(Q40161252)

18 September 2017

title

Histone deacetylase inhibitors suppress natural killer cell cytolytic activity (English)

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18 September 2017

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18 September 2017

Hans Wilhelm Doerr

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18 September 2017

Jindrich Cinatl

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18 September 2017

Jörg Kreuter

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18 September 2017

Henry Ogbomo

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18 September 2017

language of work or name

English

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publication date

1 March 2007

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18 September 2017

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inferred from page(s)

published in

FEBS Letters

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18 September 2017

volume

581

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18 September 2017

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18 September 2017

page(s)

1317-1322

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Histone deacetylase inhibitors and the promise of epigenetic (and more) treatments for cancer

18 September 2017

cites work

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Histone deacetylases induce angiogenesis by negative regulation of tumor suppressor genes

21 January 2018

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Valproic acid inhibits angiogenesis in vitro and in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Human natural killer cells: Molecular mechanisms controlling NK cell activation and tumor cell lysis

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Cancer cells become susceptible to natural killer cell killing after exposure to histone deacetylase inhibitors due to glycogen synthase kinase-3-dependent expression of MHC class I-related chain A and B.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Natural killer cell-mediated lysis of hepatoma cells via specific induction of NKG2D ligands by the histone deacetylase inhibitor sodium valproate

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

NK sensitivity of neuroblastoma cells determined by a highly sensitive coupled luminescent method

21 January 2018

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https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Valproic acid induces extracellular signal-regulated kinase 1/2 activation and inhibits apoptosis in endothelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Molecular analysis of the methylprednisolone-mediated inhibition of NK-cell function: evidence for different susceptibility of IL-2- versus IL-15-activated NK cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Evolving anticancer drug valproic acid: insights into the mechanism and clinical studies.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

The corticosteroid-induced inhibitory effect on NK cell function reflects down-regulation and/or dysfunction of triggering receptors involved in natural cytotoxicity.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Deficient expression of NCR in NK cells from acute myeloid leukemia: Evolution during leukemia treatment and impact of leukemia cells in NCRdull phenotype induction.

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Histone deacetylase inhibitors: new drugs for the treatment of inflammatory diseases?

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Corticosteroid effects on cell signalling

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Acute leukemia associated with valproic acid treatment: a novel mechanism for leukemogenesis?

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

Defective expression and function of natural killer cell-triggering receptors in patients with acute myeloid leukemia

21 January 2018

1 reference

https://api.crossref.org/works/10.1016%2FJ.FEBSLET.2007.02.045

10.1016/J.FEBSLET.2007.02.045

21 January 2018

Identifiers

DOI

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18 September 2017

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