(Q44133546)

English

Pharmacologic inhibition of poly(adenosine diphosphate-ribose) polymerase may represent a novel therapeutic approach in chronic heart failure

scientific article published in September 2002

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scholarly article

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Europe PubMed Central

PubMed publication ID

30 November 2017

Pharmacologic inhibition of poly(adenosine diphosphate-ribose) polymerase may represent a novel therapeutic approach in chronic heart failure (English)

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

chronic heart failure

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based on heuristic

inferred from title

1

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

5

object named as

Csaba Szabó

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

author name string

Lucas Liaudet

2

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Europe PubMed Central

PubMed publication ID

30 November 2017

Jon g Mabley

3

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

Katalin Komjáti

4

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Europe PubMed Central

PubMed publication ID

30 November 2017

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publication date

1 September 2002

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Europe PubMed Central

PubMed publication ID

30 November 2017

Journal of the American College of Cardiology

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Europe PubMed Central

PubMed publication ID

30 November 2017

40

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

5

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

1006-1016

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Europe PubMed Central

PubMed publication ID

30 November 2017

Inhibition of the activity of poly(ADP ribose) synthetase reduces ischemia-reperfusion injury in the heart and skeletal muscle

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Genetic disruption of poly (ADP-ribose) synthetase inhibits the expression of P-selectin and intercellular adhesion molecule-1 in myocardial ischemia/reperfusion injury

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Poly(ADP-Ribose) polymerase inhibition reduces reperfusion injury after heart transplantation

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Endothelial dysfunction in a rat model of endotoxic shock. Importance of the activation of poly (ADP-ribose) synthetase by peroxynitrite

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Protection against hemorrhagic shock in mice genetically deficient in poly(ADP-ribose)polymerase

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Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemia

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Mice lacking the poly(ADP-ribose) polymerase gene are resistant to pancreatic beta-cell destruction and diabetes development induced by streptozocin.

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Diabetic endothelial dysfunction: the role of poly(ADP-ribose) polymerase activation

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Rapid reversal of the diabetic endothelial dysfunction by pharmacological inhibition of poly(ADP-ribose) polymerase

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The role of poly(ADP-ribose) polymerase activation in the development of myocardial and endothelial dysfunction in diabetes

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Mitochondrial electron transport complex I is a potential source of oxygen free radicals in the failing myocardium

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Intravenous allopurinol decreases myocardial oxygen consumption and increases mechanical efficiency in dogs with pacing-induced heart failure

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Endothelial dysfunction in chronic myocardial infarction despite increased vascular endothelial nitric oxide synthase and soluble guanylate cyclase expression: role of enhanced vascular superoxide production

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Peroxynitrite induced nitration and inactivation of myofibrillar creatine kinase in experimental heart failure

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Allopurinol improves myocardial efficiency in patients with idiopathic dilated cardiomyopathy

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Oxidative stress-mediated cardiac cell death is a major determinant of ventricular dysfunction and failure in dog dilated cardiomyopathy

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Suppression of poly (ADP-ribose) polymerase activation by 3-aminobenzamide in a rat model of myocardial infarction: long-term morphological and functional consequences

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Myocardial ischemic preconditioning in rodents is dependent on poly (ADP-ribose) synthetase.

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Myocardial protection by PJ34, a novel potent poly (ADP-ribose) synthetase inhibitor

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Protection against myocardial ischemia and reperfusion injury by 3-aminobenzamide, an inhibitor of poly (ADP-ribose) synthetase

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Propagation of the apoptotic signal by mitochondrial waves

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Temporal evolution of endothelial dysfunction in a rat model of chronic heart failure.

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Hemodynamic stresses induce endothelial dysfunction and remodeling of pulmonary artery in experimental compensated heart failure

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Improvement of left ventricular remodeling and function by hydroxymethylglutaryl coenzyme a reductase inhibition with cerivastatin in rats with heart failure after myocardial infarction

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Progression of heart failure after myocardial infarction in the rat.

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Elevated circulating level of ghrelin in cachexia associated with chronic heart failure: relationships between ghrelin and anabolic/catabolic factors

1 reference

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An inhibitor of poly (ADP-ribose) synthetase activity reduces contractile dysfunction and preserves high energy phosphate levels during reperfusion of the ischaemic rat heart

1 reference

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BGP-15, a nicotinic amidoxime derivate protecting heart from ischemia reperfusion injury through modulation of poly(ADP-ribose) polymerase

1 reference

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Protection against hypoxia-reoxygenation in the absence of poly (ADP-ribose) synthetase in isolated working hearts

1 reference

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Heterogeneous expression and activity of endothelial and inducible nitric oxide synthases in end-stage human heart failure: their relation to lesion site and beta-adrenergic receptor therapy

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Inducible nitric oxide synthase (iNOS) in the human heart: expression and localization in congestive heart failure

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Cardiomyocyte overexpression of iNOS in mice results in peroxynitrite generation, heart block, and sudden death

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Role of myocardial inducible nitric oxide synthase in contractile dysfunction and beta-adrenergic hyporesponsiveness in rats with experimental volume-overload heart failure.

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Nitric oxide, superoxide, and peroxynitrite: the good, the bad, and ugly

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Peroxynitrite is a major contributor to cytokine-induced myocardial contractile failure

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Endothelial function in chronic congestive heart failure

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Inducible nitric oxide synthase-derived superoxide contributes to hypereactivity in small mesenteric arteries from a rat model of chronic heart failure

1 reference

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1 reference

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10.1016/S0735-1097(02)02062-4

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

30 November 2017

ResearchGate publication ID

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