNa(V)s) from the soil, sea, and salt lakes enlighten molecular mechanisms of electrical signaling and pharmacology in the brain and heart (Q34778955) (← links)
- A surface plasmon resonance approach to monitor toxin interactions with an isolated voltage-gated sodium channel paddle motif. (Q35014380) (← links)
- Reduction of Neuropathic and Inflammatory Pain through Inhibition of the Tetrahydrobiopterin Pathway (Q35797434) (← links)
- Temporal dynamics of anxiety phenotypes in a dental pulp injury model (Q35803593) (← links)
- Role of anoctamin-1 and bestrophin-1 in spinal nerve ligation-induced neuropathic pain in rats. (Q35805234) (← links)
- Novel sodium channel antagonists in the treatment of neuropathic pain. (Q35843983) (← links)
- Recent Trends in the Discovery of Small Molecule Blockers of Sodium Channels. (Q36018974) (← links)
- Matrix Metalloproteinase (MMP) Proteolysis of the Extracellular Loop of Voltage-gated Sodium Channels and Potential Alterations in Pain Signaling (Q36281738) (← links)
- A monoclonal antibody against KCNK9 K(+) channel extracellular domain inhibits tumour growth and metastasis (Q36548721) (← links)
- Transient Receptor Potential Vanilloid 4 Ion Channel Functions as a Pruriceptor in Epidermal Keratinocytes to Evoke Histaminergic Itch. (Q36876222) (← links)
- Peripheral Brain Derived Neurotrophic Factor Precursor Regulates Pain as an Inflammatory Mediator (Q36959399) (← links)
- Disrupting the dynamic equilibrium of ORAI channels determines the phenotype of malignant cells (Q36997514) (← links)
- Engineering Highly Potent and Selective Microproteins against Nav1.7 Sodium Channel for Treatment of Pain (Q37065681) (← links)
- Sodium channel Nav1.7 expression is upregulated in the dorsal root ganglia in a rat model of paclitaxel-induced peripheral neuropathy (Q37318012) (← links)
- MicroRNA-30b regulates expression of the sodium channel Nav1.7 in nerve injury-induced neuropathic pain in the rat (Q37368204) (← links)
- Hierarchical CRMP2 posttranslational modifications control NaV1.7 function (Q37549939) (← links)
- No gain, no pain: NaV1.7 as an analgesic target (Q38238866) (← links)
- The voltage-gated sodium channel: a major target of marine neurotoxins (Q38259139) (← links)
- Discovery of functional antibodies targeting ion channels (Q38282832) (← links)
- New horizons in therapeutic antibody discovery: opportunities and challenges versus small-molecule therapeutics (Q38289992) (← links)
- Voltage-Gated Sodium Channels: Structure, Function, Pharmacology, and Clinical Indications (Q38451134) (← links)
- Emerging targets in treating pain (Q38531133) (← links)
- Structural and genetic diversity in antibody repertoires from diverse species (Q38549871) (← links)
- Toward a Mechanism-Based Approach to Pain Diagnosis (Q38559303) (← links)
- NaV1.9: a sodium channel linked to human pain (Q38560993) (← links)
- Afferent hyperexcitability in neuropathic pain and the inconvenient truth about its degeneracy. (Q38585556) (← links)
- The voltage-gated sodium channel NaV 1.9 in visceral pain (Q38606237) (← links)
- Combining Human and Rodent Genetics to Identify New Analgesics (Q38699127) (← links)
- Functional Studies of Sodium Channels: From Target to Compound Identification (Q38726402) (← links)
- Voltage-gated sodium channel Nav 1.7 promotes gastric cancer progression through MACC1-mediated upregulation of NHE1. (Q38751561) (← links)