Somatic Apc mutations are selected upon their capacity to inactivate the beta-catenin downregulating activity (Q28507875)

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Language	Label	Description	Also known as
English	Somatic Apc mutations are selected upon their capacity to inactivate the beta-catenin downregulating activity	scientific journal article

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scholarly article

1 reference

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PubMed

PubMed publication ID

10992298

retrieved

24 January 2017

title

Somatic Apc mutations are selected upon their capacity to inactivate the beta-catenin downregulating activity (English)

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PubMed

PubMed publication ID

10992298

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24 January 2017

main subject

MutS homolog 2

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GOA release 2020-03-11

Eukaryotic elongation factor, selenocysteine-tRNA-specific

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GOA release 2020-03-11

author name string

R. Smits

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1

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PubMed

PubMed publication ID

10992298

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24 January 2017

N. Hofland

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2

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PubMed

PubMed publication ID

10992298

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24 January 2017

W. Edelmann

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3

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PubMed

PubMed publication ID

10992298

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24 January 2017

M. Geugien

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PubMed

PubMed publication ID

10992298

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24 January 2017

S. Jagmohan-Changur

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5

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PubMed

PubMed publication ID

10992298

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24 January 2017

C. Albuquerque

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6

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PubMed

PubMed publication ID

10992298

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24 January 2017

C. Breukel

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PubMed

PubMed publication ID

10992298

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24 January 2017

R. Kucherlapati

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8

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PubMed

PubMed publication ID

10992298

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24 January 2017

M. F. Kielman

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9

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PubMed

PubMed publication ID

10992298

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24 January 2017

R. Fodde

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10

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PubMed

PubMed publication ID

10992298

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24 January 2017

publication date

1 November 2000

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PubMed

PubMed publication ID

10992298

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24 January 2017

published in

Genes, Chromosomes and Cancer

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PubMed

PubMed publication ID

10992298

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24 January 2017

volume

29

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PubMed

PubMed publication ID

10992298

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24 January 2017

issue

3

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PubMed

PubMed publication ID

10992298

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24 January 2017

page(s)

229–239

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PubMed

PubMed publication ID

10992298

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24 January 2017

cites work

Functional interaction of an axin homolog, conductin, with beta-catenin, APC, and GSK3beta

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Crossref

reference URL

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21 January 2018

Inactivation of the mouse Msh2 gene results in mismatch repair deficiency, methylation tolerance, hyperrecombination, and predisposition to cancer

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21 January 2018

A targeted chain-termination mutation in the mouse Apc gene results in multiple intestinal tumors

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21 January 2018

Chain-terminating mutations in the APC gene lead to alterations in APC RNA and protein concentration

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reference URL

https://api.crossref.org/works/10.1002%2F1098-2264%282000%299999%3A9999%3C%3A%3AAID-GCC1033%3E3.0.CO%3B2-R

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21 January 2018

Identification and characterization of the familial adenomatous polyposis coli gene

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21 January 2018

Downregulation of beta-catenin by human Axin and its association with the APC tumor suppressor, beta-catenin and GSK3 beta

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21 January 2018

Constitutive transcriptional activation by a beta-catenin-Tcf complex in APC-/- colon carcinoma

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21 January 2018

The type of somatic mutation at APC in familial adenomatous polyposis is determined by the site of the germline mutation: a new facet to Knudson's 'two-hit' hypothesis

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21 January 2018

Genetic instabilities in human cancers

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21 January 2018

Somatic mutations of the APC gene in colorectal tumors: mutation cluster region in the APC gene

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21 January 2018

Activation of beta-catenin-Tcf signaling in colon cancer by mutations in beta-catenin or APC

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21 January 2018

A dominant mutation that predisposes to multiple intestinal neoplasia in the mouse

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21 January 2018

Regulation of intracellular beta-catenin levels by the adenomatous polyposis coli (APC) tumor-suppressor protein

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21 January 2018

Axin, an inhibitor of the Wnt signalling pathway, interacts with beta-catenin, GSK-3beta and APC and reduces the beta-catenin level

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Crossref

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21 January 2018

Mutations of chromosome 5q21 genes in FAP and colorectal cancer patients

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21 January 2018

Loss of Apc heterozygosity and abnormal tissue building in nascent intestinal polyps in mice carrying a truncated Apc gene

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21 January 2018

APC mutations occur early during colorectal tumorigenesis

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21 January 2018

MSH2 deficient mice are viable and susceptible to lymphoid tumours

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21 January 2018

Association of the APC gene product with beta-catenin

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Crossref

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https://api.crossref.org/works/10.1002%2F1098-2264%282000%299999%3A9999%3C%3A%3AAID-GCC1033%3E3.0.CO%3B2-R

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21 January 2018

The APC gene product in normal and tumor cells

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Crossref

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21 January 2018

Loss of Apc and the entire chromosome 18 but absence of mutations at the Ras and Tp53 genes in intestinal tumors from Apc1638N, a mouse model for Apc-driven carcinogenesis.

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Crossref

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21 January 2018

Apc1638N: a mouse model for familial adenomatous polyposis-associated desmoid tumors and cutaneous cysts

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21 January 2018

Apc1638T: a mouse model delineating critical domains of the adenomatous polyposis coli protein involved in tumorigenesis and development

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21 January 2018

Multiple intestinal neoplasia caused by a mutation in the murine homolog of the APC gene

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21 January 2018

Association of the APC tumor suppressor protein with catenins

1 reference

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Crossref

reference URL

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21 January 2018

Germline mutations in the 3' part of APC exon 15 do not result in truncated proteins and are associated with attenuated adenomatous polyposis coli.

1 reference

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Crossref

reference URL

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21 January 2018

Beta-catenin: a key mediator of Wnt signaling

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1002%2F1098-2264%282000%299999%3A9999%3C%3A%3AAID-GCC1033%3E3.0.CO%3B2-R

retrieved

21 January 2018

Selection, the mutation rate and cancer: Ensuring that the tail does not wag the dog

1 reference

stated in

Crossref

reference URL

https://api.crossref.org/works/10.1002%2F1098-2264%282000%299999%3A9999%3C%3A%3AAID-GCC1033%3E3.0.CO%3B2-R

retrieved

21 January 2018

Identifiers

DOI

10.1002/1098-2264(2000)9999:9999<::AID-GCC1033>3.0.CO;2-R

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

667171

OpenCitations bibliographic resource ID

667171

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

667171

PubMed publication ID

10992298

1 reference

stated in

Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

667171

 

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