(Q34341979)

English

Cell surface major histocompatibility complex class II proteins are regulated by the products of the gamma(1)34.5 and U(L)41 genes of herpes simplex virus 1.

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scholarly article

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Europe PubMed Central

PubMed publication ID

1 August 2017

Cell surface major histocompatibility complex class II proteins are regulated by the products of the gamma(1)34.5 and U(L)41 genes of herpes simplex virus 1. (English)

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

Bernard Roizman

3

object named as

Bernard Roizman

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

author name string

Joanne Trgovcich

1

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

David Johnson

2

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

language of work or name

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publication date

1 July 2002

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

number of pages

13

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Journal of Virology

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

76

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

14

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

6974-6986

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

Dendritic cells: specialized and regulated antigen processing machines

1 reference

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1 reference

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1 reference

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Tegument-specific, virus-reactive CD4 T cells localize to the cornea in herpes simplex virus interstitial keratitis in humans.

1 reference

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Mature dendritic cells infected with herpes simplex virus type 1 exhibit inhibited T-cell stimulatory capacity

1 reference

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Modulation of major histocompatibility class II protein expression by varicella-zoster virus

1 reference

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Murine cytomegalovirus infection down-regulates MHC class II expression on macrophages by induction of IL-10.

1 reference

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Viral strategies of immune evasion

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Murine cytomegalovirus inhibits interferon gamma-induced antigen presentation to CD4 T cells by macrophages via regulation of expression of major histocompatibility complex class II-associated genes

1 reference

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Human cytomegalovirus inhibits major histocompatibility complex class II expression by disruption of the Jak/Stat pathway

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1 reference

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Covalent modification of the active site threonine of proteasomal beta subunits and the Escherichia coli homolog HslV by a new class of inhibitors

1 reference

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The range and distribution of murine central nervous system cells infected with the gamma(1)34.5- mutant of herpes simplex virus 1.

1 reference

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6 July 2018

Differentiation of the shutoff of protein synthesis by virion host shutoff and mutant gamma (1)34.5 genes of herpes simplex virus 1.

1 reference

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The gamma(1)34.5 protein of herpes simplex virus 1 complexes with protein phosphatase 1alpha to dephosphorylate the alpha subunit of the eukaryotic translation initiation factor 2 and preclude the shutoff of protein synthesis by double-stranded RNA-

1 reference

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The proteolytic environment involved in MHC class II-restricted antigen presentation can be modulated by the p41 form of invariant chain

1 reference

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The extracellular domain of the Epstein-Barr virus BZLF2 protein binds the HLA-DR beta chain and inhibits antigen presentation

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Major histocompatibility complex class II-associated p41 invariant chain fragment is a strong inhibitor of lysosomal cathepsin L

1 reference

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Herpes simplex virus VP16 rescues viral mRNA from destruction by the virion host shutoff function

1 reference

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Isoforms of the invariant chain regulate transport of MHC class II molecules to antigen processing compartments

1 reference

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Processing of the herpes simplex virus regulatory protein alpha 22 mediated by the UL13 protein kinase determines the accumulation of a subset of alpha and gamma mRNAs and proteins in infected cells

1 reference

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Functional cytotoxic T cells are associated with focal lesions in the brains of SJL mice with experimental herpes simplex encephalitis

1 reference

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1 reference

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1 reference

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Herpes simplex virus turns off the TAP to evade host immunity

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Dendritic cells use macropinocytosis and the mannose receptor to concentrate macromolecules in the major histocompatibility complex class II compartment: downregulation by cytokines and bacterial products

1 reference

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Characterization of herpes simplex virus strains differing in their effects on social behaviour of infected cells

1 reference

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Herpes simplex virus 1 protein kinase is encoded by open reading frame US3 which is not essential for virus growth in cell culture

1 reference

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Virulence of and establishment of latency by genetically engineered deletion mutants of herpes simplex virus 1.

1 reference

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1 reference

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Mapping of herpes simplex virus-1 neurovirulence to gamma 134.5, a gene nonessential for growth in culture

1 reference

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1 reference

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Decreased T cell stimulatory capacity of monocyte-derived human macrophages following herpes simplex virus type 1 infection.

1 reference

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1 reference

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Either a CD4(+)or CD8(+)T cell function is sufficient for clearance of infectious virus from trigeminal ganglia and establishment of herpes simplex virus type 1 latency in mice

1 reference

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Bystander activation of CD4(+) T cells can represent an exclusive means of immunopathology in a virus infection

1 reference

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Inhibition of dendritic cell maturation by herpes simplex virus

1 reference

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Herpesvirus evasion of the immune system.

1 reference

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A viral inhibitor of peptide transporters for antigen presentation

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Infection of dendritic cells by murine cytomegalovirus induces functional paralysis

1 reference

https://pubmed.ncbi.nlm.nih.gov/12072498

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Cytomegalovirus US2 destroys two components of the MHC class II pathway, preventing recognition by CD4+ T cells

1 reference

https://pubmed.ncbi.nlm.nih.gov/12072498

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1128/JVI.76.14.6974-6986.2002

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

PMC publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 August 2017

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