(Q45103307)

9 December 2017

title

Inhibition of the electrogenic Na pump underlies delayed depolarization of cortical neurons after mechanical injury or glutamate (English)

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9 December 2017

Tavalin SJ

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9 December 2017

Ellis EF

series ordinal

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9 December 2017

Satin LS

series ordinal

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9 December 2017

language of work or name

English

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publication date

1 February 1997

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Journal of Neurophysiology

9 December 2017

published in

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9 December 2017

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9 December 2017

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9 December 2017

page(s)

632-638

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Glutamate-induced neuronal death: A succession of necrosis or apoptosis depending on mitochondrial function

9 December 2017

cites work

1 reference

https://api.crossref.org/works/10.1152%2FJN.1997.77.2.632

Oxygen and glucose consumption related to Na+-K+ transport in canine brain

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Do defecs in mitochondrial energy metabolism underlie the pathology of neurodegenerative diseases?

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Inhibition of alpha 2/alpha 3 sodium pump isoforms potentiates glutamate neurotoxicity

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Electrophysiology of glutamate neurotoxicity in vitro: induction of a calcium-dependent extended neuronal depolarization

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Oxidative stress, glutamate, and neurodegenerative disorders

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A novel neuronal messenger molecule in brain: the free radical, nitric oxide

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A new model for rapid stretch-induced injury of cells in culture: characterization of the model using astrocytes

21 January 2018

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Postnatal development of electrogenic sodium pump activity in rat hippocampal pyramidal neurons

21 January 2018

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Excessive intracellular Ca2+ inhibits glutamate-induced Na(+)-K+ pump activation in rat hippocampal neurons

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Developmental and regional differences in the localization of Na,K-ATPase activity in the rabbit hippocampus

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Improved patch-clamp techniques for high-resolution current recording from cells and cell-free membrane patches

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Importance of pharmacologic considerations in the evaluation of new treatments for acute spinal cord injury

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Pretreatment with phencyclidine, an N-methyl-D-aspartate antagonist, attenuates long-term behavioral deficits in the rat produced by traumatic brain injury

21 January 2018

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Increased vulnerability of the mildly traumatized rat brain to cerebral ischemia: the use of controlled secondary ischemia as a research tool to identify common or different mechanisms contributing to mechanical and ischemic brain injury

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Massive increases in extracellular potassium and the indiscriminate release of glutamate following concussive brain injury

21 January 2018

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Prolonged memory impairment in the absence of hippocampal cell death following traumatic brain injury in the rat.

21 January 2018

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Post-natal development of electrophysiological properties of rat cerebral cortical pyramidal neurones

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Changes in cortical extracellular levels of energy-related metabolites and amino acids following concussive brain injury in rats.

21 January 2018

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Glutamate becomes neurotoxic via the N-methyl-D-aspartate receptor when intracellular energy levels are reduced

21 January 2018

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Effects of kainic acid on high-energy metabolites in the mouse striatum

21 January 2018

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Delayed neurotoxicity of excitatory amino acids in vitro

21 January 2018

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Changes in extracellular potassium concentration in cortex and brain stem during the acute phase of experimental closed head injury

21 January 2018

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Mechanical perturbation of cultured cortical neurons reveals a stretch-induced delayed depolarization

21 January 2018

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Electrogenic sodium pump in nerve and muscle cells

21 January 2018

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https://api.crossref.org/works/10.1152%2FJN.1997.77.2.632

Activation of electrogenic sodium pump in hippocampal CA1 neurons following glutamate-induced depolarization

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJN.1997.77.2.632

Glutamate-induced intracellular acidification of cultured hippocampal neurons demonstrates altered energy metabolism resulting from Ca2+ loads

21 January 2018

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The dependence of the respiration of brain cortex on active cation transport

21 January 2018

1 reference

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21 January 2018

Identifiers

DOI

10.1152/JN.1997.77.2.632

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1 reference