(Q28255650)

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Aysegul Uner

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Nilufer Guler

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Shuang-Yin Han

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Dimitrios Iliopoulos

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Peter McCue

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Kay Huebner

ATR Regulates Fragile Site Stability

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language of work or name

English

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publication date

15 April 2004

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published in

Cancer

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volume

100

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issue

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page(s)

1605-14

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cites work

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

FRA3B and other common fragile sites: the weakest links

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Genetic alterations in cancer as a result of breakage at fragile sites

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

The FHIT gene, spanning the chromosome 3p14.2 fragile site and renal carcinoma-associated t(3;8) breakpoint, is abnormal in digestive tract cancers

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Potential cancer therapy with the fragile histidine triad gene: review of the preclinical studies

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Clonal genetic alterations in the lungs of current and former smokers

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Molecular Damage in the Bronchial Epithelium of Current and Former Smokers

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

The tumor spectrum in FHIT-deficient mice

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

The expression of Fhit protein is related inversely to disease progression in patients with breast carcinoma

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Loss of FHIT protein expression is a marker of adverse evolution in good prognosis localized breast cancer.

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Loss of heterozygosity on chromosome arm 16q in breast cancer metastases

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Different genetic pathways in the evolution of invasive breast cancer are associated with distinct morphological subtypes

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Allelic imbalance analysis of chromosome 16q shows that grade I and grade III invasive ductal breast cancers follow different genetic pathways

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Ductal invasive G2 and G3 carcinomas of the breast are the end stages of at least two different lines of genetic evolution

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

WWOX: a candidate tumor suppressor gene involved in multiple tumor types

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

The common fragile site FRA16D and its associated gene WWOX are highly conserved in the mouse at Fra8E1

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Hyaluronidase induction of a WW domain-containing oxidoreductase that enhances tumor necrosis factor cytotoxicity

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Alternative transcripts of the candidate tumor suppressor gene, WWOX, are expressed at high levels in human breast tumors

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021

Do young breast cancer patients have worse outcomes?

1 reference

https://api.crossref.org/works/10.1002%2FCNCR.20137

7 January 2021