Selegiline attenuates cardiac oxidative stress and apoptosis in heart failure: association with improvement of cardiac function (Q44314777)

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scientific article published in February 2003

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English	Selegiline attenuates cardiac oxidative stress and apoptosis in heart failure: association with improvement of cardiac function	scientific article published in February 2003

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

Selegiline attenuates cardiac oxidative stress and apoptosis in heart failure: association with improvement of cardiac function (English)

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

1 reference

based on heuristic

inferred from title

author name string

Fuzhong Qin

1

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

Junya Shite

2

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

Weike Mao

3

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

Chang-seng Liang

4

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Europe PubMed Central

PubMed publication ID

1 December 2017

language of work or name

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publication date

1 February 2003

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Europe PubMed Central

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1 December 2017

European Journal of Pharmacology

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Europe PubMed Central

PubMed publication ID

1 December 2017

461

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Europe PubMed Central

PubMed publication ID

1 December 2017

2-3

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

149-158

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Europe PubMed Central

PubMed publication ID

1 December 2017

Direct activation of mitochondrial apoptosis machinery by c-Jun N-terminal kinase in adult cardiac myocytes

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The mitochondrial apoptotic pathway is activated by serum and glucose deprivation in cardiac myocytes

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The potentiation of the anti akinetic effect after L-Dopa treatment by an inhibitor of Mao-B, deprenil

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(-)deprenyl increases activities of superoxide dismutase and catalase in certain brain regions in old male mice

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Long term treatment with (-)deprenyl reduces the optimal dose as well as the effective dose range for increasing antioxidant enzyme activities in old mouse brain.

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A high dose of long term treatment with deprenyl loses its effect on antioxidant enzyme activities as well as on survivals of Fischer-344 rats

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Effects of early decrease in oxidative stress after medical therapy in patients with class IV congestive heart failure

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Oxidative stress-mediated cardiac cell death is a major determinant of ventricular dysfunction and failure in dog dilated cardiomyopathy

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Cardiovascular changes in response to selective monoamine oxidase inhibition in the rat

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Heart-targeted overexpression of caspase3 in mice increases infarct size and depresses cardiac function.

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Chromatographic determination of 8-oxo-7,8-dihydro-2'-deoxyguanosine in cellular DNA: a validation study

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Oxidative stress and antioxidant therapy in Parkinson's disease

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Neutrophil superoxide anion--generating capacity, endothelial function and oxidative stress in chronic heart failure: effects of short- and long-term vitamin C therapy

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Peroxynitrite is a major contributor to cytokine-induced myocardial contractile failure

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Apoptosis in cardiac diseases: stress- and mitogen-activated signaling pathways

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Cell death in acromegalic cardiomyopathy

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Oxidative stress and apoptosis in heart failure progression

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Bcl-2 functions in an antioxidant pathway to prevent apoptosis

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Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarction

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Alterations in cardiac adrenergic terminal function and beta-adrenoceptor density in pacing-induced heart failure

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Comparative effects of melatonin, l-deprenyl, Trolox and ascorbate in the suppression of hydroxyl radical formation during dopamine autoxidation in vitro

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Treatment with dimethylthiourea prevents left ventricular remodeling and failure after experimental myocardial infarction in mice: role of oxidative stress

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Upregulation of antioxidant enzyme activities by deprenyl. Implications for life span extension

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Assessing the effects of deprenyl on longevity and antioxidant defenses in different animal models

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Pharmacological modifications of endogenous antioxidant enzymes with special reference to the effects of deprenyl: a possible antioxidant strategy

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Blocking caspase-activated apoptosis improves contractility in failing myocardium

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(-)-Deprenyl protection of 1-methyl-4 phenylpyridium ion (MPP+)-induced apoptosis independent of MAO-B inhibition

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The oxidative stress hypothesis of congestive heart failure: radical thoughts

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Mechanism underlying anti-apoptotic activity of a (-)deprenyl-related propargylamine, rasagiline.

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Carvedilol decreases elevated oxidative stress in human failing myocardium

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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Antioxidants attenuate myocyte apoptosis in the remote non-infarcted myocardium following large myocardial infarction

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Antioxidant vitamins prevent cardiomyocyte apoptosis produced by norepinephrine infusion in ferrets.

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Apoptotic cell death in heart failure

1 reference

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Deprenyl reduces the death of motoneurons caused by axotomy

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Norepinephrine metabolites in plasma as indicators of pharmacological inhibition of monoamine oxidase and catechol O-methyltransferase

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Selegiline enhances NGF synthesis and protects central nervous system neurons from excitotoxic and ischemic damage

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Assays of oxidative DNA damage biomarkers 8-oxo-2'-deoxyguanosine and 8-oxoguanine in nuclear DNA and biological fluids by high-performance liquid chromatography with electrochemical detection

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Selegiline improves cardiac sympathetic terminal function and beta-adrenergic responsiveness in heart failure

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(-)-D-Deprenyl attenuates apoptosis in experimental brain ischaemia

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Selegiline diminishes cardiovascular autonomic responses in Parkinson's disease.

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Mitochondrial membrane potential and nuclear changes in apoptosis caused by serum and nerve growth factor withdrawal: time course and modification by (-)-deprenyl.

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Detection of apoptotic cells in human colorectal cancer by two different in situ methods: antibody against single-stranded DNA and terminal deoxynucleotidyl transferase-mediated dUTP-biotin nick end-labeling (TUNEL) methods.

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Antioxidant mechanism and protection of nigral neurons against MPP+ toxicity by deprenyl (selegiline).

1 reference

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l-Deprenyl, blocking apoptosis and regulating gene expression in cultured retinal neurons

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Reperfusion induces myocardial apoptotic cell death

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Identifiers

10.1016/S0014-2999(03)01306-2

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 December 2017

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