The immunological basis for treatment of stiff person syndrome (Q28295552)

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scientific article published on October 12, 2010

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English	The immunological basis for treatment of stiff person syndrome	scientific article published on October 12, 2010

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scholarly article

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The immunological basis for treatment of stiff person syndrome (English)

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based on heuristic

inferred from title

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object named as

Trygve Holmøy

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2

object named as

Christian Geis

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Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A20943276

1 August 2024

language of work or name

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publication date

February 2011

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12 October 2010

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Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?resulttype=core&format=json&query=SRC%3AMED%20AND%20EXT_ID%3A20943276

1 August 2024

full work available at URL

https://api.elsevier.com/content/article/PII:S0165572810004339?httpAccept=text/xml

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10.1016/J.JNEUROIM.2010.09.014

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1 August 2024

https://api.elsevier.com/content/article/PII:S0165572810004339?httpAccept=text/plain

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Journal of Neuroimmunology

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231

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1-2

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55-60

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Neuronal damage in autoimmune neuroinflammation mediated by the death ligand TRAIL.

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Distinct antigenic features of linear epitopes at the N-terminus and C-terminus of 65 kDa glutamic acid decarboxylase (GAD65): implications for autoantigen modification during pathogenesis

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Subacute encephalomyelitis presenting as stiff-person syndrome: clinical, polygraphic, and pathologic correlations

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Cellular immunity to a determinant common to glutamate decarboxylase and coxsackie virus in insulin-dependent diabetes

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Stiff-person syndrome: persistent elevation of glutamic acid decarboxylase antibodies despite successful treatment with rituximab

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Identification of the 64K autoantigen in insulin-dependent diabetes as the GABA-synthesizing enzyme glutamic acid decarboxylase

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Treatment of stiff person syndrome with rituximab.

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Modulation of antigen presentation by autoreactive B cell clones specific for GAD65 from a type I diabetic patient

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Review of 23 patients affected by the stiff man syndrome: clinical subdivision into stiff trunk (man) syndrome, stiff limb syndrome, and progressive encephalomyelitis with rigidity

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Intrathecal pathogenic anti-aquaporin-4 antibodies in early neuromyelitis optica

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Mild experimental autoimmune encephalitis as a tool to induce blood-brain barrier dysfunction

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The stiff man and stiff man plus syndromes

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The stiff leg syndrome

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High definition profiling of autoantibodies to glutamic acid decarboxylases GAD65/GAD67 in stiff-person syndrome

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Central nervous system destruction mediated by glutamic acid decarboxylase-specific CD4+ T cells

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Identification of a dominant epitope of glutamic acid decarboxylase (GAD-65) recognized by autoantibodies in stiff-man syndrome

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Autoimmunity to gephyrin in Stiff-Man syndrome

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T-cell reactivity to glutamic acid decarboxylase in stiff-man syndrome and cerebellar ataxia associated with polyendocrine autoimmunity

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Antigen processing patterns determine GAD65-specific regulation vs. pathogenesis

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Advances in the pathogenesis and treatment of patients with stiff person syndrome

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Stiff person syndrome: advances in pathogenesis and therapeutic interventions

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The clinical spectrum of anti-GAD antibody-positive patients with stiff-person syndrome

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High-Dose Intravenous Immune Globulin for Stiff-Person Syndrome

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Stiff person syndrome: quantification, specificity, and intrathecal synthesis of GAD65 antibodies

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Autoimmunity in stiff-Man syndrome with breast cancer is targeted to the C-terminal region of human amphiphysin, a protein similar to the yeast proteins, Rvs167 and Rvs161

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The synaptic vesicle-associated protein amphiphysin is the 128-kD autoantigen of Stiff-Man syndrome with breast cancer

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Decreased synaptic vesicle recycling efficiency and cognitive deficits in amphiphysin 1 knockout mice

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Inhibition of gamma-aminobutyric acid synthesis by glutamic acid decarboxylase autoantibodies in stiff-man syndrome

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A selective activity-dependent requirement for dynamin 1 in synaptic vesicle endocytosis

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Physiologic studies of spinal inhibitory circuits in patients with stiff-person syndrome

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Characterization of CD4+ T cells specific for glutamic acid decarboxylase (GAD65) and proinsulin in a patient with stiff-person syndrome but without type 1 diabetes

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Immune reactivity to glutamic acid decarboxylase 65 in stiffman syndrome and type 1 diabetes mellitus.

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Analysis of GAD65 autoantibodies in Stiff-Person syndrome patients

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Autoimmunity to GABAA-receptor-associated protein in stiff-person syndrome

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GAD65-specific autoantibodies enhance the presentation of an immunodominant T-cell epitope from GAD65.

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Long-lived plasma cells and memory B cells produce pathogenic anti-GAD65 autoantibodies in Stiff Person Syndrome

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GAD65-Reactive T cells in a non-diabetic stiff-man syndrome patient

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Synaptic vesicle endocytosis impaired by disruption of dynamin-SH3 domain interactions

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10.1016/J.JNEUROIM.2010.09.014

1 reference

Consolidated OpenCitations Corpus – April 2017

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OpenCitations bibliographic resource ID

1 reference

Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017

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