(Q33592649)

28 July 2017

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title

The role of COP1 in repression of Arabidopsis photomorphogenic development (English)

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28 July 2017

M T Osterlund

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28 July 2017

L H Ang

series ordinal

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28 July 2017

X W Deng

series ordinal

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28 July 2017

publication date

1 March 1999

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28 July 2017

published in

Trends in Cell Biology

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28 July 2017

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28 July 2017

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28 July 2017

page(s)

113-118

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NF-kappaB activation: the I kappaB kinase revealed?

28 July 2017

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Light inactivation of Arabidopsis photomorphogenic repressor COP1 involves a cell-specific regulation of its nucleocytoplasmic partitioning

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Overexpression of Arabidopsis COP1 results in partial suppression of light-mediated development: evidence for a light-inactivable repressor of photomorphogenesis.

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Regulatory hierarchy of photomorphogenic loci: allele-specific and light-dependent interaction between the HY5 and COP1 loci

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COP1, an Arabidopsis regulatory gene, encodes a protein with both a zinc-binding motif and a G beta homologous domain

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Genetic and developmental control of nuclear accumulation of COP1, a repressor of photomorphogenesis in Arabidopsis.

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Phytochrome A null mutants of Arabidopsis display a wild-type phenotype in white light

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Mutations in the gene for the red/far-red light receptor phytochrome B alter cell elongation and physiological responses throughout Arabidopsis development

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HY4 gene of A. thaliana encodes a protein with characteristics of a blue-light photoreceptor

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Multiple photoreceptors mediate the light-induced reduction of GUS-COP1 from Arabidopsis hypocotyl nuclei

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Arabidopsis COP1 protein specifically interacts in vitro with a cytoskeleton-associated protein, CIP1

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A complement of ten essential and pleiotropic arabidopsis COP/DET/FUS genes is necessary for repression of photomorphogenesis in darkness

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Molecular interaction between COP1 and HY5 defines a regulatory switch for light control of Arabidopsis development

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Genetic and molecular analysis of an allelic series of cop1 mutants suggests functional roles for the multiple protein domains.

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Identifiers

DOI

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28 July 2017

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