(Q54034693)

23 May 2018

title

Skeletal muscle respiratory uncoupling prevents diet-induced obesity and insulin resistance in mice. (English)

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23 May 2018

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23 May 2018

Clay F. Semenkovich

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23 May 2018

John O. Holloszy

object named as

J O Holloszy

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23 May 2018

author name string

B Li

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23 May 2018

L A Nolte

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23 May 2018

D H Han

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23 May 2018

T Coleman

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23 May 2018

language of work or name

English

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publication date

1 October 2000

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23 May 2018

published in

Nature Medicine

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23 May 2018

volume

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23 May 2018

issue

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23 May 2018

page(s)

1115-1120

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23 May 2018

Oxidative phosphorylation at the fin de siècle

cites work

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Mice lacking mitochondrial uncoupling protein are cold-sensitive but not obese

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Retinoids activate proton transport by the uncoupling proteins UCP1 and UCP2.

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Expression of the mitochondrial uncoupling protein gene from the aP2 gene promoter prevents genetic obesity

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Mutation in mitochondrial tRNALeu(UUR) gene in a large pedigree with maternally transmitted type II diabetes mellitus and deafness

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A common mitochondrial DNA variant is associated with insulin resistance in adult life

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Insulin-regulated mitochondrial gene expression is associated with glucose flux in human skeletal muscle

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Chronic activation of 5'-AMP-activated protein kinase increases GLUT-4, hexokinase, and glycogen in muscle

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A high fat diet impairs stimulation of glucose transport in muscle. Functional evaluation of potential mechanisms.

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Insulin Resistance of Muscle Glucose Transport in Rats Fed a High-Fat Diet: A Reevaluation

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Intramyocellular triglyceride content is a determinant of in vivo insulin resistance in humans: a 1H-13C nuclear magnetic resonance spectroscopy assessment in offspring of type 2 diabetic parents.

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Respiratory uncoupling induces delta-aminolevulinate synthase expression through a nuclear respiratory factor-1-dependent mechanism in HeLa cells.

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Rapid separation of creatine, phosphocreatine and adenosine metabolites by ion-pair reversed-phase high-performance liquid chromatography in plasma and cardiac tissue

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COOH-terminal disruption of lipoprotein lipase in mice is lethal in homozygotes, but heterozygotes have elevated triglycerides and impaired enzyme activity

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Relative hypoglycemia and hyperinsulinemia in mice with heterozygous lipoprotein lipase (LPL) deficiency. Islet LPL regulates insulin secretion

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Diet-induced diabetes activates an osteogenic gene regulatory program in the aortas of low density lipoprotein receptor-deficient mice

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7 January 2021

Identifiers

DOI

10.1038/80450

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23 May 2018

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23 May 2018