Pages that link to "Q24302248"
The following pages link to Interaction of F1L with the BH3 domain of Bak is responsible for inhibiting vaccinia-induced apoptosis (Q24302248):
Displaying 50 items.
- BCL2 associated X, apoptosis regulator (Q408476) (← links)
- BCL2 antagonist/killer 1 (Q4875407) (← links)
- Vaccinia virus B1R kinase interacts with JIP1 and modulates c-Jun-dependent signaling (Q24297063) (← links)
- Functional and structural studies of the vaccinia virus virulence factor N1 reveal a Bcl-2-like anti-apoptotic protein (Q24305049) (← links)
- The vaccinia virus protein F1L interacts with Bim and inhibits activation of the pro-apoptotic protein Bax (Q24310440) (← links)
- Inhibition of IkappaB kinase by vaccinia virus virulence factor B14 (Q24312867) (← links)
- Vaccinia virus N1L protein resembles a B cell lymphoma-2 (Bcl-2) family protein (Q24319056) (← links)
- Vaccinia virus anti-apoptotic F1L is a novel Bcl-2-like domain-swapped dimer that binds a highly selective subset of BH3-containing death ligands (Q24321449) (← links)
- Iridovirus CARD Protein Inhibits Apoptosis through Intrinsic and Extrinsic Pathways (Q27304913) (← links)
- Host-Cell Survival and Death During Chlamydia Infection (Q27487010) (← links)
- Structure of M11L: A myxoma virus structural homolog of the apoptosis inhibitor, Bcl-2 (Q27644227) (← links)
- Inhibition of Apoptosis and NF-κB Activation by Vaccinia Protein N1 Occur via Distinct Binding Surfaces and Make Different Contributions to Virulence (Q27676390) (← links)
- Structural Insight into BH3 Domain Binding of Vaccinia Virus Antiapoptotic F1L (Q27683883) (← links)
- Modified Vaccinia Virus Ankara (Q28223836) (← links)
- A survey of host range genes in poxvirus genomes (Q28389388) (← links)
- Poxviruses and the evolution of host range and virulence (Q28658130) (← links)
- Vaccinia virus protein A49 is an unexpected member of the B-cell Lymphoma (Bcl)-2 protein family (Q30370948) (← links)
- The N Terminus of the Vaccinia Virus Protein F1L Is an Intrinsically Unstructured Region That Is Not Involved in Apoptosis Regulation (Q30387749) (← links)
- A novel Bcl-2-like inhibitor of apoptosis is encoded by the parapoxvirus ORF virus (Q33283495) (← links)
- Caspase-dependent inhibition of mousepox replication by gzmB. (Q33511238) (← links)
- Vaccinia virus protein F1L is a caspase-9 inhibitor (Q33648434) (← links)
- Induction of Noxa-mediated apoptosis by modified vaccinia virus Ankara depends on viral recognition by cytosolic helicases, leading to IRF-3/IFN-β-dependent induction of pro-apoptotic Noxa. (Q33941041) (← links)
- Ena/VASP proteins cooperate with the WAVE complex to regulate the actin cytoskeleton (Q34193675) (← links)
- Deletion of the viral anti-apoptotic gene F1L in the HIV/AIDS vaccine candidate MVA-C enhances immune responses against HIV-1 antigens (Q34465228) (← links)
- Structural determinants of caspase-9 inhibition by the vaccinia virus protein, F1L (Q35182960) (← links)
- An overview of the vaccinia virus infectome: a survey of the proteins of the poxvirus-infected cell (Q35689619) (← links)
- The Molluscum Contagiosum Virus protein MC163 localizes to the mitochondria and dampens mitochondrial mediated apoptotic responses (Q36290498) (← links)
- Sheeppox virus SPPV14 encodes a Bcl-2-like cell death inhibitor that counters a distinct set of mammalian proapoptotic proteins. (Q36363353) (← links)
- Mechanisms of apoptosis regulation by viral oncogenes in infection and tumorigenesis. (Q36468906) (← links)
- Embedded together: the life and death consequences of interaction of the Bcl-2 family with membranes (Q36799170) (← links)
- Vaccinia virus F1L protein promotes virulence by inhibiting inflammasome activation (Q36835632) (← links)
- Vaccinia virus protein N2 is a nuclear IRF3 inhibitor that promotes virulence (Q37109669) (← links)
- The fowlpox virus BCL-2 homologue, FPV039, interacts with activated Bax and a discrete subset of BH3-only proteins to inhibit apoptosis (Q37247956) (← links)
- Viral inhibitors reveal overlapping themes in regulation of cell death and innate immunity (Q37592685) (← links)
- Mitochondrial localization of viral proteins as a means to subvert host defense (Q37784282) (← links)
- Use of functional genomics to understand replication deficient poxvirus-host interactions (Q38620980) (← links)
- Analysis of the anti-apoptotic activity of four vaccinia virus proteins demonstrates that B13 is the most potent inhibitor in isolation and during viral infection (Q38968870) (← links)
- Viral exploitation of the MEK/ERK pathway - A tale of vaccinia virus and other viruses (Q39318787) (← links)
- The vaccinia virus-encoded Bcl-2 homologues do not act as direct Bax inhibitors (Q39455647) (← links)
- Vaccinia virus F1L interacts with Bak using highly divergent Bcl-2 homology domains and replaces the function of Mcl-1. (Q39539847) (← links)
- Viral pro-survival proteins block separate stages in Bax activation but changes in mitochondrial ultrastructure still occur (Q40013224) (← links)
- Poxviruses Utilize Multiple Strategies to Inhibit Apoptosis (Q40090965) (← links)
- The Bcl-2 Family in Host-Virus Interactions. (Q41922877) (← links)
- Vaccinia-induced epidermal growth factor receptor-MEK signalling and the anti-apoptotic protein F1L synergize to suppress cell death during infection (Q42023832) (← links)
- Fowlpox virus encodes a Bcl-2 homologue that protects cells from apoptotic death through interaction with the proapoptotic protein Bak. (Q42638938) (← links)
- Vaccinia virus protein A49 activates Wnt signalling by targetting the E3 ligase β-TrCP. (Q45325861) (← links)
- Modulating Vaccinia Virus Immunomodulators to Improve Immunological Memory (Q50533334) (← links)
- Expression of the Vaccinia Virus Antiapoptotic F1 Protein Is Blocked by Protein Kinase R in the Absence of the Viral E3 Protein (Q59357146) (← links)
- NF-κB activation is a turn on for vaccinia virus phosphoprotein A49 to turn off NF-κB activation (Q64092200) (← links)
- Novel Modified Vaccinia Virus Ankara Vector Expressing Anti-apoptotic Gene B13R Delays Apoptosis and Enhances Humoral Responses (Q90434144) (← links)