(Q43581654)

24 November 2017

title

The p75 neurotrophin receptor activates Akt (protein kinase B) through a phosphatidylinositol 3-kinase-dependent pathway (English)

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24 November 2017

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24 November 2017

Bhakar AL

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24 November 2017

Kennedy TE

series ordinal

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24 November 2017

Barker PA

series ordinal

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24 November 2017

language of work or name

English

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publication date

18 April 2001

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Journal of Biological Chemistry

24 November 2017

published in

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24 November 2017

volume

276

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24 November 2017

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24 November 2017

page(s)

23097-23104

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Physiology of the neurotrophins

24 November 2017

cites work

1 reference

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Akt/protein kinase B is regulated by autophosphorylation at the hypothetical PDK-2 site

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Akt promotes cell survival by phosphorylating and inhibiting a Forkhead transcription factor

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Akt/Protein kinase B inhibits cell death by preventing the release of cytochrome c from mitochondria

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Akt phosphorylation site found in human caspase-9 is absent in mouse caspase-9.

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The Akt proto-oncogene links Ras to Pak and cell survival signals

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Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death Machinery

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NF-kappaB is a target of AKT in anti-apoptotic PDGF signalling

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The p75 Neurotrophin Receptor Mediates Neuronal Apoptosis and Is Essential for Naturally Occurring Sympathetic Neuron Death

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Identification of a zinc finger protein whose subcellular distribution is regulated by serum and nerve growth factor

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The zinc finger protein NRIF interacts with the neurotrophin receptor p75(NTR) and participates in programmed cell death

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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SHIP, a new player in cytokine-induced signalling

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Neurotrophins stimulate the release of dopamine from rat mesencephalic neurons via Trk and p75Lntr receptors

21 January 2018

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Brain-derived Neurotrophic Factor Binding to the p75 Neurotrophin Receptor Reduces TrkA Signaling While Increasing Serine Phosphorylation in the TrkA Intracellular Domain

21 January 2018

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21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.M011520200

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24 November 2017

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