(Q37987485)

26 August 2017

title

The genetic basis of primary aldosteronism (English)

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26 August 2017

John W Funder

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26 August 2017

publication date

1 April 2012

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Current Hypertension Reports

26 August 2017

published in

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26 August 2017

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26 August 2017

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26 August 2017

page(s)

120-124

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High prevalence of autonomous cortisol and aldosterone secretion from adrenal adenomas

26 August 2017

cites work

1 reference

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Transactivation via the human glucocorticoid and mineralocorticoid receptor by therapeutically used steroids in CV-1 cells: a comparison of their glucocorticoid and mineralocorticoid properties

21 January 2018

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A chimaeric llβ-hydroxylase/aldosterone synthase gene causes glucocorticoid-remediable aldosteronism and human hypertension

21 January 2018

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Further study of chromosome 7p22 to identify the molecular basis of familial hyperaldosteronism type II.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11906-012-0255-X

A novel form of human mendelian hypertension featuring nonglucocorticoid-remediable aldosteronism

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11906-012-0255-X

K+ channel mutations in adrenal aldosterone-producing adenomas and hereditary hypertension

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11906-012-0255-X

Evidence for abnormal left ventricular structure and function in normotensive individuals with familial hyperaldosteronism type I.

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11906-012-0255-X

Emerging roles for two-pore-domain potassium channels and their potential therapeutic impact

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11906-012-0255-X

Invalidation of TASK1 potassium channels disrupts adrenal gland zonation and mineralocorticoid homeostasis

21 January 2018

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https://api.crossref.org/works/10.1007%2FS11906-012-0255-X

Salt-sensitive hypertension in circadian clock-deficient Cry-null mice involves dysregulated adrenal Hsd3b6.

21 January 2018

1 reference

https://api.crossref.org/works/10.1007%2FS11906-012-0255-X

TASK channel deletion in mice causes primary hyperaldosteronism

21 January 2018

1 reference

12 December 2020

Prevalence, clinical, and molecular correlates of KCNJ5 mutations in primary aldosteronism.

1 reference

12 December 2020

Primary aldosteronism: are we missing the wood for the trees?

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12 December 2020

Medicine. The genetics of primary aldosteronism

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12 December 2020

Potassium channel mutant KCNJ5 T158A expression in HAC-15 cells increases aldosterone synthesis

1 reference

12 December 2020

The genetics of primary aldosteronism: chapter two

1 reference

12 December 2020

10.1007/S11906-012-0255-X

Identifiers

DOI

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26 August 2017

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