(Q24304901)

GOA release 2020-03-11

endoderm development

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GOA release 2020-03-11

author name string

Yu Chen

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Zeng-Qiang Han

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Wei-Dong Yan

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Chu-Zhong Tang

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Ji-Yan Xie

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Hong Chen

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Da-Yi Hu

The Journal of Thoracic and Cardiovascular Surgery

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language of work or name

English

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publication date

September 2010

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published in

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volume

140

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issue

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page(s)

684-7

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cites work

Phenotypes with GATA4 or NKX2.5 mutations in familial atrial septal defect

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Spontaneous closure of secundum atrial septal defect in infants and young children

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Natural course of atrial septal aneurysm in children and the potential for spontaneous closure of associated septal defect

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Superiority of transesophageal echocardiography in detecting cardiac source of embolism in patients with cerebral ischemia of uncertain etiology

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Atrial septal mass: transesophageal echocardiographic assessment

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Parsing the Heart: Genetic Modules for Organ Assembly

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GATA4 mutations cause human congenital heart defects and reveal an interaction with TBX5

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Gene therapy and genomic strategies for cardiovascular surgery: The emerging field of surgiomics

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GATA-4 activates transcription via two novel domains that are conserved within the GATA-4/5/6 subfamily

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Analysis of the signals and mechanisms mediating nuclear trafficking of GATA-4. Loss of DNA binding is associated with localization in intranuclear speckles

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GATA4 transcription factor is required for ventral morphogenesis and heart tube formation

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Requirement of the transcription factor GATA4 for heart tube formation and ventral morphogenesis

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Gata5 is required for the development of the heart and endoderm in zebrafish.

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Molecular pathways controlling heart development

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Congenital heart disease caused by mutations in the transcription factor NKX2-5

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A novel GATA4 mutation completely segregated with atrial septal defect in a large Japanese family

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Reduced penetrance, variable expressivity, and genetic heterogeneity of familial atrial septal defects

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NKX2.5 mutations in patients with tetralogy of fallot

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Mutations in the cardiac transcription factor NKX2.5 affect diverse cardiac developmental pathways

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Novel point mutation in the cardiac transcription factor CSX/NKX2.5 associated with congenital heart disease

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