(Q45889385)

17 December 2017

title

Nitric oxide inactivates rat hepatic methionine adenosyltransferase In vivo by S-nitrosylation (English)

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17 December 2017

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17 December 2017

author name string

Corrales FJ

series ordinal

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17 December 2017

Miqueo C

series ordinal

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17 December 2017

Mato JM

series ordinal

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17 December 2017

language of work or name

English

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publication date

1 October 1998

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17 December 2017

published in

Hepatology

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17 December 2017

volume

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17 December 2017

issue

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17 December 2017

page(s)

1051-1057

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17 December 2017

Redox signaling: nitrosylation and related target interactions of nitric oxide

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Recombinant rat liver S-adenosyl-L-methionine synthetase tetramers and dimers are in equilibrium

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7 January 2021

Purification and comparison of two forms of S-adenosyl-L-methionine synthetase from rat liver

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Interaction of liver methionine adenosyltransferase with hydroxyl radical

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Cellular glutathione as a determinant of sensitivity to mercuric chloride toxicity. Prevention of toxicity by giving glutathione monoester

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Kupffer cell:hepatocyte cocultures release nitric oxide in response to bacterial endotoxin

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Anti-inflammatory glucocorticoids inhibit the induction by endotoxin of nitric oxide synthase in the lung, liver and aorta of the rat

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S-adenosyl-L-methionine synthetase and phospholipid methyltransferase are inhibited in human cirrhosis

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7 January 2021

Specific loss of the high-molecular-weight form of S-adenosyl-L-methionine synthetase in human liver cirrhosis

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7 January 2021

Rate of Disappearance From Plasma of Intravenously Administered Methionine in Patients With Liver Damage

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7 January 2021

Bcl-2 expression causes redistribution of glutathione to the nucleus

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7 January 2021

Hepatic extraction of indocyanine green is depressed early in sepsis despite increased hepatic blood flow and cardiac output

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7 January 2021

Biochemistry of endotoxins

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7 January 2021

Multiple cytokines are required to induce hepatocyte nitric oxide production and inhibit total protein synthesis

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Blood Flow Regulation by S-Nitrosohemoglobin in the Physiological Oxygen Gradient

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Activation of the Cardiac Calcium Release Channel (Ryanodine Receptor) by Poly-S-Nitrosylation

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7 January 2021

Identifiers

DOI

10.1002/HEP.510280420

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17 December 2017

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17 December 2017