(Q24302117)

English

Disabled-2 (Dab2) mediates transforming growth factor beta (TGFbeta)-stimulated fibronectin synthesis through TGFbeta-activated kinase 1 and activation of the JNK pathway

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Disabled-2 (Dab2) mediates transforming growth factor beta (TGFbeta)-stimulated fibronectin synthesis through TGFbeta-activated kinase 1 and activation of the JNK pathway (English)

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Disabled 2, mitogen-responsive phosphoprotein

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GOA release 2020-03-11

author name string

Barbara A Hocevar

1

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Celine Prunier

2

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Philip H Howe

3

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language of work or name

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publication date

8 July 2005

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Journal of Biological Chemistry

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280

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27

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25920-7

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JNK signaling pathway is required for efficient wound healing in Drosophila

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New insights into TGF-beta-Smad signalling

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A dominant negative TAK1 inhibits cellular fibrotic responses induced by TGF-beta

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Signal transduction by the JNK group of MAP kinases

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SARA, a FYVE domain protein that recruits Smad2 to the TGFbeta receptor

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Mechanisms of TGF-beta signaling from cell membrane to the nucleus

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The role of the Drosophila TAK homologue dTAK during development.

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Smad2 and Smad3 positively and negatively regulate TGF beta-dependent transcription through the forkhead DNA-binding protein FAST2

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Disabled-2 colocalizes with the LDLR in clathrin-coated pits and interacts with AP-2

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Myosin VI binds to and localises with Dab2, potentially linking receptor-mediated endocytosis and the actin cytoskeleton

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Transmembrane crosstalk between the extracellular matrix--cytoskeleton crosstalk

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A transcriptional partner for MAD proteins in TGF-beta signalling

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A highly specific inhibitor of human p38 MAP kinase binds in the ATP pocket

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Smad3/AP-1 interactions control transcriptional responses to TGF-beta in a promoter-specific manner

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Cytosolic adaptor protein Dab2 is an intracellular ligand of endocytic receptor gp600/megalin

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SP600125, an anthrapyrazolone inhibitor of Jun N-terminal kinase

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Smads bind directly to the Jun family of AP-1 transcription factors

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Transforming growth factor beta activation of p44mapk in proliferating cultures of epithelial cells

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PD 098059 is a specific inhibitor of the activation of mitogen-activated protein kinase kinase in vitro and in vivo

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Extracellular signal-regulated kinase and the small GTP-binding protein, Rac, contribute to the effects of transforming growth factor-beta1 on gene expression

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TAK1 mediates the ceramide signaling to stress-activated protein kinase/c-Jun N-terminal kinase

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Evidence for a role of Rho-like GTPases and stress-activated protein kinase/c-Jun N-terminal kinase (SAPK/JNK) in transforming growth factor beta-mediated signaling

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Involvement of the p38 mitogen-activated protein kinase pathway in transforming growth factor-beta-induced gene expression

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Interdependent SMAD and JNK signaling in transforming growth factor-beta-mediated transcription

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10.1074/JBC.M501150200

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