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Mitochondrial fission, fusion, and stress
scientific article (publication date: 31 August 2012)
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title
Mitochondrial fission, fusion, and stress
(English)
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PubMed
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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=22936770
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30 April 2017
main subject
mitochondrion
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inferred from title
mitochondrial fission
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author name string
Youle RJ
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1
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30 April 2017
van der Bliek AM
series ordinal
2
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30 April 2017
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English
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30 April 2017
publication date
31 August 2012
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30 April 2017
published in
Science
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30 April 2017
volume
337
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30 April 2017
issue
6098
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30 April 2017
page(s)
1062-5
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30 April 2017
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PINK1 and Parkin target Miro for phosphorylation and degradation to arrest mitochondrial motility
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ER tubules mark sites of mitochondrial division
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During autophagy mitochondria elongate, are spared from degradation and sustain cell viability.
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Membrane remodeling induced by the dynamin-related protein Drp1 stimulates Bax oligomerization
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Regulation of OPA1 processing and mitochondrial fusion by m-AAA protease isoenzymes and OMA1
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SLP-2 is required for stress-induced mitochondrial hyperfusion
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Parkin is recruited selectively to impaired mitochondria and promotes their autophagy
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Mitochondrial inner-membrane fusion and crista maintenance requires the dynamin-related GTPase Mgm1.
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Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin
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The evolution and role of mitochondrial fusion and fission in aging and disease
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Physiological functions of mitochondrial fusion
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Emerging role for members of the Bcl-2 family in mitochondrial morphogenesis
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A mouse model of mitochondrial disease reveals germline selection against severe mtDNA mutations
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Inter-mitochondrial complementation: Mitochondria-specific system preventing mice from expression of disease phenotypes by mutant mtDNA.
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26 November 2018
Identifiers
DOI
10.1126/SCIENCE.1219855
2 references
stated in
PubMed
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=22936770
retrieved
30 April 2017
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
646579
OpenCitations bibliographic resource ID
646579
1 reference
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
646579
PMC publication ID
4762028
2 references
stated in
PubMed
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=22936770
retrieved
30 April 2017
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
646579
PubMed publication ID
22936770
2 references
stated in
PubMed
reference URL
https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=22936770
retrieved
30 April 2017
stated in
Consolidated OpenCitations Corpus – April 2017
OpenCitations bibliographic resource ID
646579
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