HLA-E-dependent presentation of Mtb-derived antigen to human CD8+ T cells (Q36371913)

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Language	Label	Description	Also known as
English	HLA-E-dependent presentation of Mtb-derived antigen to human CD8+ T cells	scientific article

Statements

instance of

scholarly article

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Europe PubMed Central

PMC publication ID

2194265

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13 August 2017

title

HLA-E-dependent presentation of Mtb-derived antigen to human CD8+ T cells (English)

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PMC publication ID

2194265

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13 August 2017

author name string

Amy S Heinzel

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1

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2194265

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Jeff E Grotzke

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2

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2194265

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13 August 2017

Rebecca A Lines

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2194265

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13 August 2017

Deborah A Lewinsohn

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4

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2194265

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13 August 2017

Andria L McNabb

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Daniel N Streblow

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Veronique M Braud

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2194265

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Heather J Grieser

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2194265

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John T Belisle

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9

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2194265

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David M Lewinsohn

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10

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publication date

1 December 2002

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13 August 2017

published in

Journal of Experimental Medicine

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13 August 2017

volume

196

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2194265

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issue

11

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page(s)

1473-1481

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2194265

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13 August 2017

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Human T cell receptor-mediated recognition of HLA-E.

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The analysis of the natural killer-like activity of human cytolytic T lymphocytes revealed HLA-E as a novel target for TCR alpha/beta-mediated recognition

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Processing of a multiple membrane spanning Epstein-Barr virus protein for CD8(+) T cell recognition reveals a proteasome-dependent, transporter associated with antigen processing-independent pathway

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Peptide binding characteristics of the non-classical class Ib MHC molecule HLA-E assessed by a recombinant random peptide approach

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CD8+ T cells recognize an inclusion membrane-associated protein from the vacuolar pathogen Chlamydia trachomatis

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Lipoprotein access to MHC class I presentation during infection of murine macrophages with live mycobacteria.

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Surface expression of HLA-E, an inhibitor of natural killer cells, enhanced by human cytomegalovirus gpUL40.

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Characterization of human Mycobacterium bovis bacille Calmette-Guérin-reactive CD8+ T cells.

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Susceptibility of mice deficient in CD1D or TAP1 to infection with Mycobacterium tuberculosis

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An antimicrobial activity of cytolytic T cells mediated by granulysin

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HLA-E surface expression depends on binding of TAP-dependent peptides derived from certain HLA class I signal sequences

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Characterization of human CD8+ T cells reactive with Mycobacterium tuberculosis-infected antigen-presenting cells.

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HLA-E is a major ligand for the natural killer inhibitory receptor CD94/NKG2A

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HLA-E binds to natural killer cell receptors CD94/NKG2A, B and C

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Class I-like CD1A-C do not protect target cells from NK-mediated cytolysis

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Proliferating dendritic cell progenitors in human blood

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Major histocompatibility complex class I-restricted T cells are required for resistance to Mycobacterium tuberculosis infection

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Identifiers

DOI

10.1084/JEM.20020609

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Europe PubMed Central

PMC publication ID

2194265

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13 August 2017

PMC publication ID

2194265

1 reference

stated in

Europe PubMed Central

PMC publication ID

2194265

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13 August 2017

PubMed publication ID

12461082

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Europe PubMed Central

PMC publication ID

2194265

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13 August 2017

 

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