Degeneration of neuronal cells due to oxidative stress--microglial contribution (Q34826210)

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Language	Label	Description	Also known as
English	Degeneration of neuronal cells due to oxidative stress--microglial contribution	scientific article

Statements

scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

4 August 2017

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Europe PubMed Central

Degeneration of neuronal cells due to oxidative stress--microglial contribution (English)

1 reference

Europe PubMed Central

PubMed publication ID

4 August 2017

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author name string

Koutsilieri E

1

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Europe PubMed Central

PubMed publication ID

4 August 2017

Scheller C

2

1 reference

Europe PubMed Central

PubMed publication ID

4 August 2017

Tribl F

3

1 reference

Europe PubMed Central

PubMed publication ID

4 August 2017

Riederer P

4

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Europe PubMed Central

PubMed publication ID

4 August 2017

publication date

1 September 2002

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Europe PubMed Central

PubMed publication ID

4 August 2017

number of pages

6

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inferred from page(s)

Parkinsonism and Related Disorders

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Europe PubMed Central

PubMed publication ID

4 August 2017

8

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Europe PubMed Central

PubMed publication ID

4 August 2017

6

1 reference

Europe PubMed Central

PubMed publication ID

4 August 2017

401-406

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Europe PubMed Central

PubMed publication ID

4 August 2017

Control of glial immune function by neurons

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HIV-1 Tat potentiates TNF-induced NF-kappa B activation and cytotoxicity by altering the cellular redox state

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Atherosclerosis, oxidative stress, and antioxidant protection in endothelium-derived relaxing factor action

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Oxidative damage to DNA: relation to species metabolic rate and life span

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Dopamine turnover and glutathione oxidation: implications for Parkinson disease

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Mitochondrial complex I deficiency in Parkinson's disease

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Decreased catalase activity but unchanged superoxide dismutase activity in brains of patients with dementia of Alzheimer type.

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Oxidative stress: its role in the pathogenesis of amyotrophic lateral sclerosis

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Cellular mechanisms of resistance to chronic oxidative stress

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Oxidative stress in neurodegenerative diseases

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Free radicals in the physiological control of cell function

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Oxidative stress: free radical production in neural degeneration.

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Mitochondrial implication in accidental and programmed cell death: apoptosis and necrosis

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Theories of neurodegeneration

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Monoamine oxidase and mitochondrial respiration

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Caspases: Intracellular Signaling by Proteolysis

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Nitric oxide and cell death

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Oxidative damage to mitochondrial DNA and glutathione oxidation in apoptosis: studies in vivo and in vitro

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Immunologic NO synthase: elevation in severe AIDS dementia and induction by HIV-1 gp41.

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Specific and reversible inactivation of protein tyrosine phosphatases by hydrogen peroxide: evidence for a sulfenic acid intermediate and implications for redox regulation

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Roles of superoxide radical anion in signal transduction mediated by reversible regulation of protein-tyrosine phosphatase 1B

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Activation of the Lck tyrosine protein kinase by hydrogen peroxide requires the phosphorylation of Tyr-394.

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NF-kappa B: ten years after

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Reactive oxygen intermediates as apparently widely used messengers in the activation of the NF-kappa B transcription factor and HIV-1

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Amino acid analogs activate NF-kappaB through redox-dependent IkappaB-alpha degradation by the proteasome without apparent IkappaB-alpha phosphorylation. Consequence on HIV-1 long terminal repeat activation

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Crucial role of the amino-terminal tyrosine residue 42 and the carboxyl-terminal PEST domain of I kappa B alpha in NF-kappa B activation by an oxidative stress

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H2O2 and antioxidants have opposite effects on activation of NF-kappa B and AP-1 in intact cells: AP-1 as secondary antioxidant-responsive factor.

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10.1016/S1353-8020(02)00021-4

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Europe PubMed Central

PubMed publication ID

4 August 2017

release_nasgdnrb65gsvg2k656ireqs5y

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24 November 2022

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

4 August 2017

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