Pages that link to "Q24642983"
The following pages link to Oncostatin M receptor-beta mutations underlie familial primary localized cutaneous amyloidosis (Q24642983):
Displaying 33 items.
- primary cutaneous amyloidosis (Q7243115) (← links)
- Targeting of prosurvival pathways as therapeutic approaches against primary effusion lymphomas: past, present, and Future (Q28081199) (← links)
- Amyloidosis cutis dyschromica in two female siblings: cases report (Q33820296) (← links)
- A novel missense mutation in oncostatin M receptor beta causing primary localized cutaneous amyloidosis (Q33904280) (← links)
- Novel IL31RA gene mutation and ancestral OSMR mutant allele in familial primary cutaneous amyloidosis (Q34328403) (← links)
- In vitro amyloidogenic peptides of galectin-7: possible mechanism of amyloidogenesis of primary localized cutaneous amyloidosis (Q34355760) (← links)
- Atopic dermatitis in children: clinical features, pathophysiology, and treatment (Q34625597) (← links)
- Bullous variant of familial biphasic lichen amyloidosis: a unique combination of three rare presentations (Q35052289) (← links)
- Characterization of type I interferon pathway during hepatic differentiation of human pluripotent stem cells and hepatitis C virus infection (Q36145616) (← links)
- RET mutation p.S891A in a Chinese family with familial medullary thyroid carcinoma and associated cutaneous amyloidosis binding OSMR variant p.G513D. (Q36545363) (← links)
- Signaling network of Oncostatin M pathway (Q36864673) (← links)
- What causes itch in atopic dermatitis? (Q37208966) (← links)
- Pathophysiology of pruritus in primary localized cutaneous amyloidosis (Q38382739) (← links)
- Bilateral recurrent facial primary cutaneous nodular amyloidosis (PCNA). (Q39560386) (← links)
- Association of the oncostatin m receptor gene polymorphisms with papillary thyroid cancer in the korean population (Q40134908) (← links)
- Effects of oncostatin M on cell proliferation and osteogenic differentiation in C3H10T1/2 (Q42111852) (← links)
- MCP-1 as an effector of IL-31 signaling in familial primary cutaneous amyloidosis (Q45895662) (← links)
- Increase in sensory sensitivity around, but not in the central part of, the hyperkeratotic papule in lichen amyloidosis. (Q48369214) (← links)
- Detection of common mutations in sporadic primary localized cutaneous amyloidosis by DNA mass spectrometry (Q48400060) (← links)
- Loss of GPNMB Causes Autosomal-Recessive Amyloidosis Cutis Dyschromica in Humans (Q50034858) (← links)
- Familial primary localized cutaneous amyloidosis with an oncostatin M receptor-β mutation, Pro694Leu. (Q50974473) (← links)
- Is targeting interleukin-31 a cure for the itch? Lessons from amyloidosis. (Q51696680) (← links)
- Paracrine Effects of FGF23 on the Heart. (Q55221465) (← links)
- OSMR gene effect on the pathogenesis of chronic autoimmune Urticaria via the JAK/STAT3 pathway. (Q55421591) (← links)
- The RET C611Y mutation causes MEN 2A and associated cutaneous (Q57300636) (← links)
- Overexpression of microRNA-183 promotes apoptosis of substantia nigra neurons via the inhibition of OSMR in a mouse model of Parkinson's disease (Q59798268) (← links)
- Macular cutaneous amyloidosis treated with methyl aminolevulinate and daylight photodynamic therapy: A case report (Q64250009) (← links)
- [Cutaneous amyloidosis] (Q83011244) (← links)
- [Hereditary and non-hereditary cutaneous amyloidoses] (Q83553108) (← links)
- IL-11 in cardiac and renal fibrosis: Late to the party but a central player (Q89542063) (← links)
- Prokaryotic soluble overexpression and purification of oncostatin M using a fusion approach and genetically engineered E. coli strains (Q90241868) (← links)
- Interleukin-31-mediated photoablation of pruritogenic epidermal neurons reduces itch-associated behaviours in mice (Q92830114) (← links)
- OSMR controls glioma stem cell respiration and confers resistance of glioblastoma to ionizing radiation (Q98513578) (← links)