Pages that link to "Q24314203"
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The following pages link to Interaction between human MCM7 and Rad17 proteins is required for replication checkpoint signaling (Q24314203):
Displaying 50 items.
- ATR serine/threonine kinase (Q4812911) (← links)
- RAD17 checkpoint clamp loader component (Q21115925) (← links)
- minichromosome maintenance complex component 7 (Q21117085) (← links)
- Direct interaction between cohesin complex and DNA replication machinery (Q24302104) (← links)
- Human Tim/Timeless-interacting protein, Tipin, is required for efficient progression of S phase and DNA replication checkpoint (Q24315071) (← links)
- Interactions of the human MCM-BP protein with MCM complex components and Dbf4 (Q24317620) (← links)
- Interaction of chromatin-associated Plk1 and Mcm7 (Q24338260) (← links)
- Functional uncoupling of MCM helicase and DNA polymerase activities activates the ATR-dependent checkpoint. (Q24522744) (← links)
- ATRIP binding to replication protein A-single-stranded DNA promotes ATR-ATRIP localization but is dispensable for Chk1 phosphorylation (Q24523440) (← links)
- Replication licensing and cancer--a fatal entanglement? (Q24644766) (← links)
- Cdc7-Dbf4 and the human S checkpoint response to UVC (Q24672815) (← links)
- The Mcm2-7 replicative helicase: a promising chemotherapeutic target (Q26861045) (← links)
- Spy1 expression prevents normal cellular responses to DNA damage: inhibition of apoptosis and checkpoint activation (Q28261764) (← links)
- Proteomic identification of macrophage migration-inhibitory factor upon exposure to TiO2 particles (Q28566280) (← links)
- Cdt1-binding protein GRWD1 is a novel histone-binding protein that facilitates MCM loading through its influence on chromatin architecture (Q28596608) (← links)
- ATM- and cell cycle-dependent regulation of ATR in response to DNA double-strand breaks (Q29614215) (← links)
- Conserved modes of recruitment of ATM, ATR and DNA-PKcs to sites of DNA damage (Q29614218) (← links)
- Hepatitis B virus X protein induces apoptosis and cell cycle deregulation through interfering with DNA repair and checkpoint responses (Q33293386) (← links)
- Differential requirements for MCM proteins in DNA replication in Drosophila S2 cells (Q33296994) (← links)
- Mcm4 C-terminal domain of MCM helicase prevents excessive formation of single-stranded DNA at stalled replication forks (Q33364389) (← links)
- Tipin-replication protein A interaction mediates Chk1 phosphorylation by ATR in response to genotoxic stress (Q33541382) (← links)
- Incremental Genetic Perturbations to MCM2-7 Expression and Subcellular Distribution Reveal Exquisite Sensitivity of Mice to DNA Replication Stress (Q33691497) (← links)
- Optimization based tumor classification from microarray gene expression data. (Q33822080) (← links)
- The dynamics of E1A in regulating networks and canonical pathways in quiescent cells (Q33912982) (← links)
- Decreased MCM2-6 in Drosophila S2 cells does not generate significant DNA damage or cause a marked increase in sensitivity to replication interference (Q34079253) (← links)
- The interaction between checkpoint kinase 1 (Chk1) and the minichromosome maintenance (MCM) complex is required for DNA damage-induced Chk1 phosphorylation (Q34107413) (← links)
- Dimerization of the ATRIP protein through the coiled-coil motif and its implication to the maintenance of stalled replication forks (Q34148137) (← links)
- MCM Paradox: Abundance of Eukaryotic Replicative Helicases and Genomic Integrity. (Q34441575) (← links)
- Differential hRad17 expression by histologic subtype of ovarian cancer. (Q34797749) (← links)
- Proteomic analysis of proton beam irradiated human melanoma cells (Q35083440) (← links)
- BRCA1 is required for postreplication repair after UV-induced DNA damage (Q35434721) (← links)
- Phosphorylation of Minichromosome Maintenance 3 (MCM3) by Checkpoint Kinase 1 (Chk1) Negatively Regulates DNA Replication and Checkpoint Activation. (Q35583204) (← links)
- Chromatin remodeler sucrose nonfermenting 2 homolog (SNF2H) is recruited onto DNA replication origins through interaction with Cdc10 protein-dependent transcript 1 (Cdt1) and promotes pre-replication complex formation (Q35604309) (← links)
- A comparative proteomic study identified LRPPRC and MCM7 as putative actors in imatinib mesylate cross-resistance in Lucena cell line. (Q35991845) (← links)
- Post-transcriptional homeostasis and regulation of MCM2-7 in mammalian cells (Q36008189) (← links)
- Proton irradiation impacts age-driven modulations of cancer progression influenced by immune system transcriptome modifications from splenic tissue (Q36078666) (← links)
- A genome-wide RNAi screen identifies core components of the G₂-M DNA damage checkpoint (Q36372110) (← links)
- The fission yeast minichromosome maintenance (MCM)-binding protein (MCM-BP), Mcb1, regulates MCM function during prereplicative complex formation in DNA replication (Q36666107) (← links)
- Phosphorylation of MCM3 on Ser-112 regulates its incorporation into the MCM2-7 complex (Q36725691) (← links)
- Excess MCM proteins protect human cells from replicative stress by licensing backup origins of replication. (Q36752433) (← links)
- Reducing MCM levels in human primary T cells during the G(0)-->G(1) transition causes genomic instability during the first cell cycle (Q36846014) (← links)
- Phosphorylation of minichromosome maintenance protein 7 (MCM7) by cyclin/cyclin-dependent kinase affects its function in cell cycle regulation (Q37000584) (← links)
- Divergent S phase checkpoint activation arising from prereplicative complex deficiency controls cell survival (Q37327314) (← links)
- The Mcm complex: unwinding the mechanism of a replicative helicase. (Q37451513) (← links)
- Simvastatin suppresses the DNA replication licensing factor MCM7 and inhibits the growth of tamoxifen-resistant breast cancer cells (Q37620557) (← links)
- Excess Mcm2-7 license dormant origins of replication that can be used under conditions of replicative stress. (Q39729829) (← links)
- Roles of human AND-1 in chromosome transactions in S phase (Q39852067) (← links)
- Dormant origins licensed by excess Mcm2-7 are required for human cells to survive replicative stress. (Q40035632) (← links)
- Identification of carboxyl-terminal MCM3 phosphorylation sites using polyreactive phosphospecific antibodies (Q40179946) (← links)
- RNAi-mediated knockdown of MCM7 gene on CML cells and its therapeutic potential for leukemia (Q40393432) (← links)