(Q28581284)

31 January 2017

title

Vitamin D(3)-up-regulated protein-1 is a stress-responsive gene that regulates cardiomyocyte viability through interaction with thioredoxin (English)

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31 January 2017

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Yanlin Wang

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31 January 2017

Gilles W. De Keulenaer

series ordinal

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31 January 2017

Richard T. Lee

series ordinal

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31 January 2017

language of work or name

English

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publication date

19 July 2002

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Journal of Biological Chemistry

31 January 2017

published in

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31 January 2017

volume

277

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31 January 2017

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31 January 2017

page(s)

26496–26500

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Oxygen-derived free radicals and postischemic myocardial dysfunction ("stunned myocardium").

31 January 2017

cites work

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A possible interaction of thioredoxin with VDUP1 in HeLa cells detected in a yeast two-hybrid system

21 January 2018

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Identification of thioredoxin-binding protein-2/vitamin D(3) up-regulated protein 1 as a negative regulator of thioredoxin function and expression

21 January 2018

1 reference

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Vitamin D3 up-regulated protein 1 mediates oxidative stress via suppressing the thioredoxin function

21 January 2018

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Induction of tenascin-C in cardiac myocytes by mechanical deformation. Role of reactive oxygen species

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A simplified system for generating recombinant adenoviruses

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Autocrine release of angiotensin II mediates stretch-induced hypertrophy of cardiac myocytes in vitro

21 January 2018

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Endothelin-1 is involved in mechanical stress-induced cardiomyocyte hypertrophy.

21 January 2018

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Reactive oxygen species mediate amplitude-dependent hypertrophic and apoptotic responses to mechanical stretch in cardiac myocytes.

21 January 2018

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Mammalian thioredoxin is a direct inhibitor of apoptosis signal-regulating kinase (ASK) 1.

21 January 2018

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Distinct roles of thioredoxin in the cytoplasm and in the nucleus. A two-step mechanism of redox regulation of transcription factor NF-kappaB.

21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.M202133200

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31 January 2017

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